Oxidative stress induces overgrowth of the Drosophila neuromuscular junction. - Wikidata - awgldk - TriplyDB

Oxidative stress induces overgrowth of the Drosophila neuromuscular junction.

Oxidative stress induces overgrowth of the Drosophila neuromuscular junction.

http://www.wikidata.org/entity/Q35409729

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The Crossroads of Synaptic Growth Signaling, Membrane Traffic and Neurological Disease: Insights from Drosophila The synaptic maintenance problem: membrane recycling, Ca2+ homeostasis and late onset degeneration Identification of dietary alanine toxicity and trafficking dysfunction in a Drosophila model of hereditary sensory and autonomic neuropathy type 1.Reactive Oxygen Species: Physiological and Physiopathological Effects on Synaptic Plasticity Neuronal development is promoted by weakened intrinsic antioxidant defences due to epigenetic repression of Nrf2 JNK pathway activation is controlled by Tao/TAOK3 to modulate ethanol sensitivity The intricate relationship between microtubules and their associated motor proteins during axon growth and maintenance.Epsin 1 Promotes Synaptic Growth by Enhancing BMP Signal Levels in Motoneuron Nuclei.Antioxidants maintain E-cadherin levels to limit Drosophila prohemocyte differentiation.Rab8, POSH, and TAK1 regulate synaptic growth in a Drosophila model of frontotemporal dementia A novel Drosophila SOD2 mutant demonstrates a role for mitochondrial ROS in neurodevelopment and disease Dendritic growth gated by a steroid hormone receptor underlies increases in activity in the developing Drosophila locomotor system.Defective autophagy in Parkinson's disease: role of oxidative stress.Autophagy in synaptic development, function, and pathology.Poly(GR) in C9ORF72-Related ALS/FTD Compromises Mitochondrial Function and Increases Oxidative Stress and DNA Damage in iPSC-Derived Motor Neurons.Parkinsonian toxin-induced oxidative stress inhibits basal autophagy in astrocytes via NQO2/quinone oxidoreductase 2: Implications for neuroprotection.Oxidative stress and autophagy: mediators of synapse growth?parkin-induced defects in neurophysiology and locomotion are generated by metabolic dysfunction and not oxidative stress.Lipid metabolic perturbation is an early-onset phenotype in adult spinster mutants: a Drosophila model for lysosomal storage disorders.Regulation of neuronal development and function by ROS.Sphingolipids regulate neuromuscular synapse structure and function in Drosophila

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Oxidative stress induces overgrowth of the Drosophila neuromuscular junction.

http://www.wikidata.org/entity/Q35409729

description

2011 nî lūn-bûn

@nan

2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Oxidative stress induces overgrowth of the Drosophila neuromuscular junction.

@ast

Oxidative stress induces overgrowth of the Drosophila neuromuscular junction.

@en

type

label

Oxidative stress induces overgrowth of the Drosophila neuromuscular junction.

@ast

Oxidative stress induces overgrowth of the Drosophila neuromuscular junction.

@en

prefLabel

Oxidative stress induces overgrowth of the Drosophila neuromuscular junction.

@ast

Oxidative stress induces overgrowth of the Drosophila neuromuscular junction.

@en

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PNAS.1014511108

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Oxidative stress induces overgrowth of the Drosophila neuromuscular junction.

@en

P2093

Helen E Jarrett

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Iain M Robinson

http://www.w3.org/2001/XMLSchema#string

Kate Gowers

http://www.w3.org/2001/XMLSchema#string

Laura Briggs

http://www.w3.org/2001/XMLSchema#string

Salma Chalak

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Valerie J Milton

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P2860

Copper,zinc superoxide dismutase is primarily a cytosolic protein in human cells

Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1.

Regulation of JNK signaling by GSTp

Evidence for cell autonomous AP1 function in regulation of Drosophila motor-neuron plasticity

Signal transduction by the JNK group of MAP kinases

AP-1 functions upstream of CREB to control synaptic plasticity in Drosophila

A block of autophagy in lysosomal storage disorders

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Drosophila spichthyin inhibits BMP signaling and regulates synaptic growth and axonal microtubules.

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17521-17526

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scholarly article

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10.1073/PNAS.1014511108

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42

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108

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Sean T. Sweeney

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2011-10-10T00:00:00Z

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51708208

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21987827

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