Historic evidence to support a causal relationship between spirochetal infections and Alzheimer's disease.
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Periodontal disease's contribution to Alzheimer's disease progression in Down syndromeIndividuality, phenotypic differentiation, dormancy and 'persistence' in culturable bacterial systems: commonalities shared by environmental, laboratory, and clinical microbiologyA Bacterial Component to Alzheimer’s-Type Dementia Seen via a Systems Biology Approach that Links Iron Dysregulation and Inflammagen Shedding to DiseaseCan oral infection be a risk factor for Alzheimer's disease?16S rRNA Next Generation Sequencing Analysis Shows Bacteria in Alzheimer's Post-Mortem BrainPolymicrobial Infections In Brain Tissue From Alzheimer's Disease PatientsMicrobes and Alzheimer's Disease.On the translocation of bacteria and their lipopolysaccharides between blood and peripheral locations in chronic, inflammatory diseases: the central roles of LPS and LPS-induced cell death.Viscoelastic and ultrastructural characteristics of whole blood and plasma in Alzheimer-type dementia, and the possible role of bacterial lipopolysaccharides (LPS)Bacterial Amyloid and DNA are Important Constituents of Senile Plaques: Further Evidence of the Spirochetal and Biofilm Nature of Senile PlaquesHSV presence in brains of individuals without dementia: the TASTY brain seriesNeuroinflammation and Infection: Molecular Mechanisms Associated with Dysfunction of Neurovascular Unit.Apolipoprotein E Related Co-Morbidities and Alzheimer's Disease.Inflammasome Involvement in Alzheimer's Disease.The infectious etiology of Alzheimer's Disease.Rhamnolipids, Microbial Virulence Factors, in Alzheimer's Disease.Can Better Management of Periodontal Disease Delay the Onset and Progression of Alzheimer's Disease?Chronic Porphyromonas gingivalis infection accelerates the occurrence of age-related granules in ApoE-/- mice brains.Syphilitic dementia and lipid metabolism.Persistent infections, immune-senescence and Alzheimer's disease.Exploring the Association between Alzheimer's Disease, Oral Health, Microbial Endocrinology and Nutrition.Hyperhomocysteinemia, Suppressed Immunity, and Altered Oxidative Metabolism Caused by Pathogenic Microbes in Atherosclerosis and Dementia.Molecular Mechanisms for Herpes Simplex Virus Type 1 Pathogenesis in Alzheimer's Disease.Molecular Mechanisms for Herpes Simplex Virus Type 1 Pathogenesis in Alzheimer’s Disease.The Physiological Roles of Amyloid-β Peptide Hint at New Ways to Treat Alzheimer's Disease.Questions concerning the role of amyloid-β in the definition, aetiology and diagnosis of Alzheimer's diseaseThe Potential of LPS-Binding Protein to Reverse Amyloid Formation in Plasma Fibrin of Individuals With Alzheimer-Type DementiaRole of Microbes in the Development of Alzheimer's Disease: State of the Art - An International Symposium Presented at the 2017 IAGG Congress in San FranciscoCorrelative Light-Electron Microscopy detects lipopolysaccharide and its association with fibrin fibres in Parkinson's Disease, Alzheimer's Disease and Type 2 Diabetes Mellitus
P2860
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P2860
Historic evidence to support a causal relationship between spirochetal infections and Alzheimer's disease.
description
2015 nî lūn-bûn
@nan
2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Historic evidence to support a ...... tions and Alzheimer's disease.
@ast
Historic evidence to support a ...... tions and Alzheimer's disease.
@en
type
label
Historic evidence to support a ...... tions and Alzheimer's disease.
@ast
Historic evidence to support a ...... tions and Alzheimer's disease.
@en
prefLabel
Historic evidence to support a ...... tions and Alzheimer's disease.
@ast
Historic evidence to support a ...... tions and Alzheimer's disease.
@en
P2860
P356
P1476
Historic evidence to support a ...... tions and Alzheimer's disease.
@en
P2860
P356
10.3389/FNAGI.2015.00046
P577
2015-04-16T00:00:00Z