Dilation of epicardial coronary arteries by the G protein-coupled estrogen receptor agonists G-1 and ICI 182,780. - Wikidata - awgldk - TriplyDB

Dilation of epicardial coronary arteries by the G protein-coupled estrogen receptor agonists G-1 and ICI 182,780.

Dilation of epicardial coronary arteries by the G protein-coupled estrogen receptor agonists G-1 and ICI 182,780.

http://www.wikidata.org/entity/Q35447240

about

G-protein-coupled estrogen receptor as a new therapeutic target for treating coronary artery disease The G-protein-coupled estrogen receptor GPER in health and disease Estrogen biology: new insights into GPER function and clinical opportunities Aldosterone mediates its rapid effects in vascular endothelial cells through GPER activation G protein-coupled estrogen receptor in energy homeostasis and obesity pathogenesis.Oestrogen upregulates the sarcoplasmic reticulum Ca(2+) ATPase pump in coronary arteries.Estrogen-mediated renoprotection following cardiac arrest and cardiopulmonary resuscitation is robust to GPR30 gene deletion GPER/GPR30 and Regulation of Vascular Tone and Blood Pressure.Evidence that the G protein-coupled membrane receptor GPR30 contributes to the cardiovascular actions of estrogen.Deletion of G protein-coupled estrogen receptor increases endothelial vasoconstriction Improvement of vascular function by acute and chronic treatment with the GPR30 agonist G1 in experimental diabetes mellitus.A common hypofunctional genetic variant of GPER is associated with increased blood pressure in women.G protein-coupled estrogen receptor protects from atherosclerosis Estrogen receptor subtypes mediate distinct microvascular dilation and reduction in [Ca2+]I in mesenteric microvessels of female rat.Obesity, insulin resistance and diabetes: sex differences and role of oestrogen receptors.G protein-coupled oestrogen receptor 1 (GPER1)/GPR30: a new player in cardiovascular and metabolic oestrogenic signalling Unraveling the mechanisms underlying the rapid vascular effects of steroids: sorting out the receptors and the pathways.Diabetes impairs the vascular effects of aldosterone mediated by G protein-coupled estrogen receptor activation Activation of G protein-coupled estrogen receptor induces endothelium-independent relaxation of coronary artery smooth muscle.The G protein-coupled estrogen receptor GPER/GPR30 as a regulator of cardiovascular function.International Union of Basic and Clinical Pharmacology. XCVII. G Protein-Coupled Estrogen Receptor and Its Pharmacologic Modulators Estrogen- and xenoestrogen-induced ERK signaling in pituitary tumor cells involves estrogen receptor-α interactions with G protein-αi and caveolin I.Pregnancy Augments G Protein Estrogen Receptor (GPER) Induced Vasodilation in Rat Uterine Arteries via the Nitric Oxide - cGMP Signaling Pathway GPER regulates endothelin-dependent vascular tone and intracellular calcium.The role of estrogen on the pathology of cerebral aneurysms.G protein-coupled estrogen receptor-mediated effects on cytosolic calcium and nanomechanics in brain microvascular endothelial cells.Activation of G protein-coupled estrogen receptor 1 induces coronary artery relaxation via Epac/Rap1-mediated inhibition of RhoA/Rho kinase pathway in parallel with PKA.Adaptive increases in expression and vasodilator activity of estrogen receptor subtypes in a blood vessel-specific pattern during pregnancy.Bradycardic effects mediated by activation of G protein-coupled estrogen receptor in rat nucleus ambiguus.G protein-coupled estrogen receptor inhibits vascular prostanoid production and activity.The Expanding Complexity of Estrogen Receptor Signaling in the Cardiovascular System.G protein-coupled estrogen receptor-selective ligands modulate endometrial tumor growth GPER-novel membrane oestrogen receptor.Vasodilation by GPER in mesenteric arteries involves both endothelial nitric oxide and smooth muscle cAMP signaling.Hormonal effects on blood vessels.Estrogen and the cardiovascular system.Vascular effects of aldosterone: sorting out the receptors and the ligands.Membrane estrogen receptors: their role in blood pressure regulation and cardiovascular disease.Sex Differences in Vascular Physiology and Pathophysiology: Estrogen and Androgen Signaling in Health and Disease.Influence of sex on cardiovascular drug responses: role of estrogen.

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P2860

Dilation of epicardial coronary arteries by the G protein-coupled estrogen receptor agonists G-1 and ICI 182,780.

http://www.wikidata.org/entity/Q35447240

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Dilation of epicardial coronar ...... agonists G-1 and ICI 182,780.

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Dilation of epicardial coronar ...... agonists G-1 and ICI 182,780.

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Dilation of epicardial coronar ...... agonists G-1 and ICI 182,780.

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Dilation of epicardial coronar ...... agonists G-1 and ICI 182,780.

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Dilation of epicardial coronar ...... agonists G-1 and ICI 182,780.

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Dilation of epicardial coronar ...... agonists G-1 and ICI 182,780.

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Dilation of epicardial coronar ...... agonists G-1 and ICI 182,780.

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Eric R Prossnitz

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Matthias Barton

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Matthias R Meyer

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Oliver Baretella

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P2860

A transmembrane intracellular estrogen receptor mediates rapid cell signaling

Identity of an estrogen membrane receptor coupled to a G protein in human breast cancer cells

Lasofoxifene in postmenopausal women with osteoporosis

Activation of a novel estrogen receptor, GPER, is cardioprotective in male and female rats

Estrogen-induced activation of Erk-1 and Erk-2 requires the G protein-coupled receptor homolog, GPR30, and occurs via trans-activation of the epidermal growth factor receptor through release of HB-EGF

Cloning of cDNAs encoding G protein-coupled receptor expressed in human endothelial cells exposed to fluid shear stress.

A novel estrogen receptor GPER inhibits mitochondria permeability transition pore opening and protects the heart against ischemia-reperfusion injury.

Estrogen receptor alpha mediates the nongenomic activation of endothelial nitric oxide synthase by estrogen.

Global burden of cardiovascular diseases: part I: general considerations, the epidemiologic transition, risk factors, and impact of urbanization.

Estrogen and vascular function.

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10.1159/000315497

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1

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86

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