Listeria monocytogenes infects human endothelial cells by two distinct mechanisms.
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Rhombencephalitis Caused by Listeria monocytogenes in Humans and Ruminants: A Zoonosis on the Rise?Entry of Listeria monocytogenes into neurons occurs by cell-to-cell spread: an in vitro studyDissemination of Listeria monocytogenes by infected phagocytes.Listeria monocytogenes-infected phagocytes can initiate central nervous system infection in miceInvasion of the central nervous system by intracellular bacteriaPathogens penetrating the central nervous system: infection pathways and the cellular and molecular mechanisms of invasionEndothelial Cells Use a Formin-Dependent Phagocytosis-Like Process to Internalize the Bacterium Listeria monocytogenesCell-Based Screen Identifies Human Interferon-Stimulated Regulators of Listeria monocytogenes InfectionInvasive Listeria monocytogenes Gastroenteritis Leading to Stupor, Bacteremia, Fever, and Diarrhea: A Rare Life-Threatening Condition.The Key Events Dose-Response Framework: its potential for application to foodborne pathogenic microorganismsListeria monocytogenes virulence factors that stimulate endothelial cells.Interaction of Listeria monocytogenes with human brain microvascular endothelial cells: InlB-dependent invasion, long-term intracellular growth, and spread from macrophages to endothelial cells.Phosphatidylcholine-specific phospholipase C from Listeria monocytogenes is an important virulence factor in murine cerebral listeriosis.Virulent rough filaments of Listeria monocytogenes from clinical and food samples secreting wild-type levels of cell-free p60 protein.Listeria pathogenesis and molecular virulence determinantsMechanism of the intracellular killing and modulation of antibiotic susceptibility of Listeria monocytogenes in THP-1 macrophages activated by gamma interferonComparative efficacies of antibiotics in a rat model of meningoencephalitis due to Listeria monocytogenes.Listeria monocytogenes possesses adhesins for fibronectinMutants of Listeria monocytogenes defective in In vitro invasion and cell-to-cell spreading still invade and proliferate in hepatocytes of neutropenic mice.Interaction of Listeria monocytogenes with human brain microvascular endothelial cells: an electron microscopic study.Identification of new genes involved in the virulence of Listeria monocytogenes by signature-tagged transposon mutagenesis.Critical role of neutrophils in eliminating Listeria monocytogenes from the central nervous system during experimental murine listeriosis.Inducible control of virulence gene expression in Listeria monocytogenes: temporal requirement of listeriolysin O during intracellular infectionAntibodies present in normal human serum inhibit invasion of human brain microvascular endothelial cells by Listeria monocytogenesIdentification of the agr locus of Listeria monocytogenes: role in bacterial virulenceExpression of the inlAB operon by Listeria monocytogenes is not required for entry into hepatic cells in vivoEther lipid vesicle-based antigens impart protection against experimental listeriosisAnimal models of listeriosis: a comparative review of the current state of the art and lessons learned.Lymphocytes serve as a reservoir for Listeria monocytogenes growth during infection of mice.The pore-forming toxin listeriolysin O is degraded by neutrophil metalloproteinase-8 and fails to mediate Listeria monocytogenes intracellular survival in neutrophils.Listeriolysin O as a strong immunogenic molecule for the development of new anti-tumor vaccinesIdentification of genes preferentially expressed by highly virulent piscine Streptococcus agalactiae upon interaction with macrophages.Listeria monocytogenes sigmaB contributes to invasion of human intestinal epithelial cells.Listeria monocytogenes-infected human dendritic cells: uptake and host cell response.ClpC ATPase is required for cell adhesion and invasion of Listeria monocytogenes.Human endothelial cell activation and mediator release in response to Listeria monocytogenes virulence factorsAntibodies against listerial protein 60 act as an opsonin for phagocytosis of Listeria monocytogenes by human dendritic cells.Colony-stimulating factor 1-dependent cells protect against systemic infection with Listeria monocytogenes but facilitate neuroinvasion.Host cell heparan sulfate proteoglycans mediate attachment and entry of Listeria monocytogenes, and the listerial surface protein ActA is involved in heparan sulfate receptor recognition.Adhesion to the host cell surface is sufficient to mediate Listeria monocytogenes entry into epithelial cells.
P2860
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P2860
Listeria monocytogenes infects human endothelial cells by two distinct mechanisms.
description
1995 nî lūn-bûn
@nan
1995 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Listeria monocytogenes infects human endothelial cells by two distinct mechanisms.
@ast
Listeria monocytogenes infects human endothelial cells by two distinct mechanisms.
@en
type
label
Listeria monocytogenes infects human endothelial cells by two distinct mechanisms.
@ast
Listeria monocytogenes infects human endothelial cells by two distinct mechanisms.
@en
prefLabel
Listeria monocytogenes infects human endothelial cells by two distinct mechanisms.
@ast
Listeria monocytogenes infects human endothelial cells by two distinct mechanisms.
@en
P2093
P2860
P1476
Listeria monocytogenes infects human endothelial cells by two distinct mechanisms
@en
P2093
D A Drevets
P A Campbell
R T Sawyer
T A Potter
P2860
P304
P407
P577
1995-11-01T00:00:00Z