High-mobility group box-1 impairs memory in mice through both toll-like receptor 4 and Receptor for Advanced Glycation End Products.
about
The Role of Toll-Like Receptor 4 in Infectious and Noninfectious InflammationBioenergetic dysfunction and inflammation in Alzheimer's disease: a possible connectionAmyloid β: one of three danger-associated molecules that are secondary inducers of the proinflammatory cytokines that mediate Alzheimer's diseaseHigh mobility group box 1/Toll-like receptor danger signaling increases brain neuroimmune activation in alcohol dependenceThe multiple faces of RAGE--opportunities for therapeutic intervention in aging and chronic diseaseElectroacupuncture Alleviates Postoperative Cognitive Dysfunction in Aged Rats by Inhibiting Hippocampal Neuroinflammation Activated via Microglia/TLRs Pathway.High-mobility group box-1 induces proinflammatory cytokines production of Kupffer cells through TLRs-dependent signaling pathway after burn injuryHMGB1 in health and disease.Ultraviolet light exposure stimulates HMGB1 release by keratinocytesModulation of Human Macrophage Responses to Mycobacterium tuberculosis by Silver Nanoparticles of Different Size and Surface Modification.Neuroprotective effects of toll-like receptor 4 antagonism in spinal cord cultures and in a mouse model of motor neuron degeneration.Biomarkers of Epileptogenesis: The Focus on Glia and Cognitive Dysfunctions.Immunity and inflammation in status epilepticus and its sequelae: possibilities for therapeutic application.The alarmin HMGB-1 influences healing outcomes in fetal skin woundsHMGB1 and thrombin mediate the blood-brain barrier dysfunction acting as biomarkers of neuroinflammation and progression to neurodegeneration in Alzheimer's disease.Potential of the angiotensin receptor blockers (ARBs) telmisartan, irbesartan, and candesartan for inhibiting the HMGB1/RAGE axis in prevention and acute treatment of stroke.Blocking toll-like receptor 2 and 4 signaling during a stressor prevents stress-induced priming of neuroinflammatory responses to a subsequent immune challenge.Systemic HMGB1 Neutralization Prevents Postoperative Neurocognitive Dysfunction in Aged Rats.Biotic/Abiotic Stress-Driven Alzheimer's Disease.Preclinical models of overwhelming sepsis implicate the neural system that encodes contextual fear memory.HMGB1 translocation and release mediate cigarette smoke-induced pulmonary inflammation in mice through a TLR4/MyD88-dependent signaling pathway.HMGB1 in development and diseases of the central nervous system.Finding a better drug for epilepsy: antiinflammatory targetsUltraviolet radiation-induced non-melanoma skin cancer: Regulation of DNA damage repair and inflammationThe brain at risk: the sepsis syndrome and lessons from preclinical experiments.Inflammation in Epileptic Encephalopathies.Targeting oxidative stress improves disease outcomes in a rat model of acquired epilepsy.Inflammation and endothelium response in epileptic patients: A case-control study.Inflammation and epilepsy: the foundations for a new therapeutic approach in epilepsy?Receptor for Advanced Glycation End Products and its Inflammatory Ligands are Upregulated in Amyotrophic Lateral Sclerosis.Not only a bad guy: potential proneurogenic role of the RAGE/NF-κB axis in Alzheimer's disease brain.HMGB1 regulates P-glycoprotein expression in status epilepticus rat brains via the RAGE/NF-κB signaling pathway.In-vivo evidence that high mobility group box 1 exerts deleterious effects in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model and Parkinson's disease which can be attenuated by glycyrrhizinThe effects of physical exercise on nonmotor symptoms and on neuroimmune RAGE network in experimental parkinsonism.Response to “HMGB1 Mediates Cognitive Impairment in Sepsis Survivors”.Identification of learning and memory genes in canine; promoter investigation and determining the selective pressure.HMGB1 mediates microglia activation via the TLR4/NF-κB pathway in coriaria lactone induced epilepsy.Serum high-mobility group box 1 protein correlates with cognitive decline after gastrointestinal surgery.Toll-like receptor 3 activation modulates hippocampal network excitability, via glial production of interferon-β.Oleoylethanolamide prevents neuroimmune HMGB1/TLR4/NF-kB danger signaling in rat frontal cortex and depressive-like behavior induced by ethanol binge administration.
P2860
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P2860
High-mobility group box-1 impairs memory in mice through both toll-like receptor 4 and Receptor for Advanced Glycation End Products.
description
2011 nî lūn-bûn
@nan
2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
High-mobility group box-1 impa ...... vanced Glycation End Products.
@ast
High-mobility group box-1 impa ...... vanced Glycation End Products.
@en
type
label
High-mobility group box-1 impa ...... vanced Glycation End Products.
@ast
High-mobility group box-1 impa ...... vanced Glycation End Products.
@en
prefLabel
High-mobility group box-1 impa ...... vanced Glycation End Products.
@ast
High-mobility group box-1 impa ...... vanced Glycation End Products.
@en
P2093
P2860
P1476
High-mobility group box-1 impa ...... vanced Glycation End Products.
@en
P2093
Andréy Mazarati
Mattia Maroso
Mirjana Carli
Valentina Iori
P2860
P304
P356
10.1016/J.EXPNEUROL.2011.08.012
P407
P577
2011-08-22T00:00:00Z