Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease - Wikidata - awgldk - TriplyDB

Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease

Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease

http://www.wikidata.org/entity/Q35452032

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PI3K Signaling in Normal B Cells and Chronic Lymphocytic Leukemia (CLL)Role of Tumor Necrosis Factor Superfamily in Neuroinflammation and Autoimmunity Regulation of Aicda expression and AID activity Early Transcriptional Signatures of the Immune Response to a Live Attenuated Tetravalent Dengue Vaccine Candidate in Non-human Primates Potent anti-tumor response by targeting B cell maturation antigen (BCMA) in a mouse model of multiple myeloma Distinct plasma immune signatures in ME/CFS are present early in the course of illness Targeting F box protein Fbxo3 to control cytokine-driven inflammation B cell receptor signal transduction in the GC is short-circuited by high phosphatase activity Murine BAFF expression is up-regulated by estrogen and interferons: implications for sex bias in the development of autoimmunity.Regulatory roles of the tumor necrosis factor receptor BCMA.The Concise Guide to PHARMACOLOGY 2013/14: catalytic receptors.The function of BAFF on T helper cells in autoimmunity Early-onset primary antibody deficiency resembling common variable immunodeficiency challenges the diagnosis of Wiedeman-Steiner and Roifman syndromes.Alteration of naïve and memory B-cell subset in chronic graft-versus-host disease patients after treatment with mesenchymal stromal cells.IKK-induced NF-κB1 p105 proteolysis is critical for B cell antibody responses to T cell-dependent antigen.Role of sphingosine kinase 1 and sphingosine-1-phosphate in CD40 signaling and IgE class switching.FlowMax: A Computational Tool for Maximum Likelihood Deconvolution of CFSE Time Courses.Indolent small intestinal CD4+ T-cell lymphoma is a distinct entity with unique biologic and clinical features.Potential blood-based markers of celiac disease OSU-T315: a novel targeted therapeutic that antagonizes AKT membrane localization and activation of chronic lymphocytic leukemia cells IFN-β treatment requires B cells for efficacy in neuroautoimmunity.Scaffold protein JLP is critical for CD40 signaling in B lymphocytes.Impact of acute malaria on pre-existing antibodies to viral and vaccine antigens in mice and humans DNA vaccine molecular adjuvants SP-D-BAFF and SP-D-APRIL enhance anti-gp120 immune response and increase HIV-1 neutralizing antibody titers BAFF activation of the ERK5 MAP kinase pathway regulates B cell survival.Different Donor Cell Culture Methods Can Influence the Developmental Ability of Cloned Sheep Embryos.Comparing the Primary and Recall Immune Response Induced by a New EV71 Vaccine Using Systems Biology Approaches.The B Cell-Stimulatory Cytokines BLyS and APRIL Are Elevated in Human Periodontitis and Are Required for B Cell-Dependent Bone Loss in Experimental Murine Periodontitis.Gender-Specific Mechanisms Underlying the Amelioration of High-Fat Diet-Induced Glucose Intolerance in B-Cell-Activating Factor Deficient Mice Molecular signature in HCV-positive lymphomas Deleted in Breast Cancer 1 Suppresses B Cell Activation through RelB and Is Regulated by IKKα Phosphorylation.Targeting neddylation induces DNA damage and checkpoint activation and sensitizes chronic lymphocytic leukemia B cells to alkylating agents PI3K signalling in B- and T-lymphocytes: new developments and therapeutic advances SNP-SNP interaction analysis of NF-κB signaling pathway on breast cancer survival.Immune Cell Identity: Perspective from a Palimpsest B-cell survival and development controlled by the coordination of NF-κB family members RelB and cRel.BAFF/APRIL system in pediatric OMS: relation to severity, neuroinflammation, and immunotherapy The BAFF receptor transduces survival signals by co-opting the B cell receptor signaling pathway.B-cell maturation antigen, a proliferation-inducing ligand, and B-cell activating factor are candidate mediators of spinal cord injury-induced autoimmunity Signals via the adaptor MyD88 in B cells and DCs make distinct and synergistic contributions to immune activation and tissue damage in lupus

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Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease

http://www.wikidata.org/entity/Q35452032

description

2011 nî lūn-bûn

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2011 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease

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Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease

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Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease

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Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease

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Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease

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Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease

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J.1600-065X.2011.01067.X

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Immunological Reviews

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Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease

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Ana V Miletic

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Julia Jellusova

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Robert C Rickert

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P2860

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

BAFF, a novel ligand of the tumor necrosis factor family, stimulates B cell growth

AFX-like Forkhead transcription factors mediate cell-cycle regulation by Ras and PKB through p27kip1

TACI and BCMA are receptors for a TNF homologue implicated in B-cell autoimmune disease

TACI is a TRAF-interacting receptor for TALL-1, a tumor necrosis factor family member involved in B cell regulation

B cell maturation protein is a receptor for the tumor necrosis factor family member TALL-1

The transmembrane activator TACI triggers immunoglobulin class switching by activating B cells through the adaptor MyD88

Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function

Multiple nuclear factors interact with the immunoglobulin enhancer sequences

Phosphorylation of the CARMA1 linker controls NF-kappaB activation

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115-133

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10.1111/J.1600-065X.2011.01067.X

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1

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244

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2011-11-01T00:00:00Z

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