Resolving TRPV1- and TNF-α-mediated spinal cord synaptic plasticity and inflammatory pain with neuroprotectin D1.
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Biological Roles of Resolvins and Related Substances in the Resolution of PainIonotropic glutamate receptors and voltage-gated Ca²⁺ channels in long-term potentiation of spinal dorsal horn synapses and pain hypersensitivityMetaplasticity and behavior: how training and inflammation affect plastic potential within the spinal cord and recovery after injuryMicroglia and spinal cord synaptic plasticity in persistent painInhibition of mechanical allodynia in neuropathic pain by TLR5-mediated A-fiber blockade.Resolution phase lipid mediators of inflammation: agonists of resolution.Modulation of the Activities of Neuronal Ion Channels by Fatty Acid-Derived Pro-ResolventsEmerging targets in neuroinflammation-driven chronic painLipid mediators in the resolution of inflammationThe resolution code of acute inflammation: Novel pro-resolving lipid mediators in resolutionSpecialized pro-resolving mediators: endogenous regulators of infection and inflammationGlial tumor necrosis factor alpha (TNFα) generates metaplastic inhibition of spinal learningProtectins and maresins: New pro-resolving families of mediators in acute inflammation and resolution bioactive metabolomePro-Resolving Lipid Mediators (SPMs) and Their Actions in Regulating miRNA in Novel Resolution Circuits in InflammationCurcumin alleviates lumbar radiculopathy by reducing neuroinflammation, oxidative stress and nociceptive factors.Growth factors in synaptic function.Pro-resolving lipid mediators are leads for resolution physiology.Microglial TNFα Induces COX2 and PGI2 Synthase Expression in Spinal Endothelial Cells during Neuropathic Pain.β-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain.Epigenetic modification of DRG neuronal gene expression subsequent to nerve injury: etiological contribution to complex regional pain syndromes (Part I).Spinal inhibition of p38 MAP kinase reduces inflammatory and neuropathic pain in male but not female mice: Sex-dependent microglial signaling in the spinal cord.Pathological pain and the neuroimmune interface.Connexin-43 induces chemokine release from spinal cord astrocytes to maintain late-phase neuropathic pain in mice.Chemokine CXCL1 enhances inflammatory pain and increases NMDA receptor activity and COX-2 expression in spinal cord neurons via activation of CXCR2.Kainate receptor activation induces glycine receptor endocytosis through PKC deSUMOylationTRPV1 antagonist attenuates postoperative hypersensitivity by central and peripheral mechanisms.Paclitaxel induces acute pain via directly activating toll like receptor 4Involvement of CX3CL1/CX3CR1 signaling in spinal long term potentiation.Resolvin D2 is a potent endogenous inhibitor for transient receptor potential subtype V1/A1, inflammatory pain, and spinal cord synaptic plasticity in mice: distinct roles of resolvin D1, D2, and E1.Identification of lncRNA expression profile in the spinal cord of mice following spinal nerve ligation-induced neuropathic pain.Emerging role of Toll-like receptors in the control of pain and itch.TLR3 deficiency impairs spinal cord synaptic transmission, central sensitization, and pruritus in mice.Central nociceptive sensitization vs. spinal cord training: opposing forms of plasticity that dictate function after complete spinal cord injuryTranscriptional expression of voltage-gated Na⁺ and voltage-independent K⁺ channels in the developing rat superficial dorsal horn.Curcumin attenuates diabetic neuropathic pain by downregulating TNF-α in a rat modelLipoxin A4 inhibits microglial activation and reduces neuroinflammation and neuropathic pain after spinal cord hemisection.Resolvin E1 inhibits neuropathic pain and spinal cord microglial activation following peripheral nerve injury.Neuroprotectin D1 restores corneal nerve integrity and function after damage from experimental surgery.Loss of NR1 subunit of NMDARs in primary sensory neurons leads to hyperexcitability and pain hypersensitivity: involvement of Ca(2+)-activated small conductance potassium channelsFatting the brain: a brief of recent research
P2860
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P2860
Resolving TRPV1- and TNF-α-mediated spinal cord synaptic plasticity and inflammatory pain with neuroprotectin D1.
description
2011 nî lūn-bûn
@nan
2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Resolving TRPV1- and TNF-α-med ...... y pain with neuroprotectin D1.
@ast
Resolving TRPV1- and TNF-α-med ...... y pain with neuroprotectin D1.
@en
type
label
Resolving TRPV1- and TNF-α-med ...... y pain with neuroprotectin D1.
@ast
Resolving TRPV1- and TNF-α-med ...... y pain with neuroprotectin D1.
@en
prefLabel
Resolving TRPV1- and TNF-α-med ...... y pain with neuroprotectin D1.
@ast
Resolving TRPV1- and TNF-α-med ...... y pain with neuroprotectin D1.
@en
P2093
P2860
P1476
Resolving TRPV1- and TNF-α-med ...... y pain with neuroprotectin D1.
@en
P2093
Charles N Serhan
Chul-Kyu Park
Zhen-Zhong Xu
P2860
P304
15072-15085
P356
10.1523/JNEUROSCI.2443-11.2011
P407
P50
P577
2011-10-01T00:00:00Z