High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection - Wikidata - awgldk - TriplyDB

High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection

High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection

http://www.wikidata.org/entity/Q35463737

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Sex work and HIV in Cambodia: trajectories of risk and disease in two cohorts of high-risk young women in Phnom Penh, Cambodia.HIV and HCV activate the inflammasome in monocytes and macrophages via endosomal Toll-like receptors without induction of type 1 interferon.Innate immune cell networking in hepatitis C virus infection Natural Killer Cells in Viral Hepatitis.Interferon γ-induced protein 10 kinetics in treatment-naive versus treatment-experienced patients receiving interferon-free therapy for hepatitis C virus infection: implications for the innate immune response.Activation and evasion of antiviral innate immunity by hepatitis C virus.Monocytes activate natural killer cells via inflammasome-induced interleukin 18 in response to hepatitis C virus replication.Immunologic Predictors of Liver Transplantation Outcomes in HIV-HCV Co-Infected Persons.Spontaneous clearance of primary acute hepatitis C virus infection correlated with high initial viral RNA level and rapid HVR1 evolution.Temporal dynamics of inflammatory cytokines/chemokines during sofosbuvir and ribavirin therapy for genotype 2 and 3 hepatitis C infection.Immunity and hepatitis C: a review.Anti-inflammatory cytokines, pro-fibrogenic chemokines and persistence of acute HCV infection Hepatitis C virus induces interleukin-1β (IL-1β)/IL-18 in circulatory and resident liver macrophages.Hepatitis regulation by the inflammasome signaling pathway.HCV genomic RNA activates the NLRP3 inflammasome in human myeloid cells.Dose of Retroviral Infection Determines Induction of Antiviral NK Cell Responses.Intra-hepatic Depletion of Mucosal Associated Invariant T cells in Hepatitis C Virus-induced Liver Inflammation.Genetic basis for variation in plasma IL-18 levels in persons with chronic hepatitis C virus and human immunodeficiency virus-1 infections.Systemic Elevation of Proinflammatory Interleukin 18 in HIV/HCV Coinfection versus HIV or HCV Monoinfection.Interleukin-18 increases TLR4 and mannose receptor expression and modulates cytokine production in human monocytes.Human immunodeficiency virus and liver disease: A comprehensive update.

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P2860

High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection

http://www.wikidata.org/entity/Q35463737

description

2011 nî lūn-bûn

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2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection

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High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection

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High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection

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High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection

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High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection

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High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection

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P1433

The Journal of Infectious Diseases

P1476

High plasma interleukin-18 levels mark the acute phase of hepatitis C virus infection

@en

P2093

Andrea L Cox

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David L Thomas

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Jordana S Levine

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Michael A Chattergoon

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Rachel Latanich

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William O Osburn

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P2860

Genetic variation in IL28B and spontaneous clearance of hepatitis C virus

Large-scale candidate gene analysis of spontaneous clearance of hepatitis C virus

DNA microarray analysis of chimpanzee liver during acute resolving hepatitis C virus infection.

Regulatory Polymorphisms in the Interleukin‐18 Promoter Are Associated with Hepatitis C Virus Clearance

Elevation of interleukin-18 in chronic hepatitis C: implications for hepatitis C virus pathogenesis

Genetic variation in IL28B is associated with chronic hepatitis C and treatment failure: a genome-wide association study

Cloning of a new cytokine that induces IFN-gamma production by T cells

Central roles of NLRs and inflammasomes in viral infection

Diagnosis, management, and treatment of hepatitis C: an update

Spontaneous control of primary hepatitis C virus infection and immunity against persistent reinfection.

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1730-1740

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scholarly article

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10.1093/INFDIS/JIR642

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11

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204

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51704245

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21984735

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3203233

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