Sympathetic control of bone mass regulated by osteopontin. - Wikidata - awgldk - TriplyDB

Sympathetic control of bone mass regulated by osteopontin.

Sympathetic control of bone mass regulated by osteopontin.

http://www.wikidata.org/entity/Q35470587

about

Boning up on autophagy: the role of autophagy in skeletal biology Impaired Vestibular Function and Low Bone Mineral Density: Data from the Baltimore Longitudinal Study of Aging Matricellular proteins in the trabecular meshwork: review and update.Osteoblast-specific expression of the fibrous dysplasia (FD)-causing mutation Gsα(R201C) produces a high bone mass phenotype but does not reproduce FD in the mouse.Current Protocols in Mouse Biology.Impaired osteoblast and osteoclast function characterize the osteoporosis of Snyder - Robinson syndrome.Relationship of sympathetic activity to bone microstructure, turnover, and plasma osteopontin levels in women.High serum osteopontin levels are associated with low bone mineral density in postmenopausal women.Revisiting the Debate: Does Exercise Build Strong Bones in the Mature and Senescent Skeleton?Stability of mRNA influences osteoporotic bone mass via CNOT3.Sodium selenate treatment mitigates reduction of bone volume following traumatic brain injury in rats.β2-adrenergic receptor control of endosomal PTH receptor signaling via Gβγ.Probing the response of human osteoblasts following exposure to sympathetic neuron-like PC-12 cells in a 3D coculture model.β₂ adrenergic receptor activation suppresses bone morphogenetic protein (BMP)-induced alkaline phosphatase expression in osteoblast-like MC3T3E1 cells.PTH regulates β2-adrenergic receptor expression in osteoblast-like MC3T3-E1 cells.Osteopontin contributes to TGF-β1 mediated hepatic stellate cell activation.Cathepsin K Deficiency Suppresses Disuse-Induced Bone Loss.Profilin Expression Is Regulated by Bone Morphogenetic Protein (BMP) in Osteoblastic Cells.β-Adrenergic receptor antagonists and fracture risk: a meta-analysis of selectivity, gender, and site-specific effects.Mast Cell Mediators Inhibit Osteoblastic Differentiation and Extracellular Matrix Mineralization.Adrenaline inhibits osteogenesis via repressing miR-21 expression.Co(II)-mediated effects of plain and plasma immersion ion implanted cobalt-chromium alloys on the osteogenic differentiation of human mesenchymal stem cells.Poligoni Multiflori Radix enhances osteoblast formation and reduces osteoclast differentiation.

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Sympathetic control of bone mass regulated by osteopontin.

http://www.wikidata.org/entity/Q35470587

description

2011 nî lūn-bûn

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2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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name

Sympathetic control of bone mass regulated by osteopontin.

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Sympathetic control of bone mass regulated by osteopontin.

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Sympathetic control of bone mass regulated by osteopontin.

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Sympathetic control of bone mass regulated by osteopontin.

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Sympathetic control of bone mass regulated by osteopontin.

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Sympathetic control of bone mass regulated by osteopontin.

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PNAS.1109402108

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Sympathetic control of bone mass regulated by osteopontin.

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P2093

David T Denhardt

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Gerard Karsenty

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Hiroaki Hemmi

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Hisashi Kurosawa

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Kathryn Wang

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Kazuo Kaneko

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Masaki Noda

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Masashi Nagao

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Ryo Hanyu

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Shu Takeda

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P2860

Pathogenesis of osteoporosis: concepts, conflicts, and prospects

Alternative translation of osteopontin generates intracellular and secreted isoforms that mediate distinct biological activities in dendritic cells

Osteopontin as a means to cope with environmental insults: regulation of inflammation, tissue remodeling, and cell survival

Osteopontin: role in immune regulation and stress responses

Targeted disruption of the beta2 adrenergic receptor gene

Postmenopausal osteoporosis as a failure of bone's adaptation to functional loading: a hypothesis.

Sustained cyclic AMP production by parathyroid hormone receptor endocytosis.

Mechanical strain and bone cell function: a review.

Retromer terminates the generation of cAMP by internalized PTH receptors.

Enhancement of osteoclastic bone resorption and suppression of osteoblastic bone formation in response to reduced mechanical stress do not occur in the absence of osteopontin.

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17767-17772

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10.1073/PNAS.1109402108

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43

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108

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Jean-Pierre Vilardaga

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2011-10-11T00:00:00Z

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51709916

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21990347

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3203767

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