The high-mobility group box-1 nuclear factor mediates retinal injury after ischemia reperfusion.
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Genetic ablation of Pannexin1 protects retinal neurons from ischemic injuryThe role of Toll-like receptors in retinal ischemic diseasesRetinal ganglion cell (RGC) programmed necrosis contributes to ischemia-reperfusion-induced retinal damage.Ischemia-reperfusion injury of the retina is linked to necroptosis via the ERK1/2-RIP3 pathway.Tumor necrosis factor-alpha mediates activation of NF-κB and JNK signaling cascades in retinal ganglion cells and astrocytes in opposite ways.The involvement of high mobility group 1 cytokine and phospholipases A2 in diabetic retinopathy.High-mobility group box 1 protein is implicated in advanced glycation end products-induced vascular endothelial growth factor A production in the rat retinal ganglion cell line RGC-5.HMGB1 promotes the activation of NLRP3 and caspase-8 inflammasomes via NF-κB pathway in acute glaucoma.The role of high mobility group box 1 (HMGB-1) in the diabetic retinopathy inflammation and apoptosis.Inhibition of HMGB1 protects the retina from ischemia-reperfusion, as well as reduces insulin resistance proteinsNeuronal NAD(P)H oxidases contribute to ROS production and mediate RGC death after ischemiaHigh-mobility group box-1 induces decreased brain-derived neurotrophic factor-mediated neuroprotection in the diabetic retina.Ethanol extracts from Portulaca oleracea L. attenuated ischemia/reperfusion induced rat neural injury through inhibition of HMGB1 induced inflammation.The Expression of HMGB1 in Bone Marrow MSCs Is Upregulated by Hypoxia with Regulatory Effects on the Apoptosis and Adhesion.Putative role of protein kinase C in neurotoxic inflammation mediated by extracellular heat shock protein 70 after ischemia-reperfusion.Prothymosin-alpha preconditioning activates TLR4-TRIF signaling to induce protection of ischemic retina.Protective effects on the retina after ranibizumab treatment in an ischemia modelA Novel Mouse Model of Traumatic Optic Neuropathy Using External Ultrasound Energy to Achieve Focal, Indirect Optic Nerve Injury.Cellular mechanisms of high mobility group 1 (HMGB-1) protein action in the diabetic retinopathy.P2X7 receptor antagonist protects retinal ganglion cells by inhibiting microglial activation in a rat chronic ocular hypertension model.Epac1 protects the retina against ischemia/reperfusion-induced neuronal and vascular damage
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P2860
The high-mobility group box-1 nuclear factor mediates retinal injury after ischemia reperfusion.
description
2011 nî lūn-bûn
@nan
2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
The high-mobility group box-1 ...... ry after ischemia reperfusion.
@ast
The high-mobility group box-1 ...... ry after ischemia reperfusion.
@en
type
label
The high-mobility group box-1 ...... ry after ischemia reperfusion.
@ast
The high-mobility group box-1 ...... ry after ischemia reperfusion.
@en
prefLabel
The high-mobility group box-1 ...... ry after ischemia reperfusion.
@ast
The high-mobility group box-1 ...... ry after ischemia reperfusion.
@en
P2093
P2860
P356
P1476
The high-mobility group box-1 ...... ury after ischemia reperfusion
@en
P2093
Andrea Rachelle C Santos
Eleut Hernandez
Galina Dvoriantchikova
Jeff Grant
P2860
P304
P356
10.1167/IOVS.11-7793
P407
P577
2011-09-09T00:00:00Z