Differential requirements for clathrin endocytic pathway components in cellular entry by Ebola and Marburg glycoprotein pseudovirions. - Wikidata - awgldk - TriplyDB

Differential requirements for clathrin endocytic pathway components in cellular entry by Ebola and Marburg glycoprotein pseudovirions.

Differential requirements for clathrin endocytic pathway components in cellular entry by Ebola and Marburg glycoprotein pseudovirions.

http://www.wikidata.org/entity/Q35533766

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Induction of Cell-Cell Fusion by Ebola Virus Glycoprotein: Low pH Is Not a Trigger Cargo recognition in clathrin-mediated endocytosis Ebola virus entry: a curious and complex series of events.Marburg virus glycoprotein GP2: pH-dependent stability of the ectodomain α-helical bundle Forty-five years of Marburg virus research.Endocytic pathways involved in filovirus entry: advances, implications and future directions.Cellular factors implicated in filovirus entry Enveloped viruses disable innate immune responses in dendritic cells by direct activation of TAM receptors Arrestins in host-pathogen interactions.Rab1a regulates sorting of early endocytic vesicles.Anticancer kinase inhibitors impair intracellular viral trafficking and exert broad-spectrum antiviral effects.Filovirus entry: a novelty in the viral fusion world.Multicohort analysis reveals baseline transcriptional predictors of influenza vaccination responses.Ebola virus entry into host cells: identifying therapeutic strategies.Forty Years of Ebolavirus Molecular Biology: Understanding a Novel Disease Agent Through the Development and Application of New Technologies.Host Factors Involved in Ebola Virus Replication.A survey on Ebola genome and current trends in computational research on the Ebola virus.Review of computational methods for virus-host protein interaction prediction: a case study on novel Ebola-human interactions.Feasibility and biological rationale of repurposing sunitinib and erlotinib for dengue treatment.

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P2860

Differential requirements for clathrin endocytic pathway components in cellular entry by Ebola and Marburg glycoprotein pseudovirions.

http://www.wikidata.org/entity/Q35533766

description

2011 nî lūn-bûn

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2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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John A T Young

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Suchita Bhattacharyya

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Thomas J Hope

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P2860

Role of the AP2 beta-appendage hub in recruiting partners for clathrin-coated vesicle assembly

Cellular entry of ebola virus involves uptake by a macropinocytosis-like mechanism and subsequent trafficking through early and late endosomes

The EH and SH3 domain Ese proteins regulate endocytosis by linking to dynamin and Eps15.

HIV enters cells via endocytosis and dynamin-dependent fusion with endosomes

HIP1 functions in clathrin-mediated endocytosis through binding to clathrin and adaptor protein 2

T-cell immunoglobulin and mucin domain 1 (TIM-1) is a receptor for Zaire Ebolavirus and Lake Victoria Marburgvirus

Ebola virus uses clathrin-mediated endocytosis as an entry pathway.

The adaptor protein Dab2 sorts LDL receptors into coated pits independently of AP-2 and ARH

The Tyro3 receptor kinase Axl enhances macropinocytosis of Zaire ebolavirus

ARH is a modular adaptor protein that interacts with the LDL receptor, clathrin, and AP-2

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10.1016/J.VIROL.2011.07.018

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