Epigenetic suppression of GAD65 expression mediates persistent pain
about
Spatial and temporal pattern of changes in the number of GAD65-immunoreactive inhibitory terminals in the rat superficial dorsal horn following peripheral nerve injuryTranscriptional control of Gad2Epigenetic regulation of persistent painTelomeres and epigenetics: potential relevance to chronic painEpigenetic mechanisms of chronic painEpigenetic suppression of neuroligin 1 underlies amyloid-induced memory deficiencyImmunocytochemical identification of electroneutral Na⁺-coupled HCO₃⁻ transporters in freshly dissociated mouse medullary raphé neuronsGenome-wide expression analysis of Ptf1a- and Ascl1-deficient mice reveals new markers for distinct dorsal horn interneuron populations contributing to nociceptive reflex plasticityIdentification of molecular signatures specific for distinct cranial sensory ganglia in the developing chick.Analgesia induced by the epigenetic drug, L-acetylcarnitine, outlasts the end of treatment in mouse models of chronic inflammatory and neuropathic pain.MeCP2 repression of G9a in regulation of pain and morphine reward.Glutamic acid decarboxylase 65: a link between GABAergic synaptic plasticity in the lateral amygdala and conditioned fear generalizationPersistent pain facilitates response to morphine reward by downregulation of central amygdala GABAergic function.Possible Loss of GABAergic Inhibition in Mice With Induced Adenomyosis and Treatment With Epigallocatechin-3-Gallate Attenuates the Loss With Improved HyperalgesiaPersistent inflammation-induced up-regulation of brain-derived neurotrophic factor (BDNF) promotes synaptic delivery of α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor GluA1 subunits in descending pain modulatory circuits.Alpha-7 Nicotinic Receptor Signaling Pathway Participates in the Neurogenesis Induced by ChAT-Positive Neurons in the Subventricular Zone.Epigenetic mechanisms in migraine: a promising avenue?Peripheral nerve injury is associated with chronic, reversible changes in global DNA methylation in the mouse prefrontal cortexSex-related differences of cortical thickness in patients with chronic abdominal pain.Persistent pain maintains morphine-seeking behavior after morphine withdrawal through reduced MeCP2 repression of GluA1 in rat central amygdala.Neuroplasticity underlying the comorbidity of pain and depressionEpigenetic regulation of chronic painEpigenetics in the perioperative period.A Transgenic Mouse Line Expressing the Red Fluorescent Protein tdTomato in GABAergic Neurons.Activity-Dependent Bidirectional Regulation of GAD Expression in a Homeostatic Fashion Is Mediated by BDNF-Dependent and Independent PathwaysSignaling cascades for δ-opioid receptor-mediated inhibition of GABA synaptic transmission and behavioral antinociception.HDAC4 is required for inflammation-associated thermal hypersensitivity.Epigenetic upregulation of metabotropic glutamate receptor 2 in the spinal cord attenuates oestrogen-induced visceral hypersensitivity.Plasticity of DNA methylation in a nerve injury model of pain.G9a is essential for epigenetic silencing of K(+) channel genes in acute-to-chronic pain transition.Current challenges in translational pain research.Upregulation of nerve growth factor in central amygdala increases sensitivity to opioid rewardEpigenetics and the transition from acute to chronic pain.MSC Transplantation Improves Osteopenia via Epigenetic Regulation of Notch Signaling in Lupus.p300 exerts an epigenetic role in chronic neuropathic pain through its acetyltransferase activity in rats following chronic constriction injury (CCI).Enhanced binding capability of nuclear factor-κB with demethylated P2X3 receptor gene contributes to cancer pain in rats.Assessment of pain and itch behavior in a mouse model of neurofibromatosis type 1.Genes and epigenetic processes as prospective pain targetsCentral amygdala GluA1 facilitates associative learning of opioid reward.Improvements in impaired GABA and GAD65/67 production in the spinal dorsal horn contribute to exercise-induced hypoalgesia in a mouse model of neuropathic pain
P2860
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P2860
Epigenetic suppression of GAD65 expression mediates persistent pain
description
2011 nî lūn-bûn
@nan
2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Epigenetic suppression of GAD65 expression mediates persistent pain
@ast
Epigenetic suppression of GAD65 expression mediates persistent pain
@en
type
label
Epigenetic suppression of GAD65 expression mediates persistent pain
@ast
Epigenetic suppression of GAD65 expression mediates persistent pain
@en
prefLabel
Epigenetic suppression of GAD65 expression mediates persistent pain
@ast
Epigenetic suppression of GAD65 expression mediates persistent pain
@en
P2093
P2860
P356
P1433
P1476
Epigenetic suppression of GAD65 expression mediates persistent pain
@en
P2093
You-Qing Cai
Zhizhong Z Pan
P2860
P2888
P304
P356
10.1038/NM.2442
P407
P577
2011-10-09T00:00:00Z
P5875
P6179
1011296762