Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse
about
Lipids and the endothelium: bidirectional interactionsInteraction between nitric oxide signaling and gap junctions: effects on vascular functionMethylglyoxal modulates endothelial nitric oxide synthase-associated functions in EA.hy926 endothelial cells3',4'-Dihydroxyflavonol reduces superoxide and improves nitric oxide function in diabetic rat mesenteric arteries.Role of arachidonic acid lipoxygenase metabolites in acetylcholine-induced relaxations of mouse arteries.Metformin restores endothelial function in aorta of diabetic ratsImprovement of endothelium-dependent vasodilations by SKA-31 and SKA-20, activators of small- and intermediate-conductance Ca2+ -activated K+ -channels.Attenuation of atherosclerotic lesions in diabetic apolipoprotein E-deficient mice using gene silencing of macrophage migration inhibitory factorPharmacological activation of KCa3.1/KCa2.3 channels produces endothelial hyperpolarization and lowers blood pressure in conscious dogsPrunella vulgaris suppresses HG-induced vascular inflammation via Nrf2/HO-1/eNOS activationHyperglycemia and endothelial dysfunction in atherosclerosis: lessons from type 1 diabetes.Inactivation of Endothelial Small/Intermediate Conductance of Calcium-Activated Potassium Channels Contributes to Coronary Arteriolar Dysfunction in Diabetic Patients.Downregulation of connexin40 is associated with coronary endothelial cell dysfunction in streptozotocin-induced diabetic mice.Aldosterone impairs vascular reactivity by decreasing glucose-6-phosphate dehydrogenase activity.Endothelium-dependent hyperpolarizations: past beliefs and present facts.Calcium-activated potassium channels and endothelial dysfunction: therapeutic options?Endothelial Ca+-activated K+ channels in normal and impaired EDHF-dilator responses--relevance to cardiovascular pathologies and drug discoveryPCB-induced endothelial cell dysfunction: role of poly(ADP-ribose) polymerase.Reduction of large-conductance Ca²(+) -activated K(+) channel with compensatory increase of nitric oxide in insulin resistant rats.K(Ca)2 and k(ca)3 channels in learning and memory processes, and neurodegeneration.Role of the potassium channel KCa3.1 in diabetic nephropathy.Enhanced K(+)-channel-mediated endothelium-dependent local and conducted dilation of small mesenteric arteries from ApoE(-/-) mice.Methylglyoxal in Metabolic Disorders: Facts, Myths, and Promises.Synthesis and Biological Activity of Isoflavone Derivatives from Chickpea as Potent Anti-Diabetic Agents.Compensatory Vasodilator Mechanisms in the Ophthalmic Artery of Endothelial Nitric Oxide Synthase Gene Knockout Mice.Phosphatidylinositol 3-kinase-δ up-regulates L-type Ca2+ currents and increases vascular contractility in a mouse model of type 1 diabetes.Endothelium-derived hyperpolarizing factor and diabetes.BMP4 is increased in the aortas of diabetic ApoE knockout mice and enhances uptake of oxidized low density lipoprotein into peritoneal macrophages.Glucose enhances expression of TRPC1 and calcium entry in endothelial cells.Relationship among superoxide-related enzyme, PPARs, and endothelium-dependent relaxation in murine aortas previously organ-cultured in high-glucose conditions.Impaired small-conductance Ca2+-activated K+ channel-dependent EDHF responses in Type II diabetic ZDF rats.The Sulforaphane and pyridoxamine supplementation normalize endothelial dysfunction associated with type 2 diabetes.Effects of the superoxide dismutase mimetic tempol on impaired endothelium-dependent and endothelium-independent relaxations in type II diabetic rats.Altered TP receptor function in isolated, perfused kidneys of nondiabetic and diabetic ApoE-deficient mice.Type 1 diabetes and hypercholesterolaemia reveal the contribution of endothelium-derived hyperpolarizing factor to endothelium-dependent relaxation of the rat aorta.Decreased coronary arteriolar response to KCa channel opener after cardioplegic arrest in diabetic patients.TLR2 knockout protects against diabetes-mediated changes in cerebral perfusion and cognitive deficits.Advanced glycation end products impair K(Ca)3.1- and K(Ca)2.3-mediated vasodilatation via oxidative stress in rat mesenteric arteries.Hyperglycaemia disrupts conducted vasodilation in the resistance vasculature of db/db mice.Relaxin as a Therapeutic Target for the Cardiovascular Complications of Diabetes.
P2860
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P2860
Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse
description
2005 nî lūn-bûn
@nan
2005 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse
@ast
Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse
@en
type
label
Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse
@ast
Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse
@en
prefLabel
Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse
@ast
Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse
@en
P2093
P2860
P356
P1476
Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse
@en
P2093
Jacqueline Martin
Julianne Reid
Michael Hashem
William B Wiehler
Winnie Lau
P2860
P304
P356
10.1038/SJ.BJP.0706417
P407
P577
2005-12-01T00:00:00Z