NADPH oxidase DUOX1 promotes long-term persistence of oxidative stress after an exposure to irradiation. - Wikidata - awgldk - TriplyDB

NADPH oxidase DUOX1 promotes long-term persistence of oxidative stress after an exposure to irradiation.

NADPH oxidase DUOX1 promotes long-term persistence of oxidative stress after an exposure to irradiation.

http://www.wikidata.org/entity/Q35549377

about

Genetic disorders coupled to ROS deficiency The immune network in thyroid cancer DUOX1 silencing in lung cancer promotes EMT, cancer stem cell characteristics and invasive properties NADPH oxidase in brain injury and neurodegenerative disorders Oxidative stress by inorganic nanoparticles.Detection of Reactive Oxygen Species in Cells Undergoing Oncogene-Induced Senescence.NADPH Oxidase NOX4 Is a Critical Mediator of BRAFV600E-Induced Downregulation of the Sodium/Iodide Symporter in Papillary Thyroid Carcinomas.Canonical Transient Receptor Potential 6 Channel: A New Target of Reactive Oxygen Species in Renal Physiology and Pathology.NADPH oxidases: new actors in thyroid cancer?Radiation exposure and thyroid cancer: a review.Interferon gamma/NADPH oxidase defense system in immunity and cancer.Paradoxical roles of dual oxidases in cancer biology.Overexpression of Interleukin-4 in the Thyroid of Transgenic Mice Upregulates the Expression of Duox1 and the Anion Transporter Pendrin.N-Acetyl-L-cysteine protects thyroid cells against DNA damage induced by external and internal irradiation.NADPH oxidase 4 mediates ROS production in radiation-induced senescent cells and promotes migration of inflammatory cells.Reduction-oxidation (redox) system in radiation-induced normal tissue injury: molecular mechanisms and implications in radiation therapeutics.TARGETED AND OFF-TARGET (BYSTANDER AND ABSCOPAL) EFFECTS OF RADIATION THERAPY: REDOX MECHANISMS AND RISK-BENEFIT ANALYSIS.In vitro stimulatory effect of N-acetyl tryptophan-glucopyranoside against gamma radiation induced immunosuppression.DUAL OXIDASE: A NOVEL THERAPEUTIC TARGET IN ALLERGIC DISEASE.Noninvasive assessment of cutaneous alterations in mice exposed to whole body gamma irradiation using optical imaging techniques.NKX2.5 is expressed in papillary thyroid carcinomas and regulates differentiation in thyroid cells.DUOX1 Silencing in Mammary Cell Alters the Response to Genotoxic Stress.The defense and signaling role of NADPH oxidases in eukaryotic cells : Review

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P2860

NADPH oxidase DUOX1 promotes long-term persistence of oxidative stress after an exposure to irradiation.

http://www.wikidata.org/entity/Q35549377

description

2015 nî lūn-bûn

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2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2015 թվականի ապրիլին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

NADPH oxidase DUOX1 promotes l ...... er an exposure to irradiation.

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NADPH oxidase DUOX1 promotes l ...... er an exposure to irradiation.

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PNAS.1420707112

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Proceedings of the National Academy of Sciences of the United States of America

P1476

NADPH oxidase DUOX1 promotes l ...... er an exposure to irradiation.

@en

P2093

Abir Al Ghuzlan

http://www.w3.org/2001/XMLSchema#string

Corinne Dupuy

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Dana Hartl

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Florent de Vathaire

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Françoise Miot

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Ibrahima Diallo

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Jean-Michel Bidart

http://www.w3.org/2001/XMLSchema#string

Maria Carolina de Souza Dos Santos

http://www.w3.org/2001/XMLSchema#string

Martin Schlumberger

http://www.w3.org/2001/XMLSchema#string

Monique Talbot

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P2860

Purification of a novel flavoprotein involved in the thyroid NADPH oxidase. Cloning of the porcine and human cdnas

Cloning of two human thyroid cDNAs encoding new members of the NADPH oxidase family

Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a)

Feedback between p21 and reactive oxygen production is necessary for cell senescence

Differential regulation of dual NADPH oxidases/peroxidases, Duox1 and Duox2, by Th1 and Th2 cytokines in respiratory tract epithelium

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

Exposing the thyroid to radiation: a review of its current extent, risks, and implications

NADPH oxidase mediates radiation-induced oxidative stress in rat brain microvascular endothelial cells

Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

A DNA damage checkpoint response in telomere-initiated senescence

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5051-5056

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10.1073/PNAS.1420707112

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16

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112

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25848056

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