NSCLC cells demonstrate differential mode of cell death in response to the combined treatment of radiation and a DNA-PKcs inhibitor.

NSCLC cells demonstrate differential mode of cell death in response to the combined treatment of radiation and a DNA-PKcs inhibitor.

Item
http://www.wikidata.org/entity/Q35551885
Cellular Pathways in Response to Ionizing Radiation and Their Targetability for Tumor RadiosensitizationPreventing Damage Limitation: Targeting DNA-PKcs and DNA Double-Strand Break Repair Pathways for Ovarian Cancer TherapyTumor physiological changes during hypofractionated stereotactic body radiation therapy assessed using multi-parametric magnetic resonance imaging.DNA-Dependent Protein Kinase As Molecular Target for Radiosensitization of Neuroblastoma Cells.Nontoxic concentration of DNA-PK inhibitor NU7441 radio-sensitizes lung tumor cells with little effect on double strand break repair.Suppression of PC-1/PrLZ sensitizes prostate cancer cells to ionizing radiation by attenuating DNA damage repair and inducing autophagic cell death.PIG3 promotes NSCLC cell mitotic progression and is associated with poor prognosis of NSCLC patientsInflammation-induced radioresistance is mediated by ROS-dependent inactivation of protein phosphatase 1 in non-small cell lung cancer cells.Autophagy is not uniformly cytoprotective: a personalized medicine approach for autophagy inhibition as a therapeutic strategy in non-small cell lung cancer.Role of autophagy in regulating the radiosensitivity of tumor cells.DNA-PK Inhibition by NU7441 Enhances Chemosensitivity to Topoisomerase Inhibitor in Non-Small Cell Lung Carcinoma Cells by Blocking DNA Damage RepairNovel DNA targeted therapies for head and neck cancers: clinical potential and biomarkers.Inactivation of DNA-PK by knockdown DNA-PKcs or NU7441 impairs non-homologous end-joining of radiation-induced double strand break repair.EGFR tyrosine kinase inhibitor HS-10182 increases radiation sensitivity in non-small cell lung cancers with EGFR T790M mutation.NF-κB inhibition by dimethylaminoparthenolide radiosensitizes non-small-cell lung carcinoma by blocking DNA double-strand break repair.
P2860

NSCLC cells demonstrate differential mode of cell death in response to the combined treatment of radiation and a DNA-PKcs inhibitor.

Item
http://www.wikidata.org/entity/Q35551885
description
2015 nî lūn-bûn
@nan
2015 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
NSCLC cells demonstrate differ ...... tion and a DNA-PKcs inhibitor.
@ast
NSCLC cells demonstrate differ ...... tion and a DNA-PKcs inhibitor.
@en
type
label
NSCLC cells demonstrate differ ...... tion and a DNA-PKcs inhibitor.
@ast
NSCLC cells demonstrate differ ...... tion and a DNA-PKcs inhibitor.
@en
prefLabel
NSCLC cells demonstrate differ ...... tion and a DNA-PKcs inhibitor.
@ast
NSCLC cells demonstrate differ ...... tion and a DNA-PKcs inhibitor.
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698
P932
P356
P1433
P1476
NSCLC cells demonstrate differ ...... tion and a DNA-PKcs inhibitor.
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698
P932