Reversal of endocrine resistance in breast cancer: interrelationships among 14-3-3ζ, FOXM1, and a gene signature associated with mitosis.
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The Role of Forkhead Box Protein M1 in Breast Cancer Progression and Resistance to TherapyIntegration of molecular profiling and chemical imaging to elucidate fibroblast-microenvironment impact on cancer cell phenotype and endocrine resistance in breast cancerAmplified loci on chromosomes 8 and 17 predict early relapse in ER-positive breast cancersIntegrative analysis of miRNA and gene expression reveals regulatory networks in tamoxifen-resistant breast cancerNext-generation transcriptome sequencing of the premenopausal breast epithelium using specimens from a normal human breast tissue bank.Novel roles for ERK5 and cofilin as critical mediators linking ERα-driven transcription, actin reorganization, and invasiveness in breast cancer.Integration of mRNA expression profile, copy number alterations, and microRNA expression levels in breast cancer to improve grade definition.Transcriptomic analyses of RNA-binding proteins reveal eIF3c promotes cell proliferation in hepatocellular carcinoma.A cancer-related protein 14-3-3ζ is a potential tumor-associated antigen in immunodiagnosis of hepatocellular carcinoma.Altered expression of 14-3-3ζ protein in spinal cords of rat fetuses with spina bifida apertaThe forkhead transcription factor FOXM1 promotes endocrine resistance and invasiveness in estrogen receptor-positive breast cancer by expansion of stem-like cancer cellsIntegration of Network Biology and Imaging to Study Cancer Phenotypes and Responses.FoxM1 promotes breast tumorigenesis by activating PDGF-A and forming a positive feedback loop with the PDGF/AKT signaling pathway.Phosphorylation by p38 mitogen-activated protein kinase promotes estrogen receptor α turnover and functional activity via the SCF(Skp2) proteasomal complex.Mechanisms of resistance to endocrine therapy in breast cancer: focus on signaling pathways, miRNAs and genetically based resistance.Computational Inferences of the Functions of Alternative/Noncanonical Splice Isoforms Specific to HER2+/ER-/PR- Breast Cancers, a Chromosome 17 C-HPP Study.In silico discovery of mitosis regulation networks associated with early distant metastases in estrogen receptor positive breast cancers.A MicroRNA196a2* and TP63 circuit regulated by estrogen receptor-α and ERK2 that controls breast cancer proliferation and invasiveness properties.14-3-3ζ as a predictor of early time to recurrence and distant metastasis in hormone receptor-positive and -negative breast cancers.Genome-wide mapping of FOXM1 binding reveals co-binding with estrogen receptor alpha in breast cancer cells.Crosstalk between PKCα and Notch-4 in endocrine-resistant breast cancer cells.Identification of cell cycle-regulated genes periodically expressed in U2OS cells and their regulation by FOXM1 and E2F transcription factors.Candidate luminal B breast cancer genes identified by genome, gene expression and DNA methylation profilingEstrogen receptor-positive breast cancer molecular signatures and therapeutic potentials (Review).Integrative genomics of gene and metabolic regulation by estrogen receptors α and β, and their coregulators.ErbB2, FoxM1 and 14-3-3ζ prime breast cancer cells for invasion in response to ionizing radiation.MicroRNAs with a role in gene regulation and in human diseases.Estrogen Receptor-β Modulation of the ERα-p53 Loop Regulating Gene Expression, Proliferation, and Apoptosis in Breast Cancer.Mitochondrial biogenesis is required for the anchorage-independent survival and propagation of stem-like cancer cellsSecretome proteins as candidate biomarkers for aggressive thyroid carcinomas.The effect of 14-3-3ζ expression on tamoxifen resistance and breast cancer recurrence: a Danish population-based study.Tanshinone IIA inhibits the proliferation, migration and invasion of ectopic endometrial stromal cells of adenomyosis via 14-3-3ζ downregulation.14-3-3ζ loss impedes oncogene-induced mammary tumorigenesis and metastasis by attenuating oncogenic signaling.Modulation of estrogen related receptor alpha activity by the kinesin KIF17.Casticin induces ovarian cancer cell apoptosis by repressing FoxM1 through the activation of FOXO3a.Transcription factors as master regulator for cancer stemness: remove milk from fox?Suppressor of fused (Sufu) promotes epithelial-mesenchymal transition (EMT) in cervical squamous cell carcinoma.FoxM1 is a promising candidate target in the treatment of breast cancer.OCT-4: a novel estrogen receptor-α collaborator that promotes tamoxifen resistance in breast cancer cells.Involvement of multiple cellular pathways in regulating resistance to tamoxifen in BIK-suppressed MCF-7 cells.
P2860
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P2860
Reversal of endocrine resistance in breast cancer: interrelationships among 14-3-3ζ, FOXM1, and a gene signature associated with mitosis.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Reversal of endocrine resistan ...... ature associated with mitosis.
@ast
Reversal of endocrine resistan ...... ature associated with mitosis.
@en
type
label
Reversal of endocrine resistan ...... ature associated with mitosis.
@ast
Reversal of endocrine resistan ...... ature associated with mitosis.
@en
prefLabel
Reversal of endocrine resistan ...... ature associated with mitosis.
@ast
Reversal of endocrine resistan ...... ature associated with mitosis.
@en
P2860
P356
P1476
Reversal of endocrine resistan ...... ature associated with mitosis.
@en
P2093
Anna Bergamaschi
Barbara L Christensen
P2860
P2888
P356
10.1186/BCR2913
P577
2011-06-29T00:00:00Z
P5875
P6179
1006060172