Identification of homing receptors that mediate the recruitment of CD4 T cells to the genital tract following intravaginal infection with Chlamydia trachomatis.
about
Sex hormones selectively impact the endocervical mucosal microenvironment: implications for HIV transmissionHSV-2-driven increase in the expression of α4β7 correlates with increased susceptibility to vaginal SHIV(SF162P3) infectionHIV-1 envelope, integrins and co-receptor use in mucosal transmission of HIVTissue-Resident T Cells as the Central Paradigm of Chlamydia ImmunityPreferential production of interferon-gamma by CD4+ T cells expressing the homing receptor integrin alpha4/beta7Identification of novel single nucleotide polymorphisms in inflammatory genes as risk factors associated with trachomatous trichiasis.Mouse strain-dependent differences in susceptibility to Neisseria gonorrhoeae infection and induction of innate immune responses.Host chemokine and cytokine response in the endocervix within the first developmental cycle of Chlamydia muridarum.Expression of mucosal homing receptor alpha4beta7 is associated with enhanced migration to the Chlamydia-infected murine genital mucosa in vivo.Specific-antibody-secreting cells in the rectums and genital tracts of nonhuman primates following vaccination.Characterization of human immunodeficiency virus Gag-specific gamma interferon-expressing cells following protective mucosal immunization with alphavirus replicon particles.Two different homing pathways involving integrin β7 and E-selectin significantly influence trafficking of CD4 cells to the genital tract following Chlamydia muridarum infection.The combination of the gastrointestinal integrin (α4β7) and selectin ligand enhances T-Cell migration to the reproductive tract during infection with Chlamydia trachomatis.Analysis of vaginal cell populations during experimental vaginal candidiasis.Differential regulation of CD4 lymphocyte recruitment between the upper and lower regions of the genital tract during Chlamydia trachomatis infectionIn situ analysis of the evolution of the primary immune response in murine Chlamydia trachomatis genital tract infection.Inflammation and clearance of Chlamydia trachomatis in enteric and nonenteric mucosae.Chlamydia trachomatis infection does not enhance local cellular immunity against concurrent Candida vaginal infection.Early local cytokine profiles in strains of mice with different outcomes from chlamydial genital tract infectionCell adhesion molecule and lymphocyte activation marker expression during experimental vaginal candidiasis.Mouse strain-dependent chemokine regulation of the genital tract T helper cell type 1 immune response.Nasal and vaginal vaccinations have differential effects on antibody responses in vaginal and cervical secretions in humans.Chemokine and chemokine receptor dynamics during genital chlamydial infection.Immunization with a combination of integral chlamydial antigens and a defined secreted protein induces robust immunity against genital chlamydial challenge.Chemokine expression patterns differ within anatomically distinct regions of the genital tract during Chlamydia trachomatis infection.Immunity to murine chlamydial genital infection.A Chlamydia trachomatis-specific Th2 clone does not provide protection against a genital infection and displays reduced trafficking to the infected genital mucosaThe major CD8 T cell effector memory subset in the normal and Chlamydia trachomatis-infected human endocervix is low in perforinTargeting α4β7 integrin reduces mucosal transmission of simian immunodeficiency virus and protects gut-associated lymphoid tissue from infection.Vaccination against Chlamydia genital infection utilizing the murine C. muridarum model.Microbiome, sex hormones, and immune responses in the reproductive tract: challenges for vaccine development against sexually transmitted infections.Retinoic acid as a vaccine adjuvant enhances CD8+ T cell response and mucosal protection from viral challenge.Pathogenesis of genital tract disease due to Chlamydia trachomatisTumor necrosis factor alpha production from CD8+ T cells mediates oviduct pathological sequelae following primary genital Chlamydia muridarum infectionImiquimod does not affect shedding of viable chlamydiae in a murine model of Chlamydia trachomatis genital tract infectionUnique features of antiviral immune system of the vaginal mucosaChlamydial intracellular survival strategies.A distinct cellular profile is seen in the human endocervix during Chlamydia trachomatis infection.Chlamydial infection of the gastrointestinal tract: a reservoir for persistent infection.The infecting dose of Chlamydia muridarum modulates the innate immune response and ascending infection.
P2860
Q21090622-AAD97515-FA81-4431-A1B0-A13A6F8F8CE9Q27322178-2509B44A-3F6D-4A7B-92E6-356AE3EEB97FQ27497560-FC2D93A5-E959-4A57-B12A-ED3BA4A06715Q28075820-61CF00E0-6821-482A-8977-74963E2B03C1Q28367021-9459B53E-0F72-4BC4-9A4C-46744FFAE62DQ33381603-A2443813-BA92-437E-835B-CBE695BFD7FEQ33557749-DF15DD47-D0EB-4DE3-BA63-E218397EEDCCQ33557924-7E7B0B89-77FB-418A-A4E8-6AD0C163025EQ33591433-71B961CF-50E3-4BD3-869A-AE36089F63BEQ33769451-36FDB7A4-B5D6-469F-8881-4C743CB52791Q33788861-8F6A25ED-89CE-4725-8312-7B7190053F98Q33926235-5F461017-C6B5-4815-A0BE-8E843D0C4769Q33940192-C292D72F-8BFE-4DC0-A1C4-26048A52A864Q34001275-AD963F7E-3462-41E9-9B2E-81D2C617D6D2Q34003642-9739CD57-76FE-480C-A9B4-B33E9318BCE2Q34004414-2686B150-7B13-4588-8AEF-074F9F18715DQ34006511-FA2F9D46-9EAF-4FAE-A96C-75AF868256F4Q34007432-6A139EBD-B9ED-404F-B104-DE69D9B9B753Q34007525-65A635DE-C701-40AC-988E-9B0ACF483E28Q34008683-60596383-6DC9-4E14-BAFA-6533D40EA974Q34009652-CCA776E1-1ED9-4C27-9C48-8F984D12690CQ34009698-9594B9BE-68D6-4C1A-87E3-C08F67DAE096Q34117128-60FE0E44-4C8A-492C-918A-D3B6652DC546Q34119450-5440DA8A-62D4-40E4-8776-8CB0A5E37703Q34119846-3F1A0CDF-7DCA-48A0-ADB6-68C53EAEC6CAQ34124225-C644E21B-BF90-490F-83EA-CBC8C24224F7Q34129905-77043081-8B3C-416A-B3BF-E6FBEECAC471Q34502654-3A9CEECD-A806-4FAA-962D-70BA91AF6890Q34641933-E8EF390A-F745-499A-B2FB-FA9E21B1A651Q34739969-61AFC5CD-2080-484E-8014-82CDC3F03CD0Q35019917-721C8DB6-29CE-47C4-ADD9-0DBEB02EA65AQ35140438-8DD7AB86-1D08-4A8F-A7AD-1BAE667860D5Q35148159-C1ECDA31-680C-4682-8D79-39749695A423Q35329016-E75B497C-BC6C-432A-AC55-DC2335E6455AQ35751196-53C33E4A-8FFE-4AA5-B370-CE728EA69A3DQ36177229-3BD718A1-692B-4FC9-A0B5-A96D5DA80047Q36786613-943D4875-06C4-409F-B363-94A9759EDB29Q36951927-3FC79FBF-4780-4D9E-BDA5-2B604E6EF4B5Q37114957-56615DEE-2AB4-4BB0-A0FD-FE318C38C73CQ37582864-12337F23-6E94-4FFC-BA87-59893DA91FB9
P2860
Identification of homing receptors that mediate the recruitment of CD4 T cells to the genital tract following intravaginal infection with Chlamydia trachomatis.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Identification of homing recep ...... on with Chlamydia trachomatis.
@ast
Identification of homing recep ...... on with Chlamydia trachomatis.
@en
type
label
Identification of homing recep ...... on with Chlamydia trachomatis.
@ast
Identification of homing recep ...... on with Chlamydia trachomatis.
@en
prefLabel
Identification of homing recep ...... on with Chlamydia trachomatis.
@ast
Identification of homing recep ...... on with Chlamydia trachomatis.
@en
P2860
P1476
Identification of homing recep ...... on with Chlamydia trachomatis.
@en
P2093
P2860
P304
P407
P577
1997-12-01T00:00:00Z