Insulin-like growth factor-1 and post-ischemic brain injury.
about
JAK/STAT3 pathway is involved in survival of neurons in response to insulin-like growth factor and negatively regulated by suppressor of cytokine signaling-3Therapeutic Hypothermia for Neonatal Hypoxic-Ischemic Encephalopathy - Where to from Here?Role and Importance of IGF-1 in Traumatic Brain InjuriesGIGYF2 is present in endosomal compartments in the mammalian brains and enhances IGF-1-induced ERK1/2 activationmicroRNA-181a is involved in insulin-like growth factor-1-mediated regulation of the transcription factor CREB1Expression of insulin-like growth factor system genes during the early postnatal neurogenesis in the mouse hippocampusTemporal and regional changes in IGF-1/IGF-1R signaling in the mouse brain after traumatic brain injury.PEGylated insulin-like growth factor-I affords protection and facilitates recovery of lost functions post-focal ischemia.Bone marrow stromal cells promote neuromotor functional recovery, via upregulation of neurotrophic factors and synapse proteins following traumatic brain injury in ratsInhibition of the insulin-like growth factor-1 receptor (IGF1R) tyrosine kinase as a novel cancer therapy approachBlunting type 1 insulin-like growth factor receptor expression exacerbates neuronal apoptosis following hypoxic/ischemic injuryEffects of insulin-like growth factor-I on cytokine-induced sickness behavior in mice.Neuroprotective effects of enriched environment housing after transient global cerebral ischaemia are associated with the upregulation of insulin-like growth factor-1 signalling.Angiogenesis in old-aged subjects after ischemic stroke: a cautionary note for investigatorsRevenge of the "sit": how lifestyle impacts neuronal and cognitive health through molecular systems that interface energy metabolism with neuronal plasticity.Endothelin-1-induced focal cerebral ischemia in the growth hormone/IGF-1 deficient Lewis Dwarf rat.The neuroprotective effects of intramuscular insulin-like growth factor-I treatment in brain ischemic rats.Neuroprotective actions of estradiol and novel estrogen analogs in ischemia: translational implicationsSeparation of fast from slow anabolism by site-specific PEGylation of insulin-like growth factor I (IGF-I).Stratified control of IGF-I expression by hypoxia and stress hormones in osteoblasts.Spatial distribution of insulin-like growth factor binding protein-2 following hypoxic-ischemic injuryDeficiency of brain ATP-binding cassette transporter A-1 exacerbates blood-brain barrier and white matter damage after stroke.The influences of diet and exercise on mental health through hormesisNeurosphere-derived cells exert a neuroprotective action by changing the ischemic microenvironment.Short-term dietary restriction and fasting precondition against ischemia reperfusion injury in mice.Brain injury in chronically ventilated preterm neonates: collateral damage related to ventilation strategyThe effect of recombinant human growth hormone therapy in patients with completed stroke: a pilot trial.D-4F Decreases White Matter Damage After Stroke in MiceSubventricular zone-derived neural stem cell grafts protect against hippocampal degeneration and restore cognitive function in the mouse following intrahippocampal kainic acid administrationFrom blood to brain: amoeboid microglial cell, a nascent macrophage and its functions in developing brain.Neuroprotective levels of IGF-1 exacerbate epileptogenesis after brain injury.Blueberry polyphenols attenuate kainic acid-induced decrements in cognition and alter inflammatory gene expression in rat hippocampus.Antidepressant-like behavioral effects of IGF-I produced by enhanced serotonin transmission.Insulin-like Signaling Promotes Glial Phagocytic Clearance of Degenerating Axons through Regulation of Draper.Treadmill exercise reduces spinal cord injury-induced apoptosis by activating the PI3K/Akt pathway in ratsCyclic glycine-proline regulates IGF-1 homeostasis by altering the binding of IGFBP-3 to IGF-1.Proof-of Concept that an Acute Trophic Factors Intervention After Spinal Cord Injury Provides an Adequate Niche for Neuroprotection, Recruitment of Nestin-Expressing Progenitors and RegenerationHyperglycemia in stroke and possible treatments.Astrocytes require insulin-like growth factor I to protect neurons against oxidative injury.Gene expression changes and molecular pathways mediating activity-dependent plasticity in visual cortex.
P2860
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P2860
Insulin-like growth factor-1 and post-ischemic brain injury.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Insulin-like growth factor-1 and post-ischemic brain injury.
@ast
Insulin-like growth factor-1 and post-ischemic brain injury.
@en
type
label
Insulin-like growth factor-1 and post-ischemic brain injury.
@ast
Insulin-like growth factor-1 and post-ischemic brain injury.
@en
prefLabel
Insulin-like growth factor-1 and post-ischemic brain injury.
@ast
Insulin-like growth factor-1 and post-ischemic brain injury.
@en
P2093
P1476
Insulin-like growth factor-1 and post-ischemic brain injury.
@en
P2093
P304
P356
10.1016/J.PNEUROBIO.2003.08.002
P577
2003-08-01T00:00:00Z