Altered GluN2B NMDA receptor function and synaptic plasticity during early pathology in the PS2APP mouse model of Alzheimer's disease
about
Early synaptic deficits in the APP/PS1 mouse model of Alzheimer's disease involve neuronal adenosine A2A receptors.Intrahippocampal administration of a domain antibody that binds aggregated amyloid-β reverses cognitive deficits produced by diet-induced obesity.Altered Cortical Synaptic Plasticity in Response to 5-Hz Repetitive Transcranial Magnetic Stimulation as a New Electrophysiological Finding in Amnestic Mild Cognitive Impairment Converting to Alzheimer's Disease: Results from a 4-year Prospective CoRole of amyloid β protein receptors in mediating synaptic plasticity.Bis(propyl)-cognitin Prevents β-amyloid-induced Memory Deficits as Well as Synaptic Formation and Plasticity Impairments via the Activation of PI3-K Pathway.Inhibition of NMDA Receptors Prevents the Loss of BDNF Function Induced by Amyloid β.
P2860
Altered GluN2B NMDA receptor function and synaptic plasticity during early pathology in the PS2APP mouse model of Alzheimer's disease
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
Altered GluN2B NMDA receptor f ...... e model of Alzheimer's disease
@ast
Altered GluN2B NMDA receptor f ...... e model of Alzheimer's disease
@en
type
label
Altered GluN2B NMDA receptor f ...... e model of Alzheimer's disease
@ast
Altered GluN2B NMDA receptor f ...... e model of Alzheimer's disease
@en
prefLabel
Altered GluN2B NMDA receptor f ...... e model of Alzheimer's disease
@ast
Altered GluN2B NMDA receptor f ...... e model of Alzheimer's disease
@en
P2093
P2860
P1476
Altered GluN2B NMDA receptor f ...... e model of Alzheimer's disease
@en
P2093
Jean-Francois Pare
Jesse E Hanson
Lunbin Deng
Qiang Zhou
Yoland Smith
P2860
P304
P356
10.1016/J.NBD.2014.11.017
P577
2014-12-04T00:00:00Z