Innate immunity SNPs are associated with risk for severe sepsis after burn injury. - Wikidata - awgldk - TriplyDB

Innate immunity SNPs are associated with risk for severe sepsis after burn injury.

Innate immunity SNPs are associated with risk for severe sepsis after burn injury.

http://www.wikidata.org/entity/Q35575098

about

Toll-like receptor 4 region genetic variants are associated with susceptibility to melioidosis.Association between CD14 promoter -159C/T polymorphism and the risk of sepsis and mortality: a systematic review and meta-analysis.Ethical considerations in the collection of genetic data from critically ill patients: what do published studies reveal about potential directions for empirical ethics research?Differential cytokine gene expression according to outcome in a hamster model of leptospirosis.Divergent and dynamic activity of endogenous retroviruses in burn patients and their inflammatory potential.Genetic variants in genes of the inflammatory response in association with infective endocarditis.Clinical relevance of IL-6 gene polymorphism in severely injured patients.The CD14 rs2569190 TT Genotype Is Associated with an Improved 30-Day Survival in Patients with Sepsis: A Prospective Observational Cohort Study.Association of tumor necrosis factor β genetic polymorphism and sepsis susceptibility Toll-like receptor polymorphisms, inflammatory and infectious diseases, allergies, and cancer.Bench-to-bedside review: Association of genetic variation with sepsis Toll-like receptors: their roles in bacterial recognition and respiratory infections.Sepsis: links between pathogen sensing and organ damage Tumor necrosis factor gene variation and the risk of mortality after burn injury: a cohort study.Variation in the TLR4 gene influences the risk of organ failure and shock posttrauma: a cohort study.Genetic risk of acute pulmonary infections and sepsis.Mechanisms and regulation of the gene-expression response to sepsis.Genetic variations in toll-like receptor pathway genes influence asthma and atopy.Genetic predisposition to respiratory infection and sepsis.TLR4 polymorphisms and disease susceptibility.Association of TLR1, TLR2, TLR4, TLR6, and TIRAP polymorphisms with disease susceptibility.Role of Thr399Ile and Asp299Gly polymorphisms of toll-like receptor-4 gene in acute dental abscess.The relationship between toll like receptor 4 gene rs4986790 and rs4986791 polymorphisms and sepsis susceptibility: A meta-analysis.Risk of infection and sepsis in severely injured patients related to single nucleotide polymorphisms in the lectin pathway.A TREM-1 Polymorphism A/T within the Exon 2 Is Associated with Pneumonia in Burn-Injured Patients Tumor necrosis factor-α -308 G/A polymorphism and risk of sepsis, septic shock, and mortality: an updated meta-analysis.Association of CD14 rs2569190 polymorphism with mortality in shock septic patients who underwent major cardiac or abdominal surgery: A retrospective study.Variants at the promoter of the interleukin-6 gene are associated with severity and outcome of pneumococcal community-acquired pneumonia

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P2860

Innate immunity SNPs are associated with risk for severe sepsis after burn injury.

http://www.wikidata.org/entity/Q35575098

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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2006年论文

@zh-cn

name

Innate immunity SNPs are associated with risk for severe sepsis after burn injury.

@ast

Innate immunity SNPs are associated with risk for severe sepsis after burn injury.

@en

type

label

Innate immunity SNPs are associated with risk for severe sepsis after burn injury.

@ast

Innate immunity SNPs are associated with risk for severe sepsis after burn injury.

@en

prefLabel

Innate immunity SNPs are associated with risk for severe sepsis after burn injury.

@ast

Innate immunity SNPs are associated with risk for severe sepsis after burn injury.

@en

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Clinical Medicine and Research

P1476

Innate immunity SNPs are associated with risk for severe sepsis after burn injury.

@en

P2093

Brett D Arnoldo

http://www.w3.org/2001/XMLSchema#string

Corinne C Aragaki

http://www.w3.org/2001/XMLSchema#string

Gary F Purdue

http://www.w3.org/2001/XMLSchema#string

John L Hunt

http://www.w3.org/2001/XMLSchema#string

Jureta W Horton

http://www.w3.org/2001/XMLSchema#string

Ling-Yu E Chang

http://www.w3.org/2001/XMLSchema#string

Robert C Barber

http://www.w3.org/2001/XMLSchema#string

P2860

TLR4 mutations are associated with endotoxin hyporesponsiveness in humans

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

Physical contact between lipopolysaccharide and toll-like receptor 4 revealed by genetic complementation

Phylogenetic variation and polymorphism at the toll-like receptor 4 locus (TLR4)

A simple salting out procedure for extracting DNA from human nucleated cells

Relevance of mutations in the TLR4 receptor in patients with gram-negative septic shock

Consensus conference definitions for sepsis, septic shock, acute lung injury, and acute respiratory distress syndrome: time for a reevaluation.

Toll4 (TLR4) expression in cardiac myocytes in normal and failing myocardium.

Epidemiology of sepsis: an update.

TLR4 and TNF-alpha polymorphisms are associated with an increased risk for severe sepsis following burn injury.

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250-255

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10.3121/CMR.4.4.250

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4

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6590364

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17210974

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1764809

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