Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo. - Wikidata - awgldk - TriplyDB

Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.

Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.

http://www.wikidata.org/entity/Q35578499

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Insulin resistance is a cellular antioxidant defense mechanism Variation in the link between oxygen consumption and ATP production, and its relevance for animal performance Mitochondrial and skeletal muscle health with advancing age Mitochondrial involvement and impact in aging skeletal muscle Mitochondrial oxidative stress in aging and healthspan First-in-class cardiolipin-protective compound as a therapeutic agent to restore mitochondrial bioenergetics Capsiate supplementation reduces oxidative cost of contraction in exercising mouse skeletal muscle in vivo Effect of short-term thyroxine administration on energy metabolism and mitochondrial efficiency in humans Senescence-accelerated OXYS rats: a model of age-related cognitive decline with relevance to abnormalities in Alzheimer disease.Mitochondrial changes in platelets are not related to those in skeletal muscle during human septic shock.Mitochondrial function as a determinant of life span.The neuroprotective steroid progesterone promotes mitochondrial uncoupling, reduces cytosolic calcium and augments stress resistance in yeast cells Mitochondrial DNA alterations and reduced mitochondrial function in aging Mitochondrial uncoupling and lifespan.Reduced coupling of oxidative phosphorylation in vivo precedes electron transport chain defects due to mild oxidative stress in mice.Further support to the uncoupling-to-survive theory: the genetic variation of human UCP genes is associated with longevity.Pathological aspects of lipid peroxidation.Intracellular energetics and critical PO2 in resting ischemic human skeletal muscle in vivo.Running for exercise mitigates age-related deterioration of walking economy.The impact of age-related dysregulation of the angiotensin system on mitochondrial redox balance Mitochondrial function in physically active elders with sarcopenia.Regulation of skeletal muscle mitochondrial function: genes to proteins.Skeletal muscle mitochondrial capacity and insulin resistance in type 2 diabetes Mitochondrial metabolic function assessed in vivo and in vitro.Niclosamide ethanolamine-induced mild mitochondrial uncoupling improves diabetic symptoms in mice.Effects of aging on mechanical efficiency and muscle activation during level and uphill walking Ectopic lipid accumulation and reduced glucose tolerance in elderly adults are accompanied by altered skeletal muscle mitochondrial activity Insulin Resistance Is Not Associated with an Impaired Mitochondrial Function in Contracting Gastrocnemius Muscle of Goto-Kakizaki Diabetic Rats In Vivo Fish oil-derived n-3 PUFA therapy increases muscle mass and function in healthy older adults.Mitochondrial respiratory capacity and coupling control decline with age in human skeletal muscle.Age-Related and Heteroplasmy-Related Variation in Human mtDNA Copy Number Impact of age on exercise-induced ATP supply during supramaximal plantar flexion in humans.What do magnetic resonance-based measurements of Pi→ATP flux tell us about skeletal muscle metabolism?Skeletal muscle mitochondria and aging: a review.Reconsidering the Role of Mitochondria in Aging.Impaired adaptability of in vivo mitochondrial energetics to acute oxidative insult in aged skeletal muscle Age modifies respiratory complex I and protein homeostasis in a muscle type-specific manner.Higher mitochondrial respiration and uncoupling with reduced electron transport chain content in vivo in muscle of sedentary versus active subjects Understanding the physiology of the ageing individual: computational modelling of changes in metabolism and endurance.Skeletal muscle mitochondrial energetics are associated with maximal aerobic capacity and walking speed in older adults

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Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.

http://www.wikidata.org/entity/Q35578499

description

2007 nî lūn-bûn

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Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.

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Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.

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Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.

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Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.

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Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.

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Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.

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Proceedings of the National Academy of Sciences of the United States of America

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Mild mitochondrial uncoupling impacts cellular aging in human muscles in vivo.

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Catherine E Amara

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Eric G Shankland

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Kevin E Conley

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Martin J Kushmerick

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Sharon A Jubrias

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P2860

Superoxide activates mitochondrial uncoupling proteins

Effect of triiodothyronine on mitochondrial energy coupling in human skeletal muscle

The free radical theory of aging matures

Mitochondrial dysfunction in the elderly: possible role in insulin resistance

In vivo effects of uncoupling protein-3 gene disruption on mitochondrial energy metabolism.

Aconitase and ATP synthase are targets of malondialdehyde modification and undergo an age-related decrease in activity in mouse heart mitochondria.

Performance of near-infrared spectroscopy in measuring local O(2) consumption and blood flow in skeletal muscle.

Uncoupling to survive? The role of mitochondrial inefficiency in ageing.

Data on the distribution of fibre types in thirty-six human muscles. An autopsy study.

Skeletal muscle metabolism is a major determinant of resting energy expenditure.

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