53BP1 contributes to a robust genomic stability in human fibroblasts - Wikidata - awgldk - TriplyDB

53BP1 contributes to a robust genomic stability in human fibroblasts

53BP1 contributes to a robust genomic stability in human fibroblasts

http://www.wikidata.org/entity/Q35582353

about

Cancer susceptibility and reproductive trade-offs: a model of the evolution of cancer defences Relationship of spindle assembly checkpoint fidelity to species body mass, lifespan, and developmental rate.p27kip1 protein levels reflect a nexus of oncogenic signaling during cell transformation.DNA damage markers in dermal fibroblasts in vitro reflect chronological donor age.Cancer cells that survive checkpoint adaptation contain micronuclei that harbor damaged DNA.DNA Damage Detection by 53BP1: Relationship to Species Longevity.Convergent adaptation of cellular machineries in the evolution of large body masses and long life spans.Differences in 53BP1 and BRCA1 regulation between cycling and non-cycling cells.Chromosome territory relocation during DNA repair requires nuclear myosin 1 recruitment to chromatin mediated by ϒ-H2AX signaling.Gold Nanoparticles Promote Oxidant-Mediated Activation of NF-κB and 53BP1 Recruitment-Based Adaptive Response in Human Astrocytes.MicroRNA-152 and -181a participate in human dermal fibroblasts senescence acting on cell adhesion and remodeling of the extra-cellular matrix.

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53BP1 contributes to a robust genomic stability in human fibroblasts

http://www.wikidata.org/entity/Q35582353

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

53BP1 contributes to a robust genomic stability in human fibroblasts

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53BP1 contributes to a robust genomic stability in human fibroblasts

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type

label

53BP1 contributes to a robust genomic stability in human fibroblasts

@ast

53BP1 contributes to a robust genomic stability in human fibroblasts

@en

prefLabel

53BP1 contributes to a robust genomic stability in human fibroblasts

@ast

53BP1 contributes to a robust genomic stability in human fibroblasts

@en

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53BP1 contributes to a robust genomic stability in human fibroblasts

@en

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Antonello Lorenzini

http://www.w3.org/2001/XMLSchema#string

Chad Lerner

http://www.w3.org/2001/XMLSchema#string

Christian Sell

http://www.w3.org/2001/XMLSchema#string

Emanuela Caiazza

http://www.w3.org/2001/XMLSchema#string

Lauren S Fink

http://www.w3.org/2001/XMLSchema#string

Michaela Roell

http://www.w3.org/2001/XMLSchema#string

Silvana Hrelia

http://www.w3.org/2001/XMLSchema#string

Thomas Stamato

http://www.w3.org/2001/XMLSchema#string

P2860

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

Significant correlation of species longevity with DNA double strand break recognition but not with telomere length

DNA damage-induced G2-M checkpoint activation by histone H2AX and 53BP1

Differential requirement for H2AX and 53BP1 in organismal development and genome maintenance in the absence of poly(ADP)ribosyl polymerase 1.

Cell line-specific differences in the control of cell cycle progression in the absence of mitosis

53BP1, a mediator of the DNA damage checkpoint.

TopBP1 functions with 53BP1 in the G1 DNA damage checkpoint.

Cellular replicative capacity correlates primarily with species body mass not longevity.

Modelling the molecular circuitry of cancer.

Ku: a multifunctional protein involved in telomere maintenance.

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scholarly article

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10.18632/AGING.100381

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9

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3

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3227449

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