Pathologic assessment of the vulnerable human coronary plaque.
about
Pathology of coronary atherosclerosis and thrombosisTherapeutic modulation of the natural history of coronary atherosclerosis: lessons learned from serial imaging studiesGene silencing of TACE enhances plaque stability and improves vascular remodeling in a rabbit model of atherosclerosis.Shifting the Focus of Preclinical, Murine Atherosclerosis Studies From Prevention to Late-Stage Intervention.Ultrasound-enhanced bevacizumab release from echogenic liposomes for inhibition of atheroma progression.Applications of optical coherence tomography in cardiovascular medicine, Part 2The actin associated protein palladin in smooth muscle and in the development of diseases of the cardiovasculature and in cancer.PDGF-DD, a novel mediator of smooth muscle cell phenotypic modulation, is upregulated in endothelial cells exposed to atherosclerosis-prone flow patternsIntravascular photoacoustics for image-guidance and temperature monitoring during plasmonic photothermal therapy of atherosclerotic plaques: a feasibility studyReduced apoptosis and plaque necrosis in advanced atherosclerotic lesions of Apoe-/- and Ldlr-/- mice lacking CHOP.Combined optical coherence tomography and intravascular ultrasound radio frequency data analysis for plaque characterization. Classification accuracy of human coronary plaques in vitro.The impact of macrophage insulin resistance on advanced atherosclerotic plaque progression.Atherogenesis and iron: from epidemiology to cellular level.Oncostatin M receptor β deficiency attenuates atherogenesis by inhibiting JAK2/STAT3 signaling in macrophages.The vasa vasorum in diseased and nondiseased arteriesReproducibility of volumetric intravascular ultrasound radiofrequency-based analysis of coronary plaque composition in vivo.MRI discriminates thrombus composition and ST resolution after percutaneous coronary intervention in patients with ST-elevation myocardial infarctionDual-wavelength multifrequency photothermal wave imaging combined with optical coherence tomography for macrophage and lipid detection in atherosclerotic plaques using gold nanoparticles.Serum Non-High-Density Lipoprotein Cholesterol and Risk of Cardiovascular Disease in Community Dwellers with Chronic Kidney Disease: the Hisayama StudyRegulation of foam cells by adenosine.Combined two-photon luminescence microscopy and OCT for macrophage detection in the hypercholesterolemic rabbit aorta using plasmonic gold nanorose.Two-photon luminescence properties of gold nanorods.Aggressive very low-density lipoprotein (VLDL) and LDL lowering by gene transfer of the VLDL receptor combined with a low-fat diet regimen induces regression and reduces macrophage content in advanced atherosclerotic lesions in LDL receptor-deficienAtherosclerotic plaque biomarkers: beyond the horizon of the vulnerable plaque.Characterization of coronary plaques with combined use of intravascular ultrasound, virtual histology and optical coherence tomography.Specific matrix metalloproteinases play different roles in intraplaque angiogenesis and plaque instability in rabbits.The Nature of Iron Deposits Differs between Symptomatic and Asymptomatic Carotid Atherosclerotic Plaques.Acute coronary eventsModeling of Mechanical Stress Exerted by Cholesterol Crystallization on Atherosclerotic PlaquesAntiangiogenic activity of rPAI-1(23) promotes vasa vasorum regression in hypercholesterolemic mice through a plasmin-dependent mechanismPathology of drug-eluting versus bare-metal stents in saphenous vein bypass graft lesions.MR molecular imaging of aortic angiogenesis.Multimodality imaging of atherosclerotic plaque activity and composition using FDG-PET/CT and MRI in carotid and femoral arteriesDetection of the vulnerable coronary atheromatous plaque. Where are we now?Real-Time Intravascular Ultrasound and Photoacoustic Imaging.Women and coronary artery disease. Part I: basic considerations.Cholesterol-induced macrophage apoptosis requires ER stress pathways and engagement of the type A scavenger receptor.Regulator of G-protein signalling 5 protects against atherosclerosis in apolipoprotein E-deficient mice.Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipoproteins invade coronary intima via neovascularization from adventitial vasa vasorum, but not from the arterial lumen: a hypothesis.Thematic review series: patient-oriented research. Imaging atherosclerosis: state of the art.
P2860
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P2860
Pathologic assessment of the vulnerable human coronary plaque.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Pathologic assessment of the vulnerable human coronary plaque.
@ast
Pathologic assessment of the vulnerable human coronary plaque.
@en
type
label
Pathologic assessment of the vulnerable human coronary plaque.
@ast
Pathologic assessment of the vulnerable human coronary plaque.
@en
prefLabel
Pathologic assessment of the vulnerable human coronary plaque.
@ast
Pathologic assessment of the vulnerable human coronary plaque.
@en
P2093
P2860
P356
P1433
P1476
Pathologic assessment of the vulnerable human coronary plaque.
@en
P2093
F D Kolodgie
P2860
P304
P356
10.1136/HRT.2004.041798
P407
P577
2004-12-01T00:00:00Z