Gene expression changes after focal stroke, traumatic brain and spinal cord injuries.
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JAK/STAT3 pathway is involved in survival of neurons in response to insulin-like growth factor and negatively regulated by suppressor of cytokine signaling-3Circadian rhythms and mood: opportunities for multi-level analyses in genomics and neuroscience: circadian rhythm dysregulation in mood disorders provides clues to the brain's organizing principles, and a touchstone for genomics and neuroscienceEpigenetics of neural repair following spinal cord injuryAll's well that transcribes well: non-coding RNAs and post-stroke brain damageAge-dependent modulation of cortical transcriptomes in spinal cord injury and repairIn vivo inhibition of miR-155 significantly alters post-stroke inflammatory responseCritical periods after stroke study: translating animal stroke recovery experiments into a clinical trialComparative analysis of the time-dependent functional and molecular changes in spinal cord degeneration induced by the G93A SOD1 gene mutation and by mechanical compression.Expression and functional significance of SOCS-1 and SOCS-3 in astrocytesLong-term gliosis and molecular changes in the cervical spinal cord of the rhesus monkey after traumatic brain injury.The beta-hCG+erythropoietin in acute stroke (BETAS) study: a 3-center, single-dose, open-label, noncontrolled, phase IIa safety trialCuriosity and cure: translational research strategies for neural repair-mediated rehabilitationDiscovery of pituitary adenylate cyclase-activating polypeptide-regulated genes through microarray analyses in cell culture and in vivo.Human neural stem cells rapidly ameliorate symptomatic inflammation in early-stage ischemic-reperfusion cerebral injury.Bystander Effect Fuels Human Induced Pluripotent Stem Cell-Derived Neural Stem Cells to Quickly Attenuate Early Stage Neurological Deficits After Stroke.Feeding the brain and nurturing the mind: Linking nutrition and the gut microbiota to brain development.Prokineticin 2 is an endangering mediator of cerebral ischemic injuryIn Vivo Inhibition of miR-155 Promotes Recovery after Experimental Mouse StrokeBench to bedside: the quest for quality in experimental stroke research.The therapeutic potential of neural stem cells.Neuroplasticity in the context of motor rehabilitation after stroke.The role of the blood transcriptome in innate inflammation and stroke.Molecular markers and mechanisms of stroke: RNA studies of blood in animals and humans.Getting Closer to an Effective Intervention of Ischemic Stroke: The Big Promise of Stem Cell.SC1/hevin and reactive gliosis after transient ischemic stroke in young and aged rats.Oligodendrocyte death, neuroinflammation, and the effects of minocycline in a rodent model of nonarteritic anterior ischemic optic neuropathy (rNAION).
P2860
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P2860
Gene expression changes after focal stroke, traumatic brain and spinal cord injuries.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Gene expression changes after focal stroke, traumatic brain and spinal cord injuries.
@ast
Gene expression changes after focal stroke, traumatic brain and spinal cord injuries.
@en
type
label
Gene expression changes after focal stroke, traumatic brain and spinal cord injuries.
@ast
Gene expression changes after focal stroke, traumatic brain and spinal cord injuries.
@en
prefLabel
Gene expression changes after focal stroke, traumatic brain and spinal cord injuries.
@ast
Gene expression changes after focal stroke, traumatic brain and spinal cord injuries.
@en
P1476
Gene expression changes after focal stroke, traumatic brain and spinal cord injuries.
@en
P2093
S Thomas Carmichael
P304
P356
10.1097/00019052-200312000-00009
P577
2003-12-01T00:00:00Z