p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis. - Wikidata - awgldk - TriplyDB

p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis.

p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis.

http://www.wikidata.org/entity/Q35588465

about

Inactivation of p38 MAPK contributes to stem cell-like properties of non-small cell lung cancer Stress signaling and the shaping of the mammary tissue in development and cancer.p38α MAPK pathway: a key factor in colorectal cancer therapy and chemoresistance.Epithelial Xbp1 is required for cellular proliferation and differentiation during mammary gland development ID4 regulates mammary gland development by suppressing p38MAPK activity ERK1/2 and p38α/β signaling in tumor cell quiescence: opportunities to control dormant residual disease.Rsu1 contributes to cell adhesion and spreading in MCF10A cells via effects on P38 map kinase signaling Microenvironments dictating tumor cell dormancy Androgen receptor (AR) positive vs negative roles in prostate cancer cell deaths including apoptosis, anoikis, entosis, necrosis and autophagic cell death.Rsu1 contributes to regulation of cell adhesion and spreading by PINCH1-dependent and - independent mechanisms.Roles of p38 MAPKs in invasion and metastasis.Review: Cell Dynamics in Malignant Pleural Effusions.ID4 controls mammary stem cells and marks breast cancers with a stem cell-like phenotype.Loss of anchorage primarily induces non-apoptotic cell death in a human mammary epithelial cell line under atypical focal adhesion kinase signaling.The first EGF domain of coagulation factor IX attenuates cell adhesion and induces apoptosis.Genetic Analyses Reveal Functions for MAP2K3 and MAP2K6 in Mouse Testis Determination.Macrophages orchestrate breast cancer early dissemination and metastasis.Regulation of Mammary Luminal Cell Fate and Tumorigenesis by p38α.Mechanism of early dissemination and metastasis in Her2+ mammary cancer.The cJUN NH2-terminal kinase (JNK) pathway contributes to mouse mammary gland remodeling during involution.The cJUN NH2-terminal kinase (JNK) signaling pathway promotes genome stability and prevents tumor initiation.Functional significance of co-occurring mutations in PIK3CA and MAP3K1 in breast cancer.Priming stem cells with protein kinase C activator enhances early stem cell-chondrocyte interaction by increasing adhesion molecules

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P2860

p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis.

http://www.wikidata.org/entity/Q35588465

description

2011 nî lūn-bûn

@nan

2011年の論文

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2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

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2011年论文

@zh

2011年论文

@zh-cn

name

p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis.

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p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis.

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type

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p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis.

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p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis.

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p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis.

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p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis.

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SCISIGNAL.2001684

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Science Signaling

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p38α Signaling Induces Anoikis and Lumen Formation During Mammary Morphogenesis

@en

P2093

Alvaro Avivar-Valderas

http://www.w3.org/2001/XMLSchema#string

Eduardo F Farias

http://www.w3.org/2001/XMLSchema#string

Huei-Chi Wen

http://www.w3.org/2001/XMLSchema#string

Maria Soledad Sosa

http://www.w3.org/2001/XMLSchema#string

Roger J Davis

http://www.w3.org/2001/XMLSchema#string

P2860

PRAK is essential for ras-induced senescence and tumor suppression

ATF-2 is preferentially activated by stress-activated protein kinases to mediate c-jun induction in response to genotoxic agents

ATF-2 contains a phosphorylation-dependent transcriptional activation domain

The p38 MAPK pathway is essential for skeletogenesis and bone homeostasis in mice

MKK3- and MKK6-regulated gene expression is mediated by the p38 mitogen-activated protein kinase signal transduction pathway

Anoikis mechanisms

Amplification of PPM1D in human tumors abrogates p53 tumor-suppressor activity

Transcription factor ATF2 regulation by the JNK signal transduction pathway

betaTrCP- and Rsk1/2-mediated degradation of BimEL inhibits apoptosis

Specificity and mechanism of action of some commonly used protein kinase inhibitors

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ra34

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scholarly article

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10.1126/SCISIGNAL.2001684

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174

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4

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Julio Aguirre-Ghiso

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2011-05-24T00:00:00Z

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51163048

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21610252

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3229273

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