Reduced inflammation accompanies diminished myelin damage and repair in the NG2 null mouse spinal cord. - Wikidata - awgldk - TriplyDB

Reduced inflammation accompanies diminished myelin damage and repair in the NG2 null mouse spinal cord.

Reduced inflammation accompanies diminished myelin damage and repair in the NG2 null mouse spinal cord.

http://www.wikidata.org/entity/Q35588967

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Oligodendroglial alterations and the role of microglia in white matter injury: relevance to schizophrenia.Intraventricular hemorrhage induces deposition of proteoglycans in premature rabbits, but their in vivo degradation with chondroitinase does not restore myelination, ventricle size and neurological recovery Mechanisms responsible for the inhibitory effects of epothilone B on scar formation after spinal cord injury NG2 cells, a new trail for Alzheimer's disease mechanisms?Abrogation of β-catenin signaling in oligodendrocyte precursor cells reduces glial scarring and promotes axon regeneration after CNS injury.Myelin repair and functional recovery mediated by neural cell transplantation in a mouse model of multiple sclerosis.Accelerated repair of demyelinated CNS lesions in the absence of non-muscle myosin IIB Matrix metalloproteinase-14 both sheds cell surface neuronal glial antigen 2 (NG2) proteoglycan on macrophages and governs the response to peripheral nerve injury Transcriptomic analysis of the effects of a fish oil enriched diet on murine brains.Pro-inflammatory cytokines reduce the proliferation of NG2 cells and increase shedding of NG2 in vivo and in vitro.Sustained neuroprotection from a single intravitreal injection of PGJ₂ in a nonhuman primate model of nonarteritic anterior ischemic optic neuropathy.NG2 proteoglycan-dependent recruitment of tumor macrophages promotes pericyte-endothelial cell interactions required for brain tumor vascularization.Prox1 Inhibits Proliferation and Is Required for Differentiation of the Oligodendrocyte Cell Lineage in the Mouse.NG2, a common denominator for neuroinflammation, blood-brain barrier alteration, and oligodendrocyte precursor response in EAE, plays a role in dendritic cell activation Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.Early vascular deficits are correlated with delayed mammary tumorigenesis in the MMTV-PyMT transgenic mouse following genetic ablation of the NG2 proteoglycan.The reactions and role of NG2 glia in spinal cord injury.NG2 Proteoglycan-Dependent Contributions of Pericytes and Macrophages to Brain Tumor Vascularization and Progression.Proteoglycans and their roles in brain cancer.Cannabinoid receptor 2 suppresses leukocyte inflammatory migration by modulating the JNK/c-Jun/Alox5 pathway The translational biology of remyelination: past, present, and future.Glial kon/NG2 gene network for central nervous system repair.Roles of NG2-glia in ischemic stroke.Molecular mechanism of central nervous system repair by the Drosophila NG2 homologue kon-tiki.Apolipoprotein E Mimetic Promotes Functional and Histological Recovery in Lysolecithin-Induced Spinal Cord Demyelination in Mice.Role of NG2 proteoglycan in macrophage recruitment to brain tumors and sites of CNS demyelination.Distribution of corticosteroid receptors in mature oligodendrocytes and oligodendrocyte progenitors of the adult mouse brain.NG2-proteoglycan-dependent contributions of oligodendrocyte progenitors and myeloid cells to myelin damage and repair.Distinct NG2 proteoglycan-dependent roles of resident microglia and bone marrow-derived macrophages during myelin damage and repair.NG2 and NG2-positive cells delineate focal cerebral infarct demarcation in rats.Loss of Tuberous Sclerosis Complex1 in Adult Oligodendrocyte Progenitor Cells Enhances Axon Remyelination and Increases Myelin Thickness after a Focal Demyelination.ADAMTS-4 in oligodendrocytes contributes to myelination with an impact on motor function.Gene network underlying the glial regenerative response to central nervous system injury.The diversity and disparity of the glial scar.The distinct abilities of tube-formation and migration between brain and spinal cord microvascular pericytes in rats.Mesenchymal Stem Cell Protection of Neurons against Glutamate Excitotoxicity Involves Reduction of NMDA-Triggered Calcium Responses and Surface GluR1, and Is Partly Mediated by TNF.Lack of NG2 exacerbates neurological outcome and modulates glial responses after traumatic brain injury.Dissecting the multifactorial nature of demyelinating disease.

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P2860

Reduced inflammation accompanies diminished myelin damage and repair in the NG2 null mouse spinal cord.

http://www.wikidata.org/entity/Q35588967

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年论文

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name

Reduced inflammation accompani ...... he NG2 null mouse spinal cord.

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Reduced inflammation accompani ...... he NG2 null mouse spinal cord.

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Reduced inflammation accompani ...... he NG2 null mouse spinal cord.

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Reduced inflammation accompani ...... he NG2 null mouse spinal cord.

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Reduced inflammation accompani ...... he NG2 null mouse spinal cord.

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Journal of Neuroinflammation

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Reduced inflammation accompani ...... the NG2 null mouse spinal cord

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Andrej Boor

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Karolina Kucharova

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William B Stallcup

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Yunchao Chang

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P2860

NG2 proteoglycan promotes endothelial cell motility and angiogenesis via engagement of galectin-3 and alpha3beta1 integrin

Alternative activation of macrophages

A perivascular origin for mesenchymal stem cells in multiple human organs

Phosphorylation of NG2 proteoglycan by protein kinase C-alpha regulates polarized membrane distribution and cell motility

NG2 proteoglycan is expressed exclusively by mural cells during vascular morphogenesis

NG2, a member of chondroitin sulfate proteoglycans family mediates the inflammatory response of activated microglia

CXCR4 promotes differentiation of oligodendrocyte progenitors and remyelination

PDGF (alpha)-receptor is unresponsive to PDGF-AA in aortic smooth muscle cells from the NG2 knockout mouse.

Inflammation induced neurological handicap processes in multiple sclerosis: new insights from preclinical studies.

Importance of oligodendrocyte protection, BBB breakdown and inflammation for remyelination.

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