Retinal pericytes inhibit activated T cell proliferation - Wikidata - awgldk - TriplyDB

Retinal pericytes inhibit activated T cell proliferation

Retinal pericytes inhibit activated T cell proliferation

http://www.wikidata.org/entity/Q35595555

about

Renal pericytes: regulators of medullary blood flow Pericytes, an overlooked player in vascular pathobiology Immune Regulation by Pericytes: Modulating Innate and Adaptive Immunity Thioredoxin Interacting Protein (TXNIP) and Pathogenesis of Diabetic Retinopathy Pericytes at the intersection between tissue regeneration and pathology.Diabetes and retinal vascular dysfunction.Antibody-mediated retinal pericyte injury: implications for diabetic retinopathy.Neurotrauma: The Crosstalk between Neurotrophins and Inflammation in the Acutely Injured Brain.The role of antioxidation and immunomodulation in postnatal multipotent stem cell-mediated cardiac repair.Microvascular remodeling and wound healing: a role for pericytes.Could donor multipotent mesenchymal stromal cells prevent or delay the onset of diabetic retinopathy?Pericytes: brain-immune interface modulators.The complexity of cell composition of the intima of large arteries: focus on pericyte-like cells.Immune Functions and Properties of Resident Cells in the Heart and Cardiovascular System: Pericytes.The Importance of Pericytes in Healing: Wounds and other Pathologies.Immunoevasive pericytes from human pluripotent stem cells preferentially modulate induction of allogeneic regulatory T cells.Pericytes in Brain Injury and Repair After Ischemic Stroke.Differential regulation of pericyte function by the CXC receptor 3.Lipopolysaccharide activates Toll-like receptor 4 (TLR4)-mediated NF-κB signaling pathway and proinflammatory response in human pericytes Pericytes Make Spinal Cord Breathless after Injury.Pericytes modulate myelination in the central nervous system.Pericytes: The Role of Multipotent Stem Cells in Vascular Maintenance and Regenerative Medicine.Interferon-γ converts human microvascular pericytes into negative regulators of alloimmunity through induction of indoleamine 2,3-dioxygenase 1.How Plastic Are Pericytes?Perivascular cell αv integrins as a target to treat skeletal muscle fibrosis.Glioblastoma-activated pericytes support tumor growth via immunosuppression.Pericytes are heterogeneous in their origin within the same tissue.Molecular mechanisms underlying therapeutic potential of pericytes.

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P2860

Retinal pericytes inhibit activated T cell proliferation

http://www.wikidata.org/entity/Q35595555

description

2011 nî lūn-bûn

@nan

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

2011年论文

@zh

2011年论文

@zh-cn

name

Retinal pericytes inhibit activated T cell proliferation

@ast

Retinal pericytes inhibit activated T cell proliferation

@en

type

label

Retinal pericytes inhibit activated T cell proliferation

@ast

Retinal pericytes inhibit activated T cell proliferation

@en

prefLabel

Retinal pericytes inhibit activated T cell proliferation

@ast

Retinal pericytes inhibit activated T cell proliferation

@en

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Investigative Ophthalmology & Visual Science

P1476

Retinal pericytes inhibit activated T cell proliferation

@en

P2093

Dawn S Smith

http://www.w3.org/2001/XMLSchema#string

Feng Lin

http://www.w3.org/2001/XMLSchema#string

Nader Sheibani

http://www.w3.org/2001/XMLSchema#string

Suber Huang

http://www.w3.org/2001/XMLSchema#string

Timothy Kern

http://www.w3.org/2001/XMLSchema#string

Yan Li

http://www.w3.org/2001/XMLSchema#string

Zhidan Tu

http://www.w3.org/2001/XMLSchema#string

P2860

Inflammation, stress, and diabetes

Role of PDGF-B and PDGFR-beta in recruitment of vascular smooth muscle cells and pericytes during embryonic blood vessel formation in the mouse

Pericyte loss and microaneurysm formation in PDGF-B-deficient mice

Diabetic retinopathy: seeing beyond glucose-induced microvascular disease.

An activated form of transforming growth factor beta is produced by cocultures of endothelial cells and pericytes.

Pericytes and the pathogenesis of diabetic retinopathy.

PD-L1(hi) retinal pigment epithelium (RPE) cells elicited by inflammatory cytokines induce regulatory activity in uveitogenic T cells.

Decay accelerating factor can control T cell differentiation into IFN-gamma-producing effector cells via regulating local C5a-induced IL-12 production.

Attenuation of proliferation and migration of retinal pericytes in the absence of thrombospondin-1

Inflammation in diabetic retinopathy.

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9005-9010

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scholarly article

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10.1167/IOVS.11-8008

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12

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52

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2011-11-21T00:00:00Z

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51720068

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22003106

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3231798

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