Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis - Wikidata - awgldk - TriplyDB

Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis

Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis

http://www.wikidata.org/entity/Q35596103

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Central role of the gut epithelial barrier in the pathogenesis of chronic intestinal inflammation: lessons learned from animal models and human genetics Immunology of Crohn's Disease TRIM27 negatively regulates NOD2 by ubiquitination and proteasomal degradation The molecular chaperone HSP70 binds to and stabilizes NOD2, an important protein involved in Crohn disease A role for Erbin in the regulation of Nod2-dependent NF-kappaB signaling The intermediate filament protein, vimentin, is a regulator of NOD2 activity Function of Nod-like receptors in microbial recognition and host defense The human intestinal microbiome: a new frontier of human biology A chromosome 8 gene-cluster polymorphism with low human beta-defensin 2 gene copy number predisposes to Crohn disease of the colon Chronic inflammation: importance of NOD2 and NALP3 in interleukin-1beta generation Association of the T allele of an intronic single nucleotide polymorphism in the colony stimulating factor 1 receptor with Crohn's disease: a case-control study Role of Nucleotide-binding and Oligomerization Domain 2 Protein (NOD2) in the Development of Atherosclerosis Roles of NOD1 (NLRC1) and NOD2 (NLRC2) in innate immunity and inflammatory diseases NOD-Like Receptors: Master Regulators of Inflammation and Cancer Comparative genomic analysis of buffalo (Bubalus bubalis) NOD1 and NOD2 receptors and their functional role in in-vitro cellular immune response Nucleotide-oligomerization-domain-2 affects commensal gut microbiota composition and intracerebral immunopathology in acute Toxoplasma gondii induced murine ileitis Nod2: A Critical Regulator of Ileal Microbiota and Crohn's Disease Interleukin-12 and Th1 immune response in Crohn's disease: pathogenetic relevance and therapeutic implication CARD15/NOD2 mutations in Crohn's disease Evidence for the involvement of NOD2 in regulating colonic epithelial cell growth and survival The c-Jun N-terminal kinase (JNK)-binding protein (JNKBP1) acts as a negative regulator of NOD2 protein signaling by inhibiting its oligomerization process Genotypes and phenotypes in Crohn's disease: do they help in clinical management?A genomic view of the NOD-like receptor family in teleost fish: identification of a novel NLR subfamily in zebrafish.Disease phenotype and genotype are associated with shifts in intestinal-associated microbiota in inflammatory bowel diseases Mouse paneth cell secretory responses to cell surface glycolipids of virulent and attenuated pathogenic bacteria.Immune responses upon Campylobacter jejuni infection of secondary abiotic mice lacking nucleotide-oligomerization-domain-2.NOD1 contributes to mouse host defense against Helicobacter pylori via induction of type I IFN and activation of the ISGF3 signaling pathway NOD-like receptors in intestinal homeostasis and epithelial tissue repair.Induction and rescue of Nod2-dependent Th1-driven granulomatous inflammation of the ileum Nod2: a key regulator linking microbiota to intestinal mucosal immunity.Inflammatory bowel diseases phenotype, C. difficile and NOD2 genotype are associated with shifts in human ileum associated microbial composition.Host genes related to paneth cells and xenobiotic metabolism are associated with shifts in human ileum-associated microbial composition.Nucleotide oligomerization domain 2 polymorphisms in patients with intestinal failure.Bacterial interactions with cells of the intestinal mucosa: Toll-like receptors and NOD2 Nod1 and nod2 are expressed in human and murine renal tubular epithelial cells and participate in renal ischemia reperfusion injury.Crohn's disease: innate immunodeficiency?Influence of a nucleotide oligomerization domain 1 (NOD1) polymorphism and NOD2 mutant alleles on Crohn's disease phenotype NOD1 and NOD2: signaling, host defense, and inflammatory disease.Recipient's genetic R702W NOD2 variant is associated with an increased risk of bacterial infections after orthotopic liver transplantation.Critical design aspects involved in the study of Paneth cells and the intestinal microbiota.

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Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis

http://www.wikidata.org/entity/Q35596103

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2003年论文

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2003年论文

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2003年论文

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Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis

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Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis

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Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis

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Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis

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Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis

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Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis

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Expression of NOD2 in Paneth cells: a possible link to Crohn's ileitis

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E Zimmermann

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F F Chen

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G Nuñez

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J H Cho

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J K Greenson

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S Lala

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P2860

The contribution of NOD2 gene mutations to the risk and site of disease in inflammatory bowel disease

A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease

Nod2, a Nod1/Apaf-1 family member that is restricted to monocytes and activates NF-kappaB

Nod2 is a general sensor of peptidoglycan through muramyl dipeptide (MDP) detection

Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease

Regulation of intestinal alpha-defensin activation by the metalloproteinase matrilysin in innate host defense

Host recognition of bacterial muramyl dipeptide mediated through NOD2. Implications for Crohn's disease

Secretion of microbicidal alpha-defensins by intestinal Paneth cells in response to bacteria

Epidemiology of Crohn's disease and ulcerative colitis in a central Canadian province: a population-based study.

Human Nod1 confers responsiveness to bacterial lipopolysaccharides.

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