Confounding, ascertainment bias, and the blind quest for a genetic 'fountain of youth'.
about
The mathematical limits of genetic prediction for complex chronic diseaseA model of gene-gene and gene-environment interactions and its implications for targeting environmental interventions by genotypeTilting at quixotic trait loci (QTL): an evolutionary perspective on genetic causationWhat are genes "for" or where are traits "from"? What is the question?'Sifting the significance from the data' - the impact of high-throughput genomic technologies on human genetics and health careSize matters: just how big is BIG?: Quantifying realistic sample size requirements for human genome epidemiologyMendelian Randomization for Strengthening Causal Inference in Observational StudiesPrediction and interaction in complex disease genetics: experience in type 1 diabetes.Assessing the phenotypic effects in the general population of rare variants in genes for a dominant Mendelian form of diabetes.Haplotype analysis and identification of genes for a complex trait: examples from schizophrenia.On the validity of the likelihood ratio test and consistency of resulting parameter estimates in joint linkage and linkage disequilibrium analysis under improperly specified parametric models.Statistical tools for linkage analysis and genetic association studies.Translational research in central nervous system drug discovery.The balance between heritable and environmental aetiology of human disease.Genetic polymorphisms of the RAS-cytokine pathway and chronic kidney diseaseMapping translational research in personalized therapeutics: from molecular markers to health policy.SNP-specific extraction of haplotype-resolved targeted genomic regions.Common and rare forms of diabetes mellitus: towards a continuum of diabetes subtypes.The 'missing heritability' of common disorders: should health researchers care?Coming to terms with complexity: limits to a reductionist view of aging.Exploring the performance of Multifactor Dimensionality Reduction in large scale SNP studies and in the presence of genetic heterogeneity among epistatic disease models.Randomised by (your) god: robust inference from an observational study design.Design Considerations for Genetic Linkage and Association Studies.An utter refutation of the "fundamental theorem of the HapMap".How many genes underlie the occurrence of common complex diseases in the population?Relationships with Test-Tubes: Where's the Reciprocity?
P2860
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P2860
Confounding, ascertainment bias, and the blind quest for a genetic 'fountain of youth'.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Confounding, ascertainment bias, and the blind quest for a genetic 'fountain of youth'.
@ast
Confounding, ascertainment bias, and the blind quest for a genetic 'fountain of youth'.
@en
type
label
Confounding, ascertainment bias, and the blind quest for a genetic 'fountain of youth'.
@ast
Confounding, ascertainment bias, and the blind quest for a genetic 'fountain of youth'.
@en
prefLabel
Confounding, ascertainment bias, and the blind quest for a genetic 'fountain of youth'.
@ast
Confounding, ascertainment bias, and the blind quest for a genetic 'fountain of youth'.
@en
P2860
P1433
P1476
Confounding, ascertainment bias, and the blind quest for a genetic 'fountain of youth'.
@en
P2093
Joseph D Terwilliger
Kenneth M Weiss
P2860
P304
P356
10.1080/07853890310015181
P407
P577
2003-01-01T00:00:00Z