PhoQ mutations promote lipid A modification and polymyxin resistance of Pseudomonas aeruginosa found in colistin-treated cystic fibrosis patients
about
CXCL9 contributes to antimicrobial protection of the gut during citrobacter rodentium infection independent of chemokine-receptor signalingResponses of Pseudomonas aeruginosa to antimicrobialsNew amphiphilic neamine derivatives active against resistant Pseudomonas aeruginosa and their interactions with lipopolysaccharidesA divergent Pseudomonas aeruginosa palmitoyltransferase essential for cystic fibrosis-specific lipid A.Refinement of OprH-LPS Interactions by Molecular Simulations.Molecular Interactions of Lipopolysaccharide with an Outer Membrane Protein from Pseudomonas aeruginosa Probed by Solution NMR.Evolution of the Pseudomonas aeruginosa mutational resistome in an international Cystic Fibrosis cloneCharacterization of the polymyxin B resistome of Pseudomonas aeruginosa.Analysis of carbohydrates and glycoconjugates by matrix-assisted laser desorption/ionization mass spectrometry: An update for 2011-2012.Polymyxins: Antibacterial Activity, Susceptibility Testing, and Resistance Mechanisms Encoded by Plasmids or Chromosomes.The resistome of Pseudomonas aeruginosa in relationship to phenotypic susceptibilityRole of the Vibrio cholerae matrix protein Bap1 in cross-resistance to antimicrobial peptides.The ferrous iron-responsive BqsRS two-component system activates genes that promote cationic stress tolerance.Colistin resistance mechanisms in Klebsiella pneumoniae strains from Taiwan.PmrB mutations promote polymyxin resistance of Pseudomonas aeruginosa isolated from colistin-treated cystic fibrosis patientsIn Vivo Evolution of Bacterial Resistance in Two Cases of Enterobacter aerogenes Infections during Treatment with ImipenemFunctional Genome Screening to Elucidate the Colistin Resistance Mechanism.Impact of two-component regulatory systems PhoP-PhoQ and PmrA-PmrB on colistin pharmacodynamics in Pseudomonas aeruginosa.Second Generation Modifiers of Colistin Resistance Show Enhanced Activity and Lower Inherent ToxicityThe Pseudomonas aeruginosa PhoP-PhoQ two-component regulatory system is induced upon interaction with epithelial cells and controls cytotoxicity and inflammation.Microenvironmental characteristics and physiology of biofilms in chronic infections of CF patients are strongly affected by the host immune response.Polymyxin resistance of Pseudomonas aeruginosa phoQ mutants is dependent on additional two-component regulatory systems.Amino Acid Substitutions of CrrB Responsible for Resistance to Colistin through CrrC in Klebsiella pneumoniae.Rhesus θ-defensin-1 (RTD-1) exhibits in vitro and in vivo activity against cystic fibrosis strains of Pseudomonas aeruginosa.Crystal Structure of Escherichia coli originated MCR-1, a phosphoethanolamine transferase for Colistin Resistance.Rapid adaptation drives invasion of airway donor microbiota by Pseudomonas after lung transplantation.Bacterial stress responses as determinants of antimicrobial resistance.Colistin: an update on the antibiotic of the 21st century.Pseudomonas aeruginosa: new insights into pathogenesis and host defenses.Liposomal antibiotic formulations for targeting the lungs in the treatment of Pseudomonas aeruginosa.Stress responses as determinants of antimicrobial resistance in Pseudomonas aeruginosa: multidrug efflux and more.Mechanisms of polymyxin resistance: acquired and intrinsic resistance in bacteria.The Pseudomonas aeruginosa PAO1 Two-Component Regulator CarSR Regulates Calcium Homeostasis and Calcium-Induced Virulence Factor Production through Its Regulatory Targets CarO and CarP.In vivo therapeutic efficacy of frog skin-derived peptides against Pseudomonas aeruginosa-induced pulmonary infection.Lipopolysaccharide modification in Gram-negative bacteria during chronic infection.Prevalence and Molecular Characterization of mcr-1-Positive Salmonella Strains Recovered from Clinical Specimens in China.Genomics and Susceptibility Profiles of Extensively Drug-Resistant (XDR) Pseudomonas aeruginosa from Spain.Two-component systems required for virulence in Pseudomonas aeruginosa.Structural Modification of Lipopolysaccharide Conferred by mcr-1 in Gram-Negative ESKAPE Pathogens.The evolution of antimicrobial peptide resistance in Pseudomonas aeruginosa is shaped by strong epistatic interactions.
P2860
Q27321362-2BB54C0A-3D31-4EFB-AE61-DAB327EAD9E3Q28305587-A802094E-26A5-4193-9FB6-87AC98D41AA0Q28654498-5EF78B46-FAA3-430E-B796-4EE9479774DAQ30153476-F77DCF57-2065-4266-B24B-FA1DB7311961Q30274925-A3004C99-2399-4403-BF63-AD0194ECD106Q30276105-E156301D-A9EF-461A-9D93-8F33268712D1Q33911762-9B8364C0-94DD-4144-9EBB-9F55CE128B81Q34446329-BC06F67E-E4B4-417B-B4A7-F1A03A29627DQ34489424-690AB69C-185B-43D5-8E48-5B1F44B4212AQ34552966-C01A271A-4E32-4690-B9A1-9642C9E2A352Q34922676-7BEF0F9C-133B-4433-83C0-E622526E38A6Q35008777-77D51715-5855-4D48-9CD5-75C8C8D52D2DQ35172351-DBD0FF82-5E95-4569-8A33-4DD642AEC7A5Q35385177-BB439CBB-0B78-46CE-AD1A-F2411156BE5EQ35689189-690AA1DE-EF5C-4DF5-B81D-21E09E701DCAQ35785140-BD61F169-EC5C-4FA3-B56D-8A530D007AFAQ35961832-BC969072-EED6-44FD-810A-97BC41D76C75Q36018883-FE1D0D4A-346A-4595-B5CF-CD7D7AF9A00DQ36080468-AE329B72-4669-4F0B-B4FA-074E4429883BQ36163103-F2C83C76-001E-4931-A7E6-34DC450B220EQ36346231-975D6F95-3C1C-4A4B-A32A-023ED9EAFD9BQ36785850-2BCCBF12-2C7B-4C09-AAE9-BC5D3E39669EQ36933507-B6C4F141-981F-4B9D-9BB7-D31BBD4D2D37Q37223605-3237DDE7-4A30-43FA-B61B-632BCDD3755CQ37497892-D69F9E41-3FB3-482E-97FB-85C1047D1DB4Q37589273-04D1BDAE-FAC2-4C7B-8BCA-F5A1093F1315Q38012391-2B5926FA-A690-49E6-86D4-622AC2AED009Q38048852-95942582-D9E2-4D2C-BB4C-CF8753BE8AC3Q38101888-298F44A6-249A-4903-A427-0DC578863394Q38214155-C9DA2787-E2B2-49B2-BF71-C3495A79B7A9Q38267242-01303E27-9930-4867-A197-1B45FBDA1BD6Q38287949-FF975E44-50EF-4C18-8789-F0FA6A7BAEE4Q38294389-97F24C1D-0BB7-4068-9C19-A15E4A4AE76DQ38619135-137F1B45-A8BA-43CA-85C0-6EDB1C05D69DQ38807634-7373CECC-E35D-4D57-9848-125545EEA30CQ38963368-7AC0A70A-91CD-4F7F-B85F-6EF1B6970812Q40056971-4185E8D1-0CB2-4C6D-8360-06AFF3780EAFQ40210470-DD8F8604-30F8-4445-9B7F-582B9BE9B57CQ40266237-AC413B7D-B42A-41F0-BBB2-9264D3B0B7CFQ40519553-A3152025-1D4D-4024-B11E-84BC620C42CE
P2860
PhoQ mutations promote lipid A modification and polymyxin resistance of Pseudomonas aeruginosa found in colistin-treated cystic fibrosis patients
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
PhoQ mutations promote lipid A ...... eated cystic fibrosis patients
@ast
PhoQ mutations promote lipid A ...... eated cystic fibrosis patients
@en
type
label
PhoQ mutations promote lipid A ...... eated cystic fibrosis patients
@ast
PhoQ mutations promote lipid A ...... eated cystic fibrosis patients
@en
prefLabel
PhoQ mutations promote lipid A ...... eated cystic fibrosis patients
@ast
PhoQ mutations promote lipid A ...... eated cystic fibrosis patients
@en
P2093
P2860
P356
P1476
PhoQ mutations promote lipid A ...... eated cystic fibrosis patients
@en
P2093
Amanda K Miller
Laurel Stevens
Mark K Brannon
Samuel I Miller
Samuel M Moskowitz
Sara E Selgrade
P2860
P304
P356
10.1128/AAC.05391-11
P407
P577
2011-10-03T00:00:00Z