Respective contributions of maternal insulin resistance and diet to metabolic and hypothalamic phenotypes of progeny - Wikidata - awgldk - TriplyDB

Respective contributions of maternal insulin resistance and diet to metabolic and hypothalamic phenotypes of progeny

Respective contributions of maternal insulin resistance and diet to metabolic and hypothalamic phenotypes of progeny

http://www.wikidata.org/entity/Q35602673

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Developmental influences on circuits programming susceptibility to obesity Embryonic development of the hypothalamic feeding circuitry: Transcriptional, nutritional, and hormonal influences Developmental specification of metabolic circuitry The maternal womb: a novel target for cancer prevention in the era of the obesity pandemic?Long-lived crowded-litter mice exhibit lasting effects on insulin sensitivity and energy homeostasis.InsR/FoxO1 signaling curtails hypothalamic POMC neuron number.Roles of the placenta in fetal brain development Maternal diabetes compromises the organization of hypothalamic feeding circuits and impairs leptin sensitivity in offspring.Developmental programming in response to maternal overnutrition Feed your head: neurodevelopmental control of feeding and metabolism Early determinants of obesity: genetic, epigenetic, and in utero influences.Intergenerational programming of metabolic disease: evidence from human populations and experimental animal models.Reducing maternal weight improves offspring metabolism and alters (or modulates) methylation.Maternal obesity and diabetes induces latent metabolic defects and widespread epigenetic changes in isogenic mice.Developmental programming by maternal insulin resistance: hyperinsulinemia, glucose intolerance, and dysregulated lipid metabolism in male offspring of insulin-resistant mice.Metabolic programming of insulin action and secretion.Patterning, specification, and differentiation in the developing hypothalamus.Obesity Pathogenesis: An Endocrine Society Scientific Statement.

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Respective contributions of maternal insulin resistance and diet to metabolic and hypothalamic phenotypes of progeny

http://www.wikidata.org/entity/Q35602673

description

2010 nî lūn-bûn

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2010年の論文

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2010年論文

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Jill S Carmody

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Lori M Zeltser

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Phyllis Wan

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Rudolph L Leibel

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Direct insulin and leptin action on pro-opiomelanocortin neurons is required for normal glucose homeostasis and fertility

The thrifty phenotype hypothesis

Prevalence of overweight and obesity in the United States, 1999-2004

Maternal and postweaning diet interaction alters hypothalamic gene expression and modulates response to a high-fat diet in male offspring

Critical determinants of hypothalamic appetitive neuropeptide development and expression: species considerations.

Segregation of acute leptin and insulin effects in distinct populations of arcuate proopiomelanocortin neurons.

Effect of diabetes in pregnancy on offspring: follow-up research in the Pima Indians.

Childhood obesity, other cardiovascular risk factors, and premature death

Intra-uterine programming of the endocrine pancreas.

Muscle-specific PPARgamma-deficient mice develop increased adiposity and insulin resistance but respond to thiazolidinediones.

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492-499

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10.1038/OBY.2010.245

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Domenico Accili

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