Death receptor 5 signaling promotes hepatocyte lipoapoptosis - Wikidata - awgldk - TriplyDB

Death receptor 5 signaling promotes hepatocyte lipoapoptosis

Death receptor 5 signaling promotes hepatocyte lipoapoptosis

http://www.wikidata.org/entity/Q35604362

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Depolarization Controls TRAIL-Sensitization and Tumor-Selective Killing of Cancer Cells: Crosstalk with ROS ER stress regulates myeloid-derived suppressor cell fate through TRAIL-R-mediated apoptosis Novel phosphorylation and ubiquitination sites regulate reactive oxygen species-dependent degradation of anti-apoptotic c-FLIP protein Non-alcoholic fatty liver disease, vascular inflammation and insulin resistance are exacerbated by TRAIL deletion in mice Role of white adipose lipolysis in the development of NASH induced by methionine- and choline-deficient diet.Vismodegib suppresses TRAIL-mediated liver injury in a mouse model of nonalcoholic steatohepatitis.Death Receptor-Mediated Cell Death and Proinflammatory Signaling in Nonalcoholic Steatohepatitis.Role of fibroblast growth factor 21 in the early stage of NASH induced by methionine- and choline-deficient diet Palmitate Inhibits SIRT1-Dependent BMAL1/CLOCK Interaction and Disrupts Circadian Gene Oscillations in Hepatocytes.Cell death and cell death responses in liver disease: mechanisms and clinical relevance Toyocamycin attenuates free fatty acid-induced hepatic steatosis and apoptosis in cultured hepatocytes and ameliorates nonalcoholic fatty liver disease in mice.RNASET2 is required for ROS propagation during oxidative stress-mediated cell death.Myristic acid potentiates palmitic acid-induced lipotoxicity and steatohepatitis associated with lipodystrophy by sustaning de novo ceramide synthesis.Molecular mechanisms of lipotoxicity and glucotoxicity in nonalcoholic fatty liver disease.ONC201 kills solid tumor cells by triggering an integrated stress response dependent on ATF4 activation by specific eIF2α kinases.Apoptosis and necrosis in the liver Functional TRAIL receptors in monocytes and tumor-associated macrophages: A possible targeting pathway in the tumor microenvironment Chemoprevention of nonalcoholic fatty liver disease by dietary natural compounds.Lipotoxicity pathways intersect in hepatocytes: Endoplasmic reticulum stress, c-Jun N-terminal kinase-1, and death receptors.Lipotoxic lethal and sublethal stress signaling in hepatocytes: relevance to NASH pathogenesis.Lipid-Induced Signaling Causes Release of Inflammatory Extracellular Vesicles From Hepatocytes.Degradation of Keap1 activates BH3-only proteins Bim and PUMA during hepatocyte lipoapoptosis Saturated free fatty acids induce cholangiocyte lipoapoptosis.Liver inflammation and fibrosis.Degradation of cIAPs contributes to hepatocyte lipoapoptosis.A hedgehog survival pathway in 'undead' lipotoxic hepatocytes.TRAIL receptor deletion in mice suppresses the inflammation of nutrient excess.RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditions.Hepatitis C Virus Infection Increases c-Jun N-Terminal Kinase (JNK) Phosphorylation and Accentuates Hepatocyte Lipoapoptosis.Lack of fibroblast growth factor 21 accelerates metabolic liver injury characterized by steatohepatities in mice.Therapeutic opportunities for alcoholic steatohepatitis and nonalcoholic steatohepatitis: exploiting similarities and differences in pathogenesis.Cell culture models of fatty acid overload: Problems and solutions.Fisetin Induces Apoptosis Through p53-Mediated Up-Regulation of DR5 Expression in Human Renal Carcinoma Caki Cells.Human Protein Kinases and Obesity.Growth arrest and DNA damage-inducible 45α protects against nonalcoholic steatohepatitis induced by methionine- and choline-deficient diet.The pan-caspase inhibitor Emricasan (IDN-6556) decreases liver injury and fibrosis in a murine model of non-alcoholic steatohepatitis.Non-Alcoholic Fatty Liver Disease.Geranylgeranylacetone attenuates fibrogenic activity and induces apoptosis in cultured human hepatic stellate cells and reduces liver fibrosis in carbon tetrachloride-treated mice.TRAIL Deletion Prevents Liver, but Not Adipose Tissue, Inflammation during Murine Diet-Induced Obesity.To die or not to die: death signaling in nonalcoholic fatty liver disease.

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P2860

Death receptor 5 signaling promotes hepatocyte lipoapoptosis

http://www.wikidata.org/entity/Q35604362

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

@zh-tw

2011年论文

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2011年论文

@zh

2011年论文

@zh-cn

name

Death receptor 5 signaling promotes hepatocyte lipoapoptosis

@ast

Death receptor 5 signaling promotes hepatocyte lipoapoptosis

@en

type

label

Death receptor 5 signaling promotes hepatocyte lipoapoptosis

@ast

Death receptor 5 signaling promotes hepatocyte lipoapoptosis

@en

prefLabel

Death receptor 5 signaling promotes hepatocyte lipoapoptosis

@ast

Death receptor 5 signaling promotes hepatocyte lipoapoptosis

@en

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JBC.M111.280420

P1433

Journal of Biological Chemistry

P1476

Death receptor 5 signaling promotes hepatocyte lipoapoptosis

@en

P2093

Christian D Fingas

http://www.w3.org/2001/XMLSchema#string

Justin L Mott

http://www.w3.org/2001/XMLSchema#string

Nathan W Werneburg

http://www.w3.org/2001/XMLSchema#string

Niklas Finnberg

http://www.w3.org/2001/XMLSchema#string

Scott H Kaufmann

http://www.w3.org/2001/XMLSchema#string

Sophie C Cazanave

http://www.w3.org/2001/XMLSchema#string

Steven F Bronk

http://www.w3.org/2001/XMLSchema#string

Wafik S El-Deiry

http://www.w3.org/2001/XMLSchema#string

X Wei Meng

http://www.w3.org/2001/XMLSchema#string

P2860

CHOP is involved in endoplasmic reticulum stress-induced apoptosis by enhancing DR5 expression in human carcinoma cells

Role of endoplasmic reticulum stress in alpha-TEA mediated TRAIL/DR5 death receptor dependent apoptosis

N-terminal fragment of c-FLIP(L) processed by caspase 8 specifically interacts with TRAF2 and induces activation of the NF-kappaB signaling pathway

ER stress triggers apoptosis by activating BH3-only protein Bim

Nonalcoholic fatty liver disease

A real-time view of life within 100 nm of the plasma membrane.

Deletion of Fas in adipocytes relieves adipose tissue inflammation and hepatic manifestations of obesity in mice.

DR5 knockout mice are compromised in radiation-induced apoptosis

Reduced endoplasmic reticulum luminal calcium links saturated fatty acid-mediated endoplasmic reticulum stress and cell death in liver cells.

Palmitoylation of the TRAIL receptor DR4 confers an efficient TRAIL-induced cell death signalling.

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39336-39348

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scholarly article

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10.1074/JBC.M111.280420

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P407

P433

45

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P478

286

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P50

Gregory J Gores

P577

2011-09-22T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

21941003

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3234758

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