TGF-β1 signaling and Krüppel-like factor 10 regulate bone marrow-derived proangiogenic cell differentiation, function, and neovascularization. - Wikidata - awgldk - TriplyDB

TGF-β1 signaling and Krüppel-like factor 10 regulate bone marrow-derived proangiogenic cell differentiation, function, and neovascularization.

TGF-β1 signaling and Krüppel-like factor 10 regulate bone marrow-derived proangiogenic cell differentiation, function, and neovascularization.

http://www.wikidata.org/entity/Q35608186

about

Regulation of EPCs: The Gateway to Blood Vessel Formation Regulation of an inflammatory disease: Krüppel-like factors and atherosclerosis Infantile Hemangioma Originates From A Dysregulated But Not Fully Transformed Multipotent Stem Cell.Mobilization of CD34+-progenitor cells in patients with severe trauma.Genetic variants of Adam17 differentially regulate TGFβ signaling to modify vascular pathology in mice and humans.Effect of vitamin D on endothelial progenitor cells function Biology and flow cytometry of proangiogenic hematopoietic progenitors cells.Pathways mediating the interaction between endothelial progenitor cells (EPCs) and platelets KLF4 Promotes Angiogenesis by Activating VEGF Signaling in Human Retinal Microvascular Endothelial Cells.Endothelial progenitor cell: a blood cell by many other names may serve similar functions Klf10 regulates odontoblast differentiation and mineralization via promoting expression of dentin matrix protein 1 and dentin sialophosphoprotein genes Bone marrow-derived Kruppel-like factor 10 controls reendothelialization in response to arterial injury.Different but synergistic effects of bone marrow-derived VEGFR2+ and VEGFR2-CD45+ cells during hepatocellular carcinoma progression.Kruppel-like factors and vascular inflammation: implications for atherosclerosis.Krüppel-like factors in mammalian stem cells and development.Proteome variations in pancreatic stellate cells upon stimulation with proinflammatory factors.Bone regeneration via novel macroporous CPC scaffolds in critical-sized cranial defects in rats.Aging and the Krüppel-like factors.KLF10 as a Tumor Suppressor Gene and Its TGF-β Signaling.Amniotic Epithelial Cells Accelerate Diabetic Wound Healing by Modulating Inflammation and Promoting Neovascularization

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TGF-β1 signaling and Krüppel-like factor 10 regulate bone marrow-derived proangiogenic cell differentiation, function, and neovascularization.

http://www.wikidata.org/entity/Q35608186

description

2011 nî lūn-bûn

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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TGF-β1 signaling and Krüppel-l ...... ction, and neovascularization.

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TGF-β1 signaling and Krüppel-l ...... ction, and neovascularization.

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Akm Khyrul Wara

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Anthony Rosenzweig

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Basak Icli

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Dorit Leshem-Lev

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Eli I Lev

http://www.w3.org/2001/XMLSchema#string

Fehim Esen

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Jung-Soo Lee

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Kevin Croce

http://www.w3.org/2001/XMLSchema#string

Malayannan Subramaniam

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Mark A Creager

http://www.w3.org/2001/XMLSchema#string

P2860

TIEG1 null mouse-derived osteoblasts are defective in mineralization and in support of osteoclast differentiation in vitro

Therapeutic stem and progenitor cell transplantation for organ vascularization and regeneration

Pericyte loss and microaneurysm formation in PDGF-B-deficient mice

Mechanism of TGF-beta signaling to growth arrest, apoptosis, and epithelial-mesenchymal transition.

Clinical applications of blood-derived and marrow-derived stem cells for nonmalignant diseases.

Krüppel-like factor 4 regulates macrophage polarization.

Role of Kruppel-like factors in leukocyte development, function, and disease

IFN-gamma acts as anti-angiogenic cytokine in the human cornea by regulating the expression of VEGF-A and sVEGF-R1

Role of TIEG1 in biological processes and disease states.

The Kruppel-like factor KLF4 is a critical regulator of monocyte differentiation.

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6450-6460

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10.1182/BLOOD-2011-06-363713

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51558452

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21828131

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neovascularization

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3236126

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