A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway. - Wikidata - awgldk - TriplyDB

A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.

A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.

http://www.wikidata.org/entity/Q35614774

about

Recognition of Extracellular Bacteria by NLRs and Its Role in the Development of Adaptive Immunity Interactions of Salmonella with animals and plants Salmonella enterica Serovar Typhi conceals the invasion-associated type three secretion system from the innate immune system by gene regulation.Type three secretion system-mediated escape of Burkholderia pseudomallei into the host cytosol is critical for the activation of NFκB Human genome-wide RNAi screen for host factors that modulate intracellular Salmonella growth.Role of the gut microbiota in inflammatory bowel disease pathogenesis: what have we learnt in the past 10 years?Emerging significance of NLRs in inflammatory bowel disease.Receptor interacting protein-2 contributes to host defense against Anaplasma phagocytophilum infection.Manipulation of small Rho GTPases is a pathogen-induced process detected by NOD1.The actin-polymerizing activity of SipA is not essential for Salmonella enterica serovar Typhimurium-induced mucosal inflammation NOD1 and NOD2 signalling links ER stress with inflammation.Salmonellae interactions with host processes.The Gdac1 locus modifies spontaneous and Salmonella-induced colitis in mice deficient in either Gpx2 or Gpx1 gene.Detection of enteric pathogens by the nodosome.A Legionella effector modulates host cytoskeletal structure by inhibiting actin polymerization The role of innate immune signals in immunity to Brucella abortus.NOD1 and NOD2 Signaling in Infection and Inflammation.The Interplay between NLRs and Autophagy in Immunity and Inflammation.Interactions between Nod-Like Receptors and Intestinal Bacteria.Gene expression in the chicken caecum in response to infections with non-typhoid Salmonella.Insights into the molecular basis of the NOD2 signalling pathway.Single-cell analyses reveal an attenuated NF-κB response in the Salmonella-infected fibroblast.NOD1 and NOD2: Beyond Peptidoglycan Sensing.RIP2/RICK-dependent cytokine production upon Yersinia enterocolitica infection in macrophages with TLR4 deficiency.LipL21 lipoprotein binding to peptidoglycan enables Leptospira interrogans to escape NOD1 and NOD2 recognition.Precision editing of the gut microbiota ameliorates colitis.MyD88 signaling promotes both mucosal homeostatic and fibrotic responses during Salmonella-induced colitis.Minimal SPI1-T3SS effector requirement for Salmonella enterocyte invasion and intracellular proliferation in vivo.Impact of Salmonella enterica Type III Secretion System Effectors on the Eukaryotic Host Cell

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P2860

A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.

http://www.wikidata.org/entity/Q35614774

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

2011年论文

@zh

2011年论文

@zh-cn

name

A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.

@ast

A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.

@en

type

label

A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.

@ast

A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.

@en

prefLabel

A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.

@ast

A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway.

@en

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P1476

A Salmonella virulence factor activates the NOD1/NOD2 signaling pathway

@en

P2093

A Marijke Keestra

http://www.w3.org/2001/XMLSchema#string

Anita Kim

http://www.w3.org/2001/XMLSchema#string

Daisy Klein-Douwel

http://www.w3.org/2001/XMLSchema#string

Maria G Winter

http://www.w3.org/2001/XMLSchema#string

Sebastian E Winter

http://www.w3.org/2001/XMLSchema#string

P2860

IRAK (Pelle) family member IRAK-2 and MyD88 as proximal mediators of IL-1 signaling

Salmonella Typhimurium type III secretion effectors stimulate innate immune responses in cultured epithelial cells

An induced proximity model for NF-kappa B activation in the Nod1/RICK and RIP signaling pathways

Toll-like receptor (TLR) 2 and TLR5, but not TLR4, are required for Helicobacter pylori-induced NF-kappa B activation and chemokine expression by epithelial cells

CARD4/Nod1 mediates NF-kappaB and JNK activation by invasive Shigella flexneri.

Cytoplasmic flagellin activates caspase-1 and secretion of interleukin 1beta via Ipaf

MyD88: an adapter that recruits IRAK to the IL-1 receptor complex

Construction of versatile low-copy-number vectors for cloning, sequencing and gene expression in Escherichia coli

Gut inflammation provides a respiratory electron acceptor for Salmonella

Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

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scholarly article

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10.1128/MBIO.00266-11

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6

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2

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Mariana Xavier Byndloss

Renée M Tsolis

Andreas J. Bäumler

P577

2011-12-20T00:00:00Z

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22186610

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3238304

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