N-terminal domain of soluble epoxide hydrolase negatively regulates the VEGF-mediated activation of endothelial nitric oxide synthase.
about
Arachidonic acid monooxygenase: Genetic and biochemical approaches to physiological/pathophysiological relevanceOpposite effects of gene deficiency and pharmacological inhibition of soluble epoxide hydrolase on cardiac fibrosisThe 2014 Bernard B. Brodie award lecture-epoxide hydrolases: drug metabolism to therapeutics for chronic painDevelopment of an HTS assay for EPHX2 phosphatase activity and screening of nontargeted librariesRenal Ischemia/Reperfusion Injury in Soluble Epoxide Hydrolase-Deficient MiceVascular Endothelial Over-Expression of Human Soluble Epoxide Hydrolase (Tie2-sEH Tr) Attenuates Coronary Reactive Hyperemia in Mice: Role of Oxylipins and ω-HydroxylasesRole of soluble epoxide hydrolase phosphatase activity in the metabolism of lysophosphatidic acids.Combustion smoke-induced inflammation in the olfactory bulb of adult rats.Role of phosphatase activity of soluble epoxide hydrolase in regulating simvastatin-activated endothelial nitric oxide synthase.Soluble epoxide hydrolase null mice exhibit female and male differences in regulation of vascular homeostasisMechanism of the sex difference in neuronal ischemic cell deathVascular endothelial overexpression of human CYP2J2 (Tie2-CYP2J2 Tr) modulates cardiac oxylipin profiles and enhances coronary reactive hyperemia in mice.Temporal patterns of tyrosine nitration in embryo heart development.The role of receptor tyrosine kinase activation in cholangiocytes and pulmonary vascular endothelium in experimental hepatopulmonary syndrome.Endothelial dysfunction and vascular disease - a 30th anniversary update.Pharmacological profile and efficiency in vivo of diflapolin, the first dual inhibitor of 5-lipoxygenase-activating protein and soluble epoxide hydrolase.Reduced coronary reactive hyperemia in mice was reversed by the soluble epoxide hydrolase inhibitor (t-AUCB): Role of adenosine A2A receptor and plasma oxylipins.Endothelial Nox4-based NADPH oxidase regulates atherosclerosis via soluble epoxide hydrolase.Deletion or inhibition of soluble epoxide hydrolase protects against brain damage and reduces microglia-mediated neuroinflammation in traumatic brain injury.Blockade of soluble epoxide hydrolase attenuates post-ischemic neuronal hyperexcitation and confers resilience against stroke with TrkB activation.Bacterial Expression and HTS Assessment of Soluble Epoxide Hydrolase Phosphatase.
P2860
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P2860
N-terminal domain of soluble epoxide hydrolase negatively regulates the VEGF-mediated activation of endothelial nitric oxide synthase.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
N-terminal domain of soluble e ...... thelial nitric oxide synthase.
@ast
N-terminal domain of soluble e ...... thelial nitric oxide synthase.
@en
type
label
N-terminal domain of soluble e ...... thelial nitric oxide synthase.
@ast
N-terminal domain of soluble e ...... thelial nitric oxide synthase.
@en
prefLabel
N-terminal domain of soluble e ...... thelial nitric oxide synthase.
@ast
N-terminal domain of soluble e ...... thelial nitric oxide synthase.
@en
P2093
P2860
P50
P356
P1476
N-terminal domain of soluble e ...... othelial nitric oxide synthase
@en
P2093
Bruce D Hammock
Christophe Morisseau
Hsin-Han Hou
Jin-Feng Zhao
Kou-Hui Su
Susumu Imaoka
P2860
P304
P356
10.1093/CVR/CVR267
P577
2011-11-08T00:00:00Z