Amiodarone exposure during modest inflammation induces idiosyncrasy-like liver injury in rats: role of tumor necrosis factor-alpha.
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Drug-induced hepatotoxicity: metabolic, genetic and immunological basisComedications alter drug-induced liver injury reporting frequency: Data mining in the WHO VigiBase™.Trovafloxacin enhances lipopolysaccharide-stimulated production of tumor necrosis factor-α by macrophages: role of the DNA damage response.Ginseng alleviates cyclophosphamide-induced hepatotoxicity via reversing disordered homeostasis of glutathione and bile acid.Tumor necrosis factor-alpha potentiates the cytotoxicity of amiodarone in Hepa1c1c7 cells: roles of caspase activation and oxidative stressRoles of the hemostatic system and neutrophils in liver injury from co-exposure to amiodarone and lipopolysaccharide.Chlorpromazine-induced hepatotoxicity during inflammation is mediated by TIRAP-dependent signaling pathway in miceInflammatory stress and idiosyncratic hepatotoxicity: hints from animal models.Molecular mechanisms of hepatocellular apoptosis induced by trovafloxacin-tumor necrosis factor-alpha interaction.Dexamethasone Pretreatment Alleviates Isoniazid/Lipopolysaccharide Hepatotoxicity: Inhibition of Inflammatory and Oxidative Stress.Trovafloxacin potentiation of lipopolysaccharide-induced tumor necrosis factor release from RAW 264.7 cells requires extracellular signal-regulated kinase and c-Jun N-Terminal Kinase.Current challenges and controversies in drug-induced liver injury."Autoimmune(-Like)" Drug and Herb Induced Liver Injury: New Insights into Molecular Pathogenesis.Development of a rat model of D-galactosamine/lipopolysaccharide induced hepatorenal syndrome.Synergistic Cytotoxicity from Drugs and Cytokines In Vitro as an Approach to Classify Drugs According to Their Potential to Cause Idiosyncratic Hepatotoxicity: A Proof-of-Concept Study.Investigating the CYP2E1 Potential Role in the Mechanisms Behind INH/LPS-Induced Hepatotoxicity.Model-based identification of TNFα-induced IKKβ-mediated and IκBα-mediated regulation of NFκB signal transduction as a tool to quantify the impact of drug-induced liver injury compounds.
P2860
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P2860
Amiodarone exposure during modest inflammation induces idiosyncrasy-like liver injury in rats: role of tumor necrosis factor-alpha.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Amiodarone exposure during mod ...... f tumor necrosis factor-alpha.
@ast
Amiodarone exposure during mod ...... f tumor necrosis factor-alpha.
@en
type
label
Amiodarone exposure during mod ...... f tumor necrosis factor-alpha.
@ast
Amiodarone exposure during mod ...... f tumor necrosis factor-alpha.
@en
prefLabel
Amiodarone exposure during mod ...... f tumor necrosis factor-alpha.
@ast
Amiodarone exposure during mod ...... f tumor necrosis factor-alpha.
@en
P2093
P2860
P356
P1476
Amiodarone exposure during mod ...... f tumor necrosis factor-alpha.
@en
P2093
A Daniel Jones
Jack R Harkema
Jingtao Lu
Patricia E Ganey
Robert A Roth
P2860
P304
P356
10.1093/TOXSCI/KFR266
P577
2011-10-09T00:00:00Z