Quantitating the magnitude of the lymphocytic choriomeningitis virus-specific CD8 T-cell response: it is even bigger than we thought.
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Therapeutic antiviral T cells noncytopathically clear persistently infected microglia after conversion into antigen-presenting cellsRates of CTL killing in persistent viral infection in vivoInflammation-associated nitrotyrosination affects TCR recognition through reduced stability and alteration of the molecular surface of the MHC complexBlimp-1-mediated CD4 T cell exhaustion causes CD8 T cell dysfunction during chronic toxoplasmosis.Type I interferon suppresses virus-specific B cell responses by modulating CD8(+) T cell differentiationEstimating the diversity, completeness, and cross-reactivity of the T cell repertoire.Tracking the total CD8 T cell response to infection reveals substantial discordance in magnitude and kinetics between inbred and outbred hostsMyD88 plays a critical T cell-intrinsic role in supporting CD8 T cell expansion during acute lymphocytic choriomeningitis virus infection.Absence of mouse 2B4 promotes NK cell-mediated killing of activated CD8+ T cells, leading to prolonged viral persistence and altered pathogenesis.Coverage of related pathogenic species by multivalent and cross-protective vaccine design: arenaviruses as a model systemCNS recruitment of CD8+ T lymphocytes specific for a peripheral virus infection triggers neuropathogenesis during polymicrobial challenge.Analysis of A47, an immunoprevalent protein of vaccinia virus, leads to a reevaluation of the total antiviral CD8+ T cell response.IFN-λ exerts opposing effects on T cell responses depending on the chronicity of the virus infection.FOXO3 regulates CD8 T cell memory by T cell-intrinsic mechanisms.Regulation of memory CD8 T-cell differentiation by cyclin-dependent kinase inhibitor p27Kip1Autophagy is essential for effector CD8(+) T cell survival and memory formation.Migration of cytotoxic lymphocytes in cell cycle permits local MHC I-dependent control of division at sites of viral infectionEngagement of NKG2D on bystander memory CD8 T cells promotes increased immunopathology following Leishmania major infectionIntertwined pathways of programmed cell death in immunity.Depletion of alveolar macrophages ameliorates virus-induced disease following a pulmonary coronavirus infection.Diet-induced obesity does not impact the generation and maintenance of primary memory CD8 T cellsQuantifying antigen-specific CD4 T cells during a viral infection: CD4 T cell responses are larger than we think.IFN-alpha beta and self-MHC divert CD8 T cells into a distinct differentiation pathway characterized by rapid acquisition of effector functionsPolymicrobial Sepsis Increases Susceptibility to Chronic Viral Infection and Exacerbates CD8+ T Cell Exhaustion.The Timing of Stimulation and IL-2 Signaling Regulate Secondary CD8 T Cell Responses.The CD8+ T-cell response to lymphocytic choriomeningitis virus involves the L antigen: uncovering new tricks for an old virusNaive precursor frequencies and MHC binding rather than the degree of epitope diversity shape CD8+ T cell immunodominanceRegulation of innate CD8+ T-cell activation mediated by cytokines.Inflammatory IL-15 is required for optimal memory T cell responses.Dicer Regulates the Balance of Short-Lived Effector and Long-Lived Memory CD8 T Cell Lineages.Negative impact of IFN-γ on early host immune responses to retroviral infection.Intradermal NKT cell activation during DNA priming in heterologous prime-boost vaccination enhances T cell responses and protection against LeishmaniaUse of the CD107 mobilization assay reveals that cytotoxic T lymphocytes with novel MHC-Ib restriction are activated during Listeria monocytogenes infection.Naive CD4(+) T cell frequency varies for different epitopes and predicts repertoire diversity and response magnitude.CD70 deficiency impairs effector CD8 T cell generation and viral clearance but is dispensable for the recall response to lymphocytic choriomeningitis virus.Cytokine-mediated programmed proliferation of virus-specific CD8(+) memory T cells.A Context-Dependent Role for IL-21 in Modulating the Differentiation, Distribution, and Abundance of Effector and Memory CD8 T Cell Subsets.Sustained and incomplete recovery of naive CD8+ T cell precursors after sepsis contributes to impaired CD8+ T cell responses to infectionNon-invasive imaging of adaptive immunity using positron emission tomography.The great balancing act: regulation and fate of antiviral T-cell interactions
P2860
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P2860
Quantitating the magnitude of the lymphocytic choriomeningitis virus-specific CD8 T-cell response: it is even bigger than we thought.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
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2006年论文
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name
Quantitating the magnitude of ...... s even bigger than we thought.
@ast
Quantitating the magnitude of ...... s even bigger than we thought.
@en
type
label
Quantitating the magnitude of ...... s even bigger than we thought.
@ast
Quantitating the magnitude of ...... s even bigger than we thought.
@en
prefLabel
Quantitating the magnitude of ...... s even bigger than we thought.
@ast
Quantitating the magnitude of ...... s even bigger than we thought.
@en
P2093
P2860
P356
P1433
P1476
Quantitating the magnitude of ...... s even bigger than we thought.
@en
P2093
David Masopust
Kaja Murali-Krishna
Rafi Ahmed
P2860
P304
P356
10.1128/JVI.01459-06
P407
P577
2006-12-06T00:00:00Z