No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

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Tumor necrosis factor-α synthesis inhibitor, 3,6'-dithiothalidomide, reverses behavioral impairments induced by minimal traumatic brain injury in mice

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Q30520539-95BD62B0-195E-41D2-AC1E-AB0FFBE860CE

P2860

No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

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http://www.wikidata.org/entity/Q35637868

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2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

@zh-hk

2006年論文

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2006年論文

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2006年论文

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2006年论文

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2006年论文

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name

No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

@ast

No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

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type

Item

label

No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

@ast

No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

@en

prefLabel

No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

@ast

No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

@en

P1476

No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

@en

P2093

Brundin L

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Khademi M

http://www.w3.org/2001/XMLSchema#string

Lampa J

http://www.w3.org/2001/XMLSchema#string

Wallström E

http://www.w3.org/2001/XMLSchema#string

P2860

Demyelination occurring during anti-tumor necrosis factor alpha therapy for inflammatory arthritides.

Anti-TNF alpha therapy of rheumatoid arthritis: what have we learned?

Chronic exposure to tumor necrosis factor (TNF) in vitro impairs the activation of T cells through the T cell receptor/CD3 complex; reversal in vivo by anti-TNF antibodies in patients with rheumatoid arthritis.

T Cell Ig- and mucin-domain-containing molecule-3 (TIM-3) and TIM-1 molecules are differentially expressed on human Th1 and Th2 cells and in cerebrospinal fluid-derived mononuclear cells in multiple sclerosis.

Three or more routes for leukocyte migration into the central nervous system.

Increased peripheral T cell reactivity to microbial antigens and collagen type II in rheumatoid arthritis after treatment with soluble TNFalpha receptors.

Chronic tumor necrosis factor alters T cell responses by attenuating T cell receptor signaling.

Autologous and allogeneic MLC-reactivity in patients with rheumatoid arthritis.

Treatment of multiple sclerosis with gamma interferon: exacerbations associated with activation of the immune system.

Central nervous system nitric oxide formation in cerebral systemic lupus erythematosus.

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1237-1240

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scholarly article

case report

P356

10.1136/ARD.2006.055194

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P407

English

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http://www.w3.org/2001/XMLSchema#string

P478

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Elisabet Svenungsson

Mats Andersson

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2006-04-10T00:00:00Z

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P5875

7172714

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P698

16606644

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P921

cerebrospinal fluid

P932

1798287

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No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

about

P2860

P2860

No signs of immunoactivation in the cerebrospinal fluid during treatment with infliximab.

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