Novel senescence associated gene, YPEL3, is repressed by estrogen in ER+ mammary tumor cells and required for tamoxifen-induced cellular senescence.
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Environmental immune disruptors, inflammation and cancer riskThe potential for chemical mixtures from the environment to enable the cancer hallmark of sustained proliferative signallingExamination of ERα signaling pathways in bone of mutant mouse models reveals the importance of ERE-dependent signalingYPEL3 suppresses epithelial-mesenchymal transition and metastasis of nasopharyngeal carcinoma cells through the Wnt/β-catenin signaling pathway.Combining animal personalities with transcriptomics resolves individual variation within a wild-type zebrafish population and identifies underpinning molecular differences in brain function.The protein p17 signaling pathways in cancer.
P2860
Novel senescence associated gene, YPEL3, is repressed by estrogen in ER+ mammary tumor cells and required for tamoxifen-induced cellular senescence.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Novel senescence associated ge ...... n-induced cellular senescence.
@ast
Novel senescence associated ge ...... n-induced cellular senescence.
@en
type
label
Novel senescence associated ge ...... n-induced cellular senescence.
@ast
Novel senescence associated ge ...... n-induced cellular senescence.
@en
prefLabel
Novel senescence associated ge ...... n-induced cellular senescence.
@ast
Novel senescence associated ge ...... n-induced cellular senescence.
@en
P2093
P2860
P356
P1476
Novel senescence associated ge ...... en-induced cellular senescence
@en
P2093
J Nicholas Maiorano
Kelly R Miller
Rebecca Tuttle
Steven J Berberich
Yongping Gao
P2860
P304
P356
10.1002/IJC.26239
P577
2011-08-09T00:00:00Z