Deletion of thioredoxin-interacting protein in mice impairs mitochondrial function but protects the myocardium from ischemia-reperfusion injury
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ROS-Mediated NLRP3 Inflammasome Activation in Brain, Heart, Kidney, and Testis Ischemia/Reperfusion InjuryThioredoxins, glutaredoxins, and peroxiredoxins--molecular mechanisms and health significance: from cofactors to antioxidants to redox signalingThe Emerging Role of Thioredoxin-Interacting Protein in Myocardial Ischemia/Reperfusion InjuryCharacterization, design, and function of the mitochondrial proteome: from organs to organismsThioredoxin A Is Essential for Motility and Contributes to Host Infection of Listeria monocytogenes via Redox Interactions.Minireview: Thioredoxin-interacting protein: regulation and function in the pancreatic β-cell.Thioredoxin-interacting protein regulates protein disulfide isomerases and endoplasmic reticulum stress.Identification of differentially expressed transcripts and pathways in blood one week and six months following implant of left ventricular assist devices.Redox redux: protecting the ischemic myocardium.Expression of SGLT1 in Human Hearts and Impairment of Cardiac Glucose Uptake by Phlorizin during Ischemia-Reperfusion Injury in Mice.Acute inhibition of GSK causes mitochondrial remodeling.Calcium channel blockers act through nuclear factor Y to control transcription of key cardiac genes.Thioredoxin-Interacting Protein Deficiency Protects against Diabetic Nephropathy.Thioredoxin and thioredoxin target proteins: from molecular mechanisms to functional significance.TXNIP/TRX/NF-κB and MAPK/NF-κB pathways involved in the cardiotoxicity induced by Venenum Bufonis in rats.TXNIP shuttling: missing link between oxidative stress and inflammasome activation.Two-Step Reactivation of Dormant Cones in Retinitis PigmentosaHECT-Type Ubiquitin E3 Ligase ITCH Interacts With Thioredoxin-Interacting Protein and Ameliorates Reactive Oxygen Species-Induced Cardiotoxicity.Caspase 1 activation is protective against hepatocyte cell death by up-regulating beclin 1 protein and mitochondrial autophagy in the setting of redox stress.Identification of Redox and Glucose-Dependent Txnip Protein InteractionsTXNIP regulates myocardial fatty acid oxidation via miR-33a signalingAcute hyperglycaemia enhances oxidative stress and aggravates myocardial ischaemia/reperfusion injury: role of thioredoxin-interacting protein.Redox signaling in cardiovascular health and disease.A critical role for thioredoxin-interacting protein in diabetes-related impairment of angiogenesis.Reprogramming-derived gene cocktail increases cardiomyocyte proliferation for heart regeneration.Targeted metabolomics connects thioredoxin-interacting protein (TXNIP) to mitochondrial fuel selection and regulation of specific oxidoreductase enzymes in skeletal muscle.Use of a mouse in vitro fertilization model to understand the developmental origins of health and disease hypothesis.3, 4-dihydroxyl-phenyl lactic acid restores NADH dehydrogenase 1 α subunit 10 to ameliorate cardiac reperfusion injury.Heat shock proteins in tendinopathy: novel molecular regulators.Alarmins in tendinopathy: unravelling new mechanisms in a common disease.Mesenchymal stem cell exosome ameliorates reperfusion injury through proteomic complementation.Thioredoxin-interacting protein and myocardial mitochondrial function in ischemia-reperfusion injury.Thioredoxin-interacting protein: pathophysiology and emerging pharmacotherapeutics in cardiovascular disease and diabetes.Regulation of cellular redox signaling by matricellular proteins in vascular biology, immunology, and cancerSufentanil protects the rat myocardium against ischemia-reperfusion injury via activation of the ERK1/2 pathway.Pyrroloquinoline quinone inhibits oxygen/glucose deprivation-induced apoptosis by activating the PI3K/AKT pathway in cardiomyocytes.Critical role of TXNIP in oxidative stress, DNA damage and retinal pericyte apoptosis under high glucose: implications for diabetic retinopathyA REDD1/TXNIP pro-oxidant complex regulates ATG4B activity to control stress-induced autophagy and sustain exercise capacity.Differential expression of alarmins-S100A9, IL-33, HMGB1 and HIF-1α in supraspinatus tendinopathy before and after treatment.Thioredoxin-interacting protein mediates high glucose-induced reactive oxygen species generation by mitochondria and the NADPH oxidase, Nox4, in mesangial cells
P2860
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P2860
Deletion of thioredoxin-interacting protein in mice impairs mitochondrial function but protects the myocardium from ischemia-reperfusion injury
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Deletion of thioredoxin-intera ...... om ischemia-reperfusion injury
@ast
Deletion of thioredoxin-intera ...... om ischemia-reperfusion injury
@en
type
label
Deletion of thioredoxin-intera ...... om ischemia-reperfusion injury
@ast
Deletion of thioredoxin-intera ...... om ischemia-reperfusion injury
@en
prefLabel
Deletion of thioredoxin-intera ...... om ischemia-reperfusion injury
@ast
Deletion of thioredoxin-intera ...... om ischemia-reperfusion injury
@en
P2093
P2860
P356
P1476
Deletion of thioredoxin-intera ...... om ischemia-reperfusion injury
@en
P2093
Christine E Seidman
Edward P Feener
Jae Bum Kim
Jonathan G Seidman
Jun Yoshioka
Richard T Lee
Samuel Lee
William A Chutkow
P2860
P304
P356
10.1172/JCI44927
P407
P577
2011-12-27T00:00:00Z