Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3-mediated pathway - Wikidata - awgldk - TriplyDB

Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3-mediated pathway

Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3-mediated pathway

http://www.wikidata.org/entity/Q35647715

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The RIP1/RIP3 necrosome forms a functional amyloid signaling complex required for programmed necrosis Post-translational modifications as key regulators of TNF-induced necroptosis Poly-ubiquitination in TNFR1-mediated necroptosis An outline of necrosome triggers The interplay between human herpes simplex virus infection and the apoptosis and necroptosis cell death pathways Necroptosis in acute kidney injury: a shedding light Necroptotic Cell Death Signaling and Execution Pathway: Lessons from Knockout Mice Apoptosis, Necrosis, and Necroptosis in the Gut and Intestinal Homeostasis Fueling the flames: Mammalian programmed necrosis in inflammatory diseases Photoreceptor cell death and rescue in retinal detachment and degenerations The two faces of receptor interacting protein kinase-1 Interplay between Inflammation and Cellular Stress Triggered by Flaviviridae Viruses Execution of RIPK3-regulated necrosis Programmed necrosis in the cross talk of cell death and inflammation The TLR signalling adaptor TRIF/TICAM-1 has an N-terminal helical domain with structural similarity to IFIT proteins Casp8p41: The Protean Mediator of Death in CD4 T-cells that Replicate HIV Necroptosis: The Trojan horse in cell autonomous antiviral host defense Herpes Simplex Virus 1 (HSV-1) and HSV-2 Mediate Species-Specific Modulations of Programmed Necrosis through the Viral Ribonucleotide Reductase Large Subunit R1 Die another way--non-apoptotic mechanisms of cell death Type I interferon induces necroptosis in macrophages during infection with Salmonella enterica serovar Typhimurium TAK1 binding protein 2 is essential for liver protection from stressors Identification of a Transcription Factor That Regulates Host Cell Exit and Virulence of Mycobacterium tuberculosis Mlkl knockout mice demonstrate the indispensable role of Mlkl in necroptosis The Viral Mimetic Polyinosinic:Polycytidylic Acid Alters the Growth Characteristics of Small Intestinal and Colonic Crypt Cultures The Gβγ-Src signaling pathway regulates TNF-induced necroptosis via control of necrosome translocation A cellular screen identifies ponatinib and pazopanib as inhibitors of necroptosis.Suppression of injuries caused by a lytic RNA virus (mengovirus) and their uncoupling from viral reproduction by mutual cell/virus disarmament.RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL The Pseudokinase MLKL and the Kinase RIPK3 Have Distinct Roles in Autoimmune Disease Caused by Loss of Death-Receptor-Induced Apoptosis.Toll-like receptor-mediated down-regulation of the deubiquitinase cylindromatosis (CYLD) protects macrophages from necroptosis in wild-derived mice Caspase-8 mediates caspase-1 processing and innate immune defense in response to bacterial blockade of NF-κB and MAPK signaling RIP1 suppresses innate immune necrotic as well as apoptotic cell death during mammalian parturition.RIPK1 blocks early postnatal lethality mediated by caspase-8 and RIPK3 Necroptosis mediates TNF-induced toxicity of hippocampal neurons.Type-I interferon signaling through ISGF3 complex is required for sustained Rip3 activation and necroptosis in macrophages.A toll-like receptor 9 antagonist improves bladder function and white matter sparing in spinal cord injury.Necroptotic signaling in adaptive and innate immunity.Hematopoietic RIPK1 deficiency results in bone marrow failure caused by apoptosis and RIPK3-mediated necroptosis.Direct activation of RIP3/MLKL-dependent necrosis by herpes simplex virus 1 (HSV-1) protein ICP6 triggers host antiviral defense.Toll-like receptor-mediated responses by placental Hofbauer cells (HBCs): a potential pro-inflammatory role for fetal M2 macrophages

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P2860

Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3-mediated pathway

http://www.wikidata.org/entity/Q35647715

description

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Proceedings of the National Academy of Sciences of the United States of America

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Toll-like receptors activate p ...... ting kinase-3-mediated pathway

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P2093

Feng Shao

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Sudan He

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Xiaodong Wang

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Yuqiong Liang

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P2860

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

Inhibition of ADP/ATP exchange in receptor-interacting protein-mediated necrosis.

cIAPs block Ripoptosome formation, a RIP1/caspase-8 containing intracellular cell death complex differentially regulated by cFLIP isoforms

Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation

Recognition of double-stranded RNA and activation of NF-kappaB by Toll-like receptor 3

The role of pattern-recognition receptors in innate immunity: update on Toll-like receptors

Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product

Catalytic activity of the caspase-8-FLIP(L) complex inhibits RIPK3-dependent necrosis

Identification of Lps2 as a key transducer of MyD88-independent TIR signalling

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20054-20059

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22123964

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P921

Toll-like receptor

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3250173

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