MHCI negatively regulates synapse density during the establishment of cortical connections. - Wikidata - awgldk - TriplyDB

MHCI negatively regulates synapse density during the establishment of cortical connections.

MHCI negatively regulates synapse density during the establishment of cortical connections.

http://www.wikidata.org/entity/Q35652867

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Lyme disease and pediatric autoimmune neuropsychiatric disorders associated with streptococcal infections (PANDAS): an overview Major Histocompatibility Complex I Expression by Motor Neurons and Its Implication in Amyotrophic Lateral Sclerosis Epidemiologic studies of exposure to prenatal infection and risk of schizophrenia and autism Major Histocompatibility Complex class I proteins are critical for maintaining neuronal structural complexity in the aging brain β2-microglobulin is a systemic pro-aging factor that impairs cognitive function and neurogenesis Maternal immune activation: Implications for neuropsychiatric disorders.MHC-I expression renders catecholaminergic neurons susceptible to T-cell-mediated degeneration Functional deficiency of MHC class I enhances LTP and abolishes LTD in the nucleus accumbens of mice.MHC class I immune proteins are critical for hippocampus-dependent memory and gate NMDAR-dependent hippocampal long-term depression.DNA methylation analysis of the autistic brain reveals multiple dysregulated biological pathways.Major histocompatibility complex I in brain development and schizophrenia The major histocompatibility complex and autism spectrum disorder.Neuronal MHC Class I Expression Is Regulated by Activity Driven Calcium Signaling.MHC class I limits hippocampal synapse density by inhibiting neuronal insulin receptor signaling Cryptic protein-protein interaction motifs in the cytoplasmic domain of MHCI proteins.Neuroglial expression of the MHCI pathway and PirB receptor is upregulated in the hippocampus with advanced aging.Major histocompatibility complex class I proteins in brain development and plasticity.Toxoplasma gondii and cognitive deficits in schizophrenia: an animal model perspective PirB regulates asymmetries in hippocampal circuitry Secondary association of PDLIM5 with paranoid schizophrenia in Emirati patients Developmental Sculpting of Intracortical Circuits by MHC Class I H2-Db and H2-Kb.Expression and alternative splicing of classical and nonclassical MHCI genes in the hippocampus and neuromuscular junction.Rescue of the Functional Alterations of Motor Cortical Circuits in Arginase Deficiency by Neonatal Gene Therapy.Impaired synaptic development in a maternal immune activation mouse model of neurodevelopmental disorders MHCI requires MEF2 transcription factors to negatively regulate synapse density during development and in disease.Chronic Binge Alcohol Administration Dysregulates Hippocampal Genes Involved in Immunity and Neurogenesis in Simian Immunodeficiency Virus-Infected Macaques.The Major Histocompatibility Complex (MHC) in Schizophrenia: A Review.Astrocyte immune responses in epilepsy.Genetics of schizophrenia.Molecular basis of alcoholism.Maternal immune activation and abnormal brain development across CNS disorders.Neuronal MHC-I expression and its implications in synaptic function, axonal regeneration and Parkinson's and other brain diseases Immune mediators in the brain and peripheral tissues in autism spectrum disorder.Excitatory/inhibitory imbalance in autism spectrum disorders: Implications for interventions and therapeutics.Genetic architecture, epigenetic influence and environment exposure in the pathogenesis of Autism.Quantitation of circulating GDF-11 and β2-MG in aged patients with age-related impairment in cognitive function.Long-term depression-associated signaling is required for an in vitro model of NMDA receptor-dependent synapse pruning.Synaptoimmunology - roles in health and disease.Synapse elimination and learning rules co-regulated by MHC class I H2-Db.CNS-wide Sexually Dimorphic Induction of the Major Histocompatibility Complex 1 Pathway With Aging.

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P2860

MHCI negatively regulates synapse density during the establishment of cortical connections.

http://www.wikidata.org/entity/Q35652867

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2011 nî lūn-bûn

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MHCI negatively regulates syna ...... hment of cortical connections.

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MHCI negatively regulates syna ...... hment of cortical connections.

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MHCI negatively regulates syna ...... hment of cortical connections.

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MHCI negatively regulates syna ...... hment of cortical connections.

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MHCI negatively regulates syna ...... shment of cortical connections

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Bradford M Elmer

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Faten El-Sabeawy

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Leigh A Needleman

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Marian W Glynn

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Paula A Garay

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Xiao-Bo Liu

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P2860

EphB-mediated degradation of the RhoA GEF Ephexin5 relieves a developmental brake on excitatory synapse formation

Functional requirement for class I MHC in CNS development and plasticity

Activity-dependent regulation of MEF2 transcription factors suppresses excitatory synapse number

Specific associations of fluorescent beta-2-microglobulin with cell surfaces. The affinity of different H-2 and HLA antigens for beta-2-microglobulin

Total arrest of spontaneous and evoked synaptic transmission but normal synaptogenesis in the absence of Munc13-mediated vesicle priming

Quantification of synapse formation and maintenance in vivo in the absence of synaptic release

Colocalization of multiple GABA(A) receptor subtypes with gephyrin at postsynaptic sites

Synaptic assembly of the brain in the absence of neurotransmitter secretion

Selective clustering of glutamate and gamma-aminobutyric acid receptors opposite terminals releasing the corresponding neurotransmitters.

Monitoring secretory membrane with FM1-43 fluorescence.

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21358642

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3251641

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