Repeated administration of the noradrenergic neurotoxin N-(2-chloroethyl)-N-ethyl-2-bromobenzylamine (DSP-4) modulates neuroinflammation and amyloid plaque load in mice bearing amyloid precursor protein and presenilin-1 mutant transgenes

Repeated administration of the noradrenergic neurotoxin N-(2-chloroethyl)-N-ethyl-2-bromobenzylamine (DSP-4) modulates neuroinflammation and amyloid plaque load in mice bearing amyloid precursor protein and presenilin-1 mutant transgenes

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http://www.wikidata.org/entity/Q35663346
Noradrenergic regulation of glial activation: molecular mechanisms and therapeutic implications.Locus coeruleus damage and noradrenaline reductions in multiple sclerosis and experimental autoimmune encephalomyelitis.Noradrenergic dysfunction in Alzheimer's disease.The noradrenaline precursor L-DOPS reduces pathology in a mouse model of Alzheimer's diseaseA noradrenergic lesion exacerbates neurodegeneration in a Down syndrome mouse model.Causes, consequences, and cures for neuroinflammation mediated via the locus coeruleus: noradrenergic signaling system.Loss of locus coeruleus noradrenergic neurons alters the inflammatory response to LPS in substantia nigra but does not affect nigral cell loss.Effects of DSP4 on the noradrenergic phenotypes and its potential molecular mechanisms in SH-SY5Y cells.Trigeminal, Visceral and Vestibular Inputs May Improve Cognitive Functions by Acting through the Locus Coeruleus and the Ascending Reticular Activating System: A New Hypothesis.
P2860

Repeated administration of the noradrenergic neurotoxin N-(2-chloroethyl)-N-ethyl-2-bromobenzylamine (DSP-4) modulates neuroinflammation and amyloid plaque load in mice bearing amyloid precursor protein and presenilin-1 mutant transgenes

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http://www.wikidata.org/entity/Q35663346
description
2007 nî lūn-bûn
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Repeated administration of the ...... presenilin-1 mutant transgenes
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Repeated administration of the ...... presenilin-1 mutant transgenes
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