Glucocorticoids repress NF-kappaB-driven genes by disturbing the interaction of p65 with the basal transcription machinery, irrespective of coactivator levels in the cell
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Altered subcellular distribution of MSK1 induced by glucocorticoids contributes to NF-kappaB inhibition.Retinoic acid receptors inhibit AP1 activation by regulating extracellular signal-regulated kinase and CBP recruitment to an AP1-responsive promoter.Therapeutic potential of inhibition of the NF-kappaB pathway in the treatment of inflammation and cancerOne hormone, two actions: anti- and pro-inflammatory effects of glucocorticoidsMicroRNAs as tools to predict glucocorticoid response in inflammatory bowel diseasesIntegrating Interleukin-6 into depression diagnosis and treatmentEndothelial Response to Glucocorticoids in Inflammatory DiseasesAcetylation of RelA at discrete sites regulates distinct nuclear functions of NF-kappaB.Identification of glucocorticoid receptor domains involved in transrepression of transforming growth factor-beta actionTherapeutic effects of acetylsalicylic acid in giant cell arteritisCompound A, a selective glucocorticoid receptor modulator, enhances heat shock protein Hsp70 gene promoter activationCytosolic glucocorticoid receptor interaction with nuclear factor-kappa B proteins in rat liver cellsGenome-wide predictors of NF-κB recruitment and transcriptional activityIdentification of a negative regulatory cis-element in the enhancer core region of the prostate-specific antigen promoter: implications for intersection of androgen receptor and nuclear factor-kappaB signalling in prostate cancer cells.Crosstalk in inflammation: the interplay of glucocorticoid receptor-based mechanisms and kinases and phosphatasesMinireview: Glucocorticoids in autoimmunity: unexpected targets and mechanisms.Abrogation of glucocorticoid receptor dimerization correlates with dissociated glucocorticoid behavior of compound a.The glucocorticoid receptor blocks P-TEFb recruitment by NFkappaB to effect promoter-specific transcriptional repression.Transcription factor AP1 potentiates chromatin accessibility and glucocorticoid receptor bindingA fully dissociated compound of plant origin for inflammatory gene repressionSeries introduction: the transcription factor NF-kappaB and human diseaseCross-talk between glucocorticoid receptor and AP-1.Increased p300 expression inhibits glucocorticoid receptor-T-cell receptor antagonism but does not affect thymocyte positive selectionTranscription factor-mediated molecular mechanisms involved in the functional cross-talk between cytokines and glucocorticoids.Alternate surfaces of transcriptional coregulator GRIP1 function in different glucocorticoid receptor activation and repression contexts.Transcriptional and post-transcriptional mechanisms of glucocorticoid antiproliferative effects.Foxa1 and Foxa2 regulate bile duct development in mice.Growing old with nuclear factor-kappaBPeptide-based inhibition of NF-κB rescues diaphragm muscle contractile dysfunction in a murine model of Duchenne muscular dystrophy.The regulation of muscle mass by endogenous glucocorticoids.The thyroid hormone receptor antagonizes CREB-mediated transcriptionTumor necrosis factor inhibits glucocorticoid receptor function in mice: a strong signal toward lethal shock.Arvelexin from Brassica rapa suppresses NF-κB-regulated pro-inflammatory gene expression by inhibiting activation of IκB kinaseMacrophage glucocorticoid receptors regulate Toll-like receptor 4-mediated inflammatory responses by selective inhibition of p38 MAP kinaseO-GlcNAc transferase is involved in glucocorticoid receptor-mediated transrepression.Molecular mechanisms of glucocorticoids in the control of inflammation and lymphocyte apoptosis.The molecular basis for the effectiveness, toxicity, and resistance to glucocorticoids: focus on the treatment of rheumatoid arthritis.Inhibition of autophagy overcomes glucocorticoid resistance in lymphoid malignant cells.Inflammatory targets of therapy in sickle cell disease.Combinatorial roles of nuclear receptors in inflammation and immunity.
P2860
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P2860
Glucocorticoids repress NF-kappaB-driven genes by disturbing the interaction of p65 with the basal transcription machinery, irrespective of coactivator levels in the cell
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Glucocorticoids repress NF-kap ...... coactivator levels in the cell
@ast
Glucocorticoids repress NF-kap ...... coactivator levels in the cell
@en
type
label
Glucocorticoids repress NF-kap ...... coactivator levels in the cell
@ast
Glucocorticoids repress NF-kap ...... coactivator levels in the cell
@en
prefLabel
Glucocorticoids repress NF-kap ...... coactivator levels in the cell
@ast
Glucocorticoids repress NF-kap ...... coactivator levels in the cell
@en
P2093
P2860
P356
P1476
Glucocorticoids repress NF-kap ...... coactivator levels in the cell
@en
P2093
G Haegeman
K De Bosscher
L Vermeulen
S Plaisance
W Vanden Berghe
P2860
P304
P356
10.1073/PNAS.97.8.3919
P407
P577
2000-04-01T00:00:00Z