In an Ovine Model of Polycystic Ovary Syndrome (PCOS) Prenatal Androgens Suppress Female Fetal Renal Gluconeogenesis

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The epigenomics of polycystic ovarian syndrome: from pathogenesis to clinical manifestations.Current aspects of polycystic ovary syndrome: A literature review.The role of fructose‑1,6‑bisphosphatase 1 in abnormal development of ovarian follicles caused by high testosterone concentration.

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P2860

In an Ovine Model of Polycystic Ovary Syndrome (PCOS) Prenatal Androgens Suppress Female Fetal Renal Gluconeogenesis

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2015 nî lūn-bûn

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2015年の論文

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In an Ovine Model of Polycysti ...... le Fetal Renal Gluconeogenesis

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In an Ovine Model of Polycysti ...... le Fetal Renal Gluconeogenesis

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In an Ovine Model of Polycysti ...... le Fetal Renal Gluconeogenesis

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In an Ovine Model of Polycysti ...... le Fetal Renal Gluconeogenesis

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In an Ovine Model of Polycysti ...... le Fetal Renal Gluconeogenesis

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In an Ovine Model of Polycysti ...... le Fetal Renal Gluconeogenesis

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In an Ovine Model of Polycysti ...... le Fetal Renal Gluconeogenesis

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Alan S McNeilly

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Fiona Connolly

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Katharina Späth

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Lyndsey Boswell

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Michael T Rae

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W Colin Duncan

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P2860

Control of blood glucose in the absence of hepatic glucose production during prolonged fasting in mice: induction of renal and intestinal gluconeogenesis by glucagon

Factors that control the tissue-specific transcription of the gene for phosphoenolpyruvate carboxykinase-C

3'-Untranslated region of phosphoenolpyruvate carboxykinase mRNA contains multiple instability elements that bind AUF1

Maternal low protein diet restricted to the preimplantation period induces a gender-specific change on hepatic gene expression in rat fetuses

Prenatal androgen exposure programs metabolic dysfunction in female mice.

Renal gluconeogenesis: its importance in human glucose homeostasis.

Developmental programming: impact of prenatal testosterone excess and postnatal weight gain on insulin sensitivity index and transfer of traits to offspring of overweight females

Regulation of glucose production by the liver.

The in utero programming effect of increased maternal androgens and a direct fetal intervention on liver and metabolic function in adult sheep.

Cortisol metabolism and the role of 11beta-hydroxysteroid dehydrogenase.

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10.1371/JOURNAL.PONE.0132113

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polycystic ovary syndrome

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In an Ovine Model of Polycystic Ovary Syndrome (PCOS) Prenatal Androgens Suppress Female Fetal Renal Gluconeogenesis

about

P2860

P2860

In an Ovine Model of Polycystic Ovary Syndrome (PCOS) Prenatal Androgens Suppress Female Fetal Renal Gluconeogenesis

description

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