Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury. - Wikidata - awgldk - TriplyDB

Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury.

Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury.

http://www.wikidata.org/entity/Q35684746

about

EphA2-receptor deficiency exacerbates myocardial infarction and reduces survival in hyperglycemic mice.Transactivation of the receptor-tyrosine kinase ephrin receptor A2 is required for the low molecular weight hyaluronan-mediated angiogenesis that is implicated in tumor progression.Matrix metalloproteinases and protein tyrosine kinases: potential novel targets in acute lung injury and ARDS Evidence that the EphA2 receptor exacerbates ischemic brain injury.EphA2 Expression Regulates Inflammation and Fibroproliferative Remodeling in Atherosclerosis.Eph Receptor Tyrosine Kinases in Tumor Immunity.Tobacco smoke induces epithelial barrier dysfunction via receptor EphA2 signaling.ADAM12-cleaved ephrin-A1 contributes to lung metastasis.EphA2 Receptor Signaling Mediates Inflammatory Responses in Lipopolysaccharide-Induced Lung Injury Clinical implications of the plasma EphA2 receptor level in critically ill patients with septic shock.MicroRNA-302b negatively regulates IL-1β production in response to MSU crystals by targeting IRAK4 and EphA2.A Novel EphA2 Inhibitor Exerts Beneficial Effects in PI-IBS in Vivo and in Vitro Models via Nrf2 and NF-κB Signaling Pathways.

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P2860

Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury.

http://www.wikidata.org/entity/Q35684746

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

2012年论文

@zh

2012年论文

@zh-cn

name

Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury.

@ast

Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury.

@en

type

label

Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury.

@ast

Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury.

@en

prefLabel

Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury.

@ast

Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury.

@en

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P356

RCMB.2011-0044OC

P1433

American Journal of Respiratory Cell and Molecular Biology

P1476

Eph-A2 promotes permeability and inflammatory responses to bleomycin-induced lung injury

@en

P2093

Kurt R Stenmark

http://www.w3.org/2001/XMLSchema#string

Todd C Carpenter

http://www.w3.org/2001/XMLSchema#string

William Schroeder

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P2860

EphA2 mediates ligand-dependent inhibition and ligand-independent promotion of cell migration and invasion via a reciprocal regulatory loop with Akt

Soluble monomeric EphrinA1 is released from tumor cells and is a functional ligand for the EphA2 receptor

Blockade of EphA receptor tyrosine kinase activation inhibits vascular endothelial cell growth factor-induced angiogenesis.

Role of B61, the ligand for the Eck receptor tyrosine kinase, in TNF-alpha-induced angiogenesis

TGF-beta is a critical mediator of acute lung injury

Expression of Eph receptors and their ligands, ephrins, during lipopolysaccharide fever in rats

Essential role of Vav family guanine nucleotide exchange factors in EphA receptor-mediated angiogenesis

EphA2 engages Git1 to suppress Arf6 activity modulating epithelial cell-cell contacts.

Soluble ephrin a1 is necessary for the growth of HeLa and SK-BR3 cells

Endothelial EphA receptor stimulation increases lung vascular permeability.

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40-47

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scholarly article

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10.1165/RCMB.2011-0044OC

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1

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46

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2012-01-01T00:00:00Z

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21799118

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3262654

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