Interleukin-17A contributes to myocardial ischemia/reperfusion injury by regulating cardiomyocyte apoptosis and neutrophil infiltration.
about
The Predictive Role of Inflammatory Biomarkers in Atrial Fibrillation as Seen through Neutrophil-Lymphocyte Ratio MirrorCommunication in the heart: the role of the innate immune system in coordinating cellular responses to ischemic injuryInterleukin-17 and acute coronary syndromeThe Roles of CD137 Signaling in AtherosclerosisC1 esterase inhibitor reduces lower extremity ischemia/reperfusion injury and associated lung damage.Ghrelin protected neonatal rat cardiomyocyte against hypoxia/reoxygenation injury by inhibiting apoptosis through Akt-mTOR signal.The PI3K/Akt/mTOR pathway is involved in CVB3-induced autophagy of HeLa cells.Cardiac fibroblasts mediate IL-17A-driven inflammatory dilated cardiomyopathy.T cells in organ ischemia reperfusion injury.IL-17A mediates early post-transplant lesions after heterotopic trachea allotransplantation in MiceIdentification of a non-growth factor role for GM-CSF in advanced atherosclerosis: promotion of macrophage apoptosis and plaque necrosis through IL-23 signaling.The complex regulation of tanshinone IIA in rats with hypertension-induced left ventricular hypertrophyNovel antibody against a glutamic acid-rich human fibrinogen-like protein 2-derived peptide near Ser91 inhibits hfgl2 prothrombinase activityEndoplasmic Reticulum Is Involved in Myocardial Injury in a Miniature Swine Model of Coronary Artery Stenosis Exposed to Acceleration-Associated Stress.Interleukin-17A contributes to the development of post-operative atrial fibrillation by regulating inflammation and fibrosis in rats with sterile pericarditis.Luteolin Inhibits Ischemia/Reperfusion-Induced Myocardial Injury in Rats via Downregulation of microRNA-208b-3p.Studies on the role of apoptosis after transient myocardial ischemia: genetic deletion of the executioner caspases-3 and -7 does not limit infarct size and ventricular remodeling.Vagus nerve stimulation attenuates myocardial ischemia/reperfusion injury by inhibiting the expression of interleukin-17ADeleterious effect of the IL-23/IL-17A axis and γδT cells on left ventricular remodeling after myocardial infarction.Elevated plasma levels of Th17-related cytokines are associated with increased risk of atrial fibrillationAdvanced oxidation protein products induce cardiomyocyte death via Nox2/Rac1/superoxide-dependent TRAF3IP2/JNK signaling.Knockout of interleukin-17A protects against sepsis-associated acute kidney injury.HSPA12B Attenuated Acute Myocardial Ischemia/reperfusion Injury via Maintaining Endothelial Integrity in a PI3K/Akt/mTOR-dependent Mechanism.IL-9 Inhibits Viral Replication in Coxsackievirus B3-Induced Myocarditis.Glaucocalyxin A Ameliorates Myocardial Ischemia-Reperfusion Injury in Mice by Suppression of Microvascular Thrombosis.Interleukin-37 and Dendritic Cells Treated With Interleukin-37 Plus Troponin I Ameliorate Cardiac Remodeling After Myocardial InfarctionBlockage of transient receptor potential vanilloid 4 alleviates myocardial ischemia/reperfusion injury in mice.Hydrogen sulfide attenuates the recruitment of CD11b⁺Gr-1⁺ myeloid cells and regulates Bax/Bcl-2 signaling in myocardial ischemia injury.Cardiac intercellular communication: are myocytes and fibroblasts fair-weather friends?Control of inflammatory heart disease by CD4+ T cells.Contributions of cardiomyocyte-cardiac fibroblast-immune cell interactions in heart failure development.Regulatory RNAs and paracrine networks in the heart.Role of innate and adaptive immune mechanisms in cardiac injury and repair.Immune cell and other noncardiomyocyte regulation of cardiac hypertrophy and remodeling.Immunopathogenesis of Myocarditis: The Interplay Between Cardiac Fibroblast Cells, Dendritic Cells, Macrophages and CD4+ T Cells.Where Does Inflammation Fit?IL-21 promotes myocardial ischaemia/reperfusion injury through the modulation of neutrophil infiltration.Interleukin-17 as a factor linking the pathogenesis of psoriasis with metabolic disorders.15-deoxy-Δ¹²,¹⁴-PGJ₂ promotes inflammation and apoptosis in cardiomyocytes via the DP2/MAPK/TNFα axis.Role of the immune system in cardiac tissue damage and repair following myocardial infarction.
P2860
Q26742118-2EE41732-BDD7-446A-A493-10DA7836E9F0Q26830128-5CE05DDB-764A-4791-84F4-A4A2BC7DE4DFQ27025823-F846338A-B75A-47C8-A318-98301C60D89BQ28076613-20543C8C-5BE2-4C9B-9AFC-B35E43208B44Q31132259-A93408EB-E280-4CC1-9D72-3D42D8754CAFQ32180674-890DAA50-85A2-4CEA-9C5E-3C67086A0277Q33783958-A7016F85-C48C-4409-A15C-EFD5B40DF156Q33826888-AF98C238-A30A-4EA9-9898-7A9D230D4B25Q33940412-4C409E44-EF63-48D4-9D64-CAAF5CB17C98Q34922333-9EE96410-6BC0-4877-BFBB-37169E475533Q34979530-606BA8A3-82B1-4FF6-8782-A3CDB160D7BDQ35125156-8AE36BB1-B93F-44CF-A05E-EC36E91B613AQ35146839-44352CE5-2B9C-41A0-AD38-EC15899E5FD0Q35689499-74BB5B6D-9D5F-46BE-8C15-ACD4FDC462E2Q35827568-A36E8C0C-C321-46A0-8A3E-5A5528550586Q35866988-9A49C67B-9855-49A3-83DA-CF6A444D94F4Q35939072-CBBF5495-1548-4069-91ED-ABB2D7067F5AQ36502342-AD356625-C470-480D-8429-4FA2425EF5B8Q36522680-D763BEE5-A35B-47EA-BD12-6CBB43B10B87Q36919277-AB9B6617-83A8-4736-BAB3-F5AED7251789Q37023199-AEB00742-0A9B-47A3-8933-EC8B66D0AAC2Q37029951-565712D3-9AEA-46CB-934D-A35393C4C598Q37267487-247745CA-F13B-46FA-8175-DFF3B410B7F4Q37314067-18CB4A1F-2A9D-4A3B-B3B6-10B5BF4784E5Q37324328-2B9F1629-E66C-4DFE-BAC8-7897AD5FD1F7Q37556429-EB91EC52-5162-4B29-9E00-61CF480383F4Q37644143-9BDBB0FF-D500-4957-BCF1-92B70A6E92B6Q37720537-31F0E6BD-EA34-4A7E-B73B-D4DD20215BFEQ38046798-C4E9D5CC-7498-4E1E-960F-ADCBCDF4100BQ38108397-C3E5CE69-0488-406B-BC20-2823558C40CCQ38112347-31BEAB8E-4651-4296-975F-677FA1AE0B59Q38190443-B1DC2E0A-BC09-4EB6-8F25-2F561599607BQ38329556-76D795E1-61C6-485C-BBD9-E99D187E9A6AQ38380871-A0E3556E-BA62-4C28-A9B3-E4966A4B9FA3Q38407555-2679FD2E-8584-4019-BB76-3B8DCBA926A6Q38639570-46BC0FA5-3E98-4035-A511-BF691A75D51CQ38907629-EA9A0A8A-6843-434A-A5FB-956FC35CE157Q38970666-8076C338-97ED-45A3-8C4E-643F76B453A0Q39008479-67127C80-B2CF-4F3A-B2C1-16F2E7EFDAA3Q39363562-35C642F2-4198-4FAE-AAB1-CB9A6EB91A1A
P2860
Interleukin-17A contributes to myocardial ischemia/reperfusion injury by regulating cardiomyocyte apoptosis and neutrophil infiltration.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Interleukin-17A contributes to ...... s and neutrophil infiltration.
@ast
Interleukin-17A contributes to ...... s and neutrophil infiltration.
@en
type
label
Interleukin-17A contributes to ...... s and neutrophil infiltration.
@ast
Interleukin-17A contributes to ...... s and neutrophil infiltration.
@en
prefLabel
Interleukin-17A contributes to ...... s and neutrophil infiltration.
@ast
Interleukin-17A contributes to ...... s and neutrophil infiltration.
@en
P2093
P2860
P1476
Interleukin-17A contributes to ...... is and neutrophil infiltration
@en
P2093
Bing-Jie Lv
Guo-Ping Shi
Harish Jevallee
Shao-Fang Nie
Su-Feng Zhou
P2860
P304
P356
10.1016/J.JACC.2011.10.863
P407
P577
2012-01-01T00:00:00Z