Concordance of increased B1 cell subset and lupus phenotypes in mice and humans is dependent on BLK expression levels.
about
The SLE variant Ala71Thr of BLK severely decreases protein abundance and binding to BANK1 through impairment of the SH3 domain function.Autoimmunity in 2015.The juvenile idiopathic inflammatory myopathies: pathogenesis, clinical and autoantibody phenotypes, and outcomesB1a cells play a pathogenic role in the development of autoimmune arthritis.Omics studies: their use in diagnosis and reclassification of SLE and other systemic autoimmune diseases.Abnormal B-cell development in Systemic Lupus Erythematosus: what the genetics tell us.BLK pathway-associated rs13277113 GA genotype is more frequent in SLE patients and associated with low gene expression and increased flares.CD6 expression has no effect on atherosclerosis in apolipoprotein E-deficient mice.
P2860
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P2860
Concordance of increased B1 cell subset and lupus phenotypes in mice and humans is dependent on BLK expression levels.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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name
Concordance of increased B1 ce ...... dent on BLK expression levels.
@ast
Concordance of increased B1 ce ...... dent on BLK expression levels.
@en
type
label
Concordance of increased B1 ce ...... dent on BLK expression levels.
@ast
Concordance of increased B1 ce ...... dent on BLK expression levels.
@en
prefLabel
Concordance of increased B1 ce ...... dent on BLK expression levels.
@ast
Concordance of increased B1 ce ...... dent on BLK expression levels.
@en
P2093
P2860
P50
P356
P1476
Concordance of increased B1 ce ...... dent on BLK expression levels.
@en
P2093
Alejandro Díaz-Barreiro
Bernard Lauwerys
Fadi El Salem
Harini Bagavant
Mireia Castillo-Martín
Nieves Varela
Ramesh Kumar
Ying-Yu Wu
P2860
P304
P356
10.4049/JIMMUNOL.1402736
P407
P577
2015-05-13T00:00:00Z