Potentiation of Excitotoxicity in HIV-1 Associated Dementia and the Significance of Glutaminase.
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HIV-1, methamphetamine and astrocytes at neuroinflammatory CrossroadsNicotine attenuates the effect of HIV-1 proteins on the neural circuits of working and contextual memoriesSerial deletion reveals structural basis and stability for the core enzyme activity of human glutaminase 1 isoforms: relevance to excitotoxic neurodegenerationBrain dysfunction in the era of combination antiretroviral therapy: implications for the treatment of the aging population of HIV-infected individualsGlutamate metabolism and HIV-associated neurocognitive disorders.Gene expression analysis in the thalamus and cerebrum of horses experimentally infected with West Nile virus.Cerebral metabolite changes prior to and after antiretroviral therapy in primary HIV infectionVoltage-gated potassium channels in human immunodeficiency virus type-1 (HIV-1)-associated neurocognitive disorders.Glutamate production by HIV-1 infected human macrophage is blocked by the inhibition of glutaminaseGlutaminase dysregulation in HIV-1-infected human microglia mediates neurotoxicity: relevant to HIV-1-associated neurocognitive disorders.Glutaminase-containing microvesicles from HIV-1-infected macrophages and immune-activated microglia induce neurotoxicity.HIV-1, methamphetamine and astrocyte glutamate regulation: combined excitotoxic implications for neuro-AIDS.Expression of ephrin receptors and ligands in postmortem brains of HIV-infected subjects with and without cognitive impairment.HIV-infected macrophages mediate neuronal apoptosis through mitochondrial glutaminaseIL-1β and TNF-α induce neurotoxicity through glutamate production: a potential role for neuronal glutaminase.In vitro glutaminase regulation and mechanisms of glutamate generation in HIV-1-infected macrophage.A coat of many colors: neuroimmune crosstalk in human immunodeficiency virus infection.Virus-induced transcriptional changes in the brain include the differential expression of genes associated with interferon, apoptosis, interleukin 17 receptor A, and glutamate signaling as well as flavivirus-specific upregulation of tRNA synthetasesN-(Pivaloyloxy)alkoxy-carbonyl Prodrugs of the Glutamine Antagonist 6-Diazo-5-oxo-l-norleucine (DON) as a Potential Treatment for HIV Associated Neurocognitive Disorders.Challenging New Targets for CNS-HIV Infection.Pathogenesis of Human Immunodeficiency Virus Type-1 (HIV-1)-Associated Dementia: Role of Voltage-Gated Potassium Channels.Glutaminase C overexpression in the brain induces learning deficits, synaptic dysfunctions, and neuroinflammation in mice.Glutaminase 1 regulates the release of extracellular vesicles during neuroinflammation through key metabolic intermediate alpha-ketoglutarate.
P2860
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P2860
Potentiation of Excitotoxicity in HIV-1 Associated Dementia and the Significance of Glutaminase.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Potentiation of Excitotoxicity ...... e Significance of Glutaminase.
@ast
Potentiation of Excitotoxicity ...... e Significance of Glutaminase.
@en
type
label
Potentiation of Excitotoxicity ...... e Significance of Glutaminase.
@ast
Potentiation of Excitotoxicity ...... e Significance of Glutaminase.
@en
prefLabel
Potentiation of Excitotoxicity ...... e Significance of Glutaminase.
@ast
Potentiation of Excitotoxicity ...... e Significance of Glutaminase.
@en
P2093
P2860
P1476
Potentiation of Excitotoxicity ...... e Significance of Glutaminase.
@en
P2093
Jialin Zheng
Nathan B Erdmann
Nick P Whitney
P2860
P304
P356
10.1016/J.CNR.2006.09.009
P577
2006-12-01T00:00:00Z