Mitochondrial dysfunction and glutamate excitotoxicity studied in primary neuronal cultures.
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The neuroprotective properties of calorie restriction, the ketogenic diet, and ketone bodiesDomoic acid toxicologic pathology: a reviewThe Role of Astrocytes in Multiple Sclerosis ProgressionMitochondrial stress and the pathogenesis of diabetic neuropathySex differences in mitochondrial (dys)function: Implications for neuroprotectionConditioned medium of human adipose-derived mesenchymal stem cells mediates protection in neurons following glutamate excitotoxicity by regulating energy metabolism and GAP-43 expressionNeurotoxicity of methamphetamine and 3,4-methylenedioxymethamphetamineCellular calcium deficiency plays a role in neuronal death caused by proteasome inhibitors.Differential NMDA receptor-dependent calcium loading and mitochondrial dysfunction in CA1 vs. CA3 hippocampal neuronsProgressive multiple sclerosis: from pathogenic mechanisms to treatment.GM1 and NGF modulate Ca2+ homeostasis and GAP43 mRNA expression in cultured dorsal root ganglion neurons with excitotoxicity induced by glutamate.Stimulation of glutamate receptors in cultured hippocampal neurons causes Ca2+-dependent mitochondrial contraction.Asiatic acid, a pentacyclic triterpene in Centella asiatica, attenuates glutamate-induced cognitive deficits in mice and apoptosis in SH-SY5Y cells.Transcriptomic responses in mouse brain exposed to chronic excess of the neurotransmitter glutamate.Liquiritin modulates ERK‑ and AKT/GSK‑3β‑dependent pathways to protect against glutamate‑induced cell damage in differentiated PC12 cells.Amphetamine toxicities: classical and emerging mechanisms.High yield derivation of enriched glutamatergic neurons from suspension-cultured mouse ESCs for neurotoxicology researchMolecular mechanisms of excitotoxicity and their relevance to pathogenesis of neurodegenerative diseasesMetabolic and functional differences between brain and spinal cord mitochondria underlie different predisposition to pathology.Coupling of the NMDA receptor to neuroprotective and neurodestructive events.Differential neuroprotective potential of CRMP2 peptide aptamers conjugated to cationic, hydrophobic, and amphipathic cell penetrating peptidesKainic Acid-induced neurotoxicity: targeting glial responses and glia-derived cytokines.18F-fluorobenzyl triphenyl phosphonium: a noninvasive sensor of brown adipose tissue thermogenesisThe role of N-methyl-D-aspartate receptor activation in homocysteine-induced death of retinal ganglion cellsNeuroprotection against traumatic brain injury by a peptide derived from the collapsin response mediator protein 2 (CRMP2)Neuroprotective and antioxidant effects of novel benzofuran-2-carboxamide derivatives.Human Schwann cells retain essential phenotype characteristics after immortalizationNeuronal cell death in neurodegenerative diseases: recurring themes around protein handling.Mitochondrial stop and go: signals that regulate organelle movement.The therapeutic potential of mitochondrial channels in cancer, ischemia-reperfusion injury, and neurodegeneration.Susceptibility to bystander DNA damage is influenced by replication and transcriptional activity.Calcium-dependent mitochondrial function and dysfunction in neurons.Mitochondrial swelling measurement in situ by optimized spatial filtering: astrocyte-neuron differencesnNOS(+) striatal neurons, a subpopulation spared in Huntington's Disease, possess functional NMDA receptors but fail to generate mitochondrial ROS in response to an excitotoxic challengeAssessing neuronal bioenergetic status.Oxidative impairment of hippocampal long-term potentiation involves activation of protein phosphatase 2A and is prevented by ketone bodies.Role of K(ATP) channels in protection against neuronal excitatory insults.The dichotomy of NMDA receptor signaling.Glutamate receptor activation triggers OPA1 release and induces apoptotic cell death in ischemic rat retina.Kainate-induced calcium overload of cortical neurons in vitro: Dependence on expression of AMPAR GluA2-subunit and down-regulation by subnanomolar ouabain.
P2860
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P2860
Mitochondrial dysfunction and glutamate excitotoxicity studied in primary neuronal cultures.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Mitochondrial dysfunction and ...... in primary neuronal cultures.
@ast
Mitochondrial dysfunction and ...... in primary neuronal cultures.
@en
type
label
Mitochondrial dysfunction and ...... in primary neuronal cultures.
@ast
Mitochondrial dysfunction and ...... in primary neuronal cultures.
@en
prefLabel
Mitochondrial dysfunction and ...... in primary neuronal cultures.
@ast
Mitochondrial dysfunction and ...... in primary neuronal cultures.
@en
P356
P1476
Mitochondrial dysfunction and ...... in primary neuronal cultures.
@en
P2093
Nicholls DG
P304
P356
10.2174/1566524043479239
P577
2004-03-01T00:00:00Z